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High glucose induces mitochondrial dysfunction independently of protein O-GlcNAcylation.
- Published in:
- Biochemical Journal, 2015, v. 467, n. 1, p. 115, doi. 10.1042/BJ20141018
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- Publication type:
- Article
Hyperglycemia suppresses cardiomyocyte bioenergetic reserve independent of O‐GlcNAcylation (1155.5).
- Published in:
- FASEB Journal, 2014, v. 28, p. N.PAG, doi. 10.1096/fasebj.28.1_supplement.1155.5
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- Article
Cardiomyocyte Oga haploinsufficiency increases O-GlcNAcylation but hastens ventricular dysfunction following myocardial infarction.
- Published in:
- PLoS ONE, 2020, v. 15, n. 11, p. 1, doi. 10.1371/journal.pone.0242250
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- Article
Chronic Benzene Exposure Aggravates Pressure Overload-Induced Cardiac Dysfunction.
- Published in:
- Toxicological Sciences, 2022, v. 185, n. 1, p. 64, doi. 10.1093/toxsci/kfab125
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- Article
E2f1 deletion attenuates infarct-induced ventricular remodeling without affecting O-GlcNAcylation.
- Published in:
- Basic Research in Cardiology, 2019, v. 114, n. 4, p. N.PAG, doi. 10.1007/s00395-019-0737-y
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- Publication type:
- Article
Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophy.
- Published in:
- Basic Research in Cardiology, 2017, v. 112, n. 3, p. 1, doi. 10.1007/s00395-017-0612-7
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- Article