Found: 9
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Role of glycogen synthase kinase-3β and PPAR-γ on epithelial-to-mesenchymal transition in DSS-induced colorectal fibrosis.
- Published in:
- PLoS ONE, 2017, v. 12, n. 2, p. 1, doi. 10.1371/journal.pone.0171093
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- Article
Inactivation of mitochondrial MUL1 E3 ubiquitin ligase deregulates mitophagy and prevents diet-induced obesity in mice.
- Published in:
- Frontiers in Molecular Biosciences, 2024, p. 1, doi. 10.3389/fmolb.2024.1397565
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- Article
Mitochondrial MUL1 E3 ubiquitin ligase regulates Hypoxia Inducible Factor (HIF-1α) and metabolic reprogramming by modulating the UBXN7 cofactor protein.
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- Scientific Reports, 2020, v. 10, n. 1, p. 1, doi. 10.1038/s41598-020-58484-8
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- Article
Tebuconazole and Econazole Act Synergistically in Mediating Mitochondrial Stress, Energy Imbalance, and Sequential Activation of Autophagy and Apoptosis in Mouse Sertoli TM4 Cells: Possible Role of AMPK/ULK1 Axis.
- Published in:
- Toxicological Sciences, 2019, v. 169, n. 1, p. 209, doi. 10.1093/toxsci/kfz031
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- Article
The C-terminus of the prototypical M2 muscarinic receptor localizes to the mitochondria and regulates cell respiration under stress conditions.
- Published in:
- PLoS Biology, 2024, v. 22, n. 4, p. 1, doi. 10.1371/journal.pbio.3002582
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- Article
Protein Stability Regulation in Osteosarcoma: The Ubiquitin-like Modifications and Glycosylation as Mediators of Tumor Growth and as Targets for Therapy.
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- Cells (2073-4409), 2024, v. 13, n. 6, p. 537, doi. 10.3390/cells13060537
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- Article
Carvacrol reduces adipogenic differentiation by modulating autophagy and ChREBP expression.
- Published in:
- PLoS ONE, 2018, v. 13, n. 11, p. 1, doi. 10.1371/journal.pone.0206894
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- Article
Variants of G protein-coupled receptors: a reappraisal of their role in receptor regulation.
- Published in:
- Biochemical Society Transactions, 2016, v. 44, n. 2, p. 589, doi. 10.1042/BST20150239
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- Article
Role of the Mitochondrial E3 Ubiquitin Ligases as Possible Therapeutic Targets in Cancer Therapy.
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- International Journal of Molecular Sciences, 2023, v. 24, n. 24, p. 17176, doi. 10.3390/ijms242417176
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- Article