The contribution of reactive oxygen species and p38 mitogen-activated protein kinase to myofilament oxidation and progression of heart failure in rabbits.
- Published in:
- 2010
- By:
- Publication type:
- journal article
Works by Di Lisa, F
Results: 7
1
2
P429 The activity of monoamine oxidases is tightly related to mitochondrial membrane fluidity altered by aging, obesity and oxidative stress.
- Published in:
- Cardiovascular Research, 2014, v. 103, n. suppl_1, p. S79, doi. 10.1093/cvr/cvu091.109
- By:
- Publication type:
- Article
3
P138 p66ShcA adaptor protein facilitates heart rupture via activation of MMP-2 in an in vivo model of myocardial infarction in mice.
- Published in:
- Cardiovascular Research, 2014, v. 103, n. suppl_1, p. S24, doi. 10.1093/cvr/cvu082.77
- By:
- Publication type:
- Article
4
31 Metabolomic identification of substrates for monoamine oxidases in hearts subjected to oxidative stress.
- Published in:
- Cardiovascular Research, 2014, v. 103, n. suppl_1, p. S4, doi. 10.1093/cvr/cvu077.3
- By:
- Publication type:
- Article
5
P253 Oxidative stress causes the release of a specific subset of proteins from viable cardiac myocytes.
- Published in:
- Cardiovascular Research, 2014, v. 103, n. suppl_1, p. S45, doi. 10.1093/cvr/cvu082.184
- By:
- Publication type:
- Article
6
771 The opening of the permeability transition pore is responsible for mitochondrial dysfunction and cell death caused by N-methyl-N′-nitro-N-nitrosoguanidine (MNNG)
- Published in:
- 2004
- By:
- Publication type:
- Abstract
7
764 Activation of PPARalpha is followed by its down regulation in the murine atrial cell line HL5
- Published in:
- 2004
- By:
- Publication type:
- Abstract