Works by Crews, Fulton T.
Results: 63
NADPH oxidase and aging drive microglial activation, oxidative stress, and dopaminergic neurodegeneration following systemic LPS administration.
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- Glia, 2013, v. 61, n. 6, p. 855, doi. 10.1002/glia.22479
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- Article
ATP-P2X<sub>7</sub> receptor signaling controls basal and TNFα-stimulated glial cell proliferation.
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- Glia, 2012, v. 60, n. 4, p. 661, doi. 10.1002/glia.22302
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- Article
Systemic LPS causes chronic neuroinflammation and progressive neurodegeneration.
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- Glia, 2007, v. 55, n. 5, p. 453, doi. 10.1002/glia.20467
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- Article
Ethanol Induction of Innate Immune Signals Across BV2 Microglia and SH-SY5Y Neuroblastoma Involves Induction of IL-4 and IL-13.
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- Brain Sciences (2076-3425), 2019, v. 9, n. 9, p. 228, doi. 10.3390/brainsci9090228
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- Article
Insulin Stimulates Phosphatidylinositol 3-Kinase Activity in Rat Neuronal Primary Cultures.
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- Journal of Neurochemistry, 1993, v. 61, n. 1, p. 360, doi. 10.1111/j.1471-4159.1993.tb03578.x
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- Article
Calcium- Versus G Protein-Mediated Phosphoinositide Hydrolysis in Rat Cerebral Cortical Synaptoneurosomes.
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- Journal of Neurochemistry, 1990, v. 55, n. 3, p. 1022, doi. 10.1111/j.1471-4159.1990.tb04592.x
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- Article
Changes in Neuroimmune and Neuronal Death Markers after Adolescent Alcohol Exposure in Rats are Reversed by Donepezil.
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- Scientific Reports, 2019, v. 9, n. 1, p. N.PAG, doi. 10.1038/s41598-019-47039-1
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- Article
Pharmacological Treatment of Alcohol Dependence.
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- JAMA: Journal of the American Medical Association, 1999, v. 281, n. 14, p. 1318, doi. 10.1001/jama.281.14.1318
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- Article
NADPH oxidase and endoplasmic reticulum stress is associated with neuronal degeneration in orbitofrontal cortex of individuals with alcohol use disorder.
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- Addiction Biology, 2023, v. 28, n. 1, p. 1, doi. 10.1111/adb.13262
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- Article
The Toll-like receptor 7 agonist imiquimod increases ethanol self-administration and induces expression of Toll-like receptor related genes.
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- 2022
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- journal article
Long-lasting microbial dysbiosis and altered enteric neurotransmitters in adult rats following adolescent binge ethanol exposure.
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- 2021
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- journal article
Neuroimmune and epigenetic involvement in adolescent binge ethanol-induced loss of basal forebrain cholinergic neurons: Restoration with voluntary exercise.
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- 2020
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- journal article
Phorbol Ester-Induced Upregulation of Angiotensin II Receptors in Neuronal Cultures Is Potentiated by a Calcium Ionophore.
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- Journal of Neurochemistry, 1988, v. 51, n. 1, p. 153, doi. 10.1111/j.1471-4159.1988.tb04849.x
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- Article
Variations in Membrane Sensitivity of Brain Region Synaptosomes to the Effects of Ethanol In Vitro and Chronic In Vivo Treatment.
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- Journal of Neurochemistry, 1987, v. 49, n. 1, p. 158, doi. 10.1111/j.1471-4159.1987.tb03408.x
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- Article
Protein Kinase C Agonists Increase the Expression of Angiotensin II Receptors in Neuronal Cultures.
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- Journal of Neurochemistry, 1987, v. 48, n. 6, p. 1954, doi. 10.1111/j.1471-4159.1987.tb05760.x
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- Article
Cholinergic- and Adrenergic-Stimulated Inositide Hydrolysis in Brain: Interaction, Regional Distribution, and Coupling Mechanisms.
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- Journal of Neurochemistry, 1985, v. 45, n. 4, p. 1076, doi. 10.1111/j.1471-4159.1985.tb05526.x
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- Article
Identification and Properties of Methyltransferases That Synthesize Phosphatidylcholine in Rat Brain Synaptosomes.
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- Journal of Neurochemistry, 1980, v. 34, n. 6, p. 1491, doi. 10.1111/j.1471-4159.1980.tb11229.x
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- Article
Microglial depletion and repopulation in brain slice culture normalizes sensitized proinflammatory signaling.
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- 2020
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- journal article
Calcium- versus G-Protein-Activated Phosphoinositide Hydrolysis in Synaptoneurosomes from Young and Old Rats<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1989, v. 568, n. 1, p. 187, doi. 10.1111/j.1749-6632.1989.tb12507.x
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- Article
Adenosine and Its Analogs Stimulate Phosphoinositide Hydrolysis in the Kidney.
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- Pharmacology, 1990, v. 40, n. 2, p. 90, doi. 10.1159/000138646
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- Article
Epigenetic regulation of microglia and neurons by proinflammatory signaling following adolescent intermittent ethanol (AIE) exposure and in human AUD.
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- Advances in Drug & Alcohol Research, 2024, p. 1, doi. 10.3389/adar.2024.12094
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- Article
A multicomponent ethanol response battery across a cumulative dose ethanol challenge reveals diminished adolescent rat ethanol responsivity relative to adults.
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- Advances in Drug & Alcohol Research, 2024, p. 1, doi. 10.3389/adar.2023.11888
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- Article
Ethanol Induces Secretion of Proinflammatory Extracellular Vesicles That Inhibit Adult Hippocampal Neurogenesis Through G9a/GLP-Epigenetic Signaling.
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- Frontiers in Immunology, 2022, v. 13, p. 1, doi. 10.3389/fimmu.2022.866073
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- Article
Persistent Decreases in Adult Subventricular and Hippocampal Neurogenesis Following Adolescent Intermittent Ethanol Exposure.
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- Frontiers in Behavioral Neuroscience, 2017, p. 1, doi. 10.3389/fnbeh.2017.0015
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- Article
Regulation of inositol transport by glucose and protein kinase C in mesangial cells.
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- Kidney International, 1992, v. 42, n. 1, p. 33, doi. 10.1038/ki.1992.257
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- Article
Chronic ethanol increases systemic TLR3 agonist-induced neuroinflammation and neurodegeneration.
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- Journal of Neuroinflammation, 2012, v. 9, n. 1, p. 130, doi. 10.1186/1742-2094-9-130
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- Article
Increased systemic and brain cytokine production and neuroinflammation by endotoxin following ethanol treatment.
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- Journal of Neuroinflammation, 2008, v. 5, p. 1, doi. 10.1186/1742-2094-5-10
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- Article
Adolescent Intermittent Alcohol Exposure: Deficits in Object Recognition Memory and Forebrain Cholinergic Markers.
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- PLoS ONE, 2015, v. 10, n. 10, p. 1, doi. 10.1371/journal.pone.0140042
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- Article
Glutamate/NMDA excitotoxicity and HMGB1/TLR4 neuroimmune toxicity converge as components of neurodegeneration.
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- AIMS Molecular Science, 2015, v. 2, n. 2, p. 77, doi. 10.3934/molsci.2015.2.77
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- Article
Ethanol induces interferon expression in neurons via TRAIL: role of astrocyte-to-neuron signaling.
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- Psychopharmacology, 2019, v. 236, n. 10, p. 2881, doi. 10.1007/s00213-018-5153-8
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- Article
Interaction of Nutrition and Binge Ethanol Treatment on Brain Damage and Withdrawal.
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- Journal of Biomedical Science, 2001, v. 8, n. 1, p. 134, doi. 10.1007/BF02255982
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- Article
Cholinergic REST-G9a gene repression through HMGB1-TLR4 neuroimmune signaling regulates basal forebrain cholinergic neuron phenotype.
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- Frontiers in Molecular Neuroscience, 2022, v. 15, p. 1, doi. 10.3389/fnmol.2022.992627
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- Article
Binge ethanol exposure during adolescence leads to a persistent loss of neurogenesis in the dorsal and ventral hippocampus that is associated with impaired adult cognitive functioning.
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- Frontiers in Neuroscience, 2015, v. 9, p. 1, doi. 10.3389/fnins.2015.00035
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- Article
Inflammasome-IL-1β signaling mediates ethanol inhibition of hippocampal neurogenesis.
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- Frontiers in Neuroscience, 2012, p. 1, doi. 10.3389/fnins.2012.00077
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- Article
Microglial depletion alters the brain neuroimmune response to acute binge ethanol withdrawal.
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- 2017
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- journal article
Microglial-derived miRNA let-7 and HMGB1 contribute to ethanol-induced neurotoxicity via TLR7.
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- 2017
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- journal article
Abstinence following Alcohol Drinking Produces Depression-Like Behavior and Reduced Hippocampal Neurogenesis in Mice.
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- Neuropsychopharmacology, 2009, v. 34, n. 5, p. 1209, doi. 10.1038/npp.2008.90
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- Article
Adolescent Intermittent Ethanol Exposure Is Associated with Increased Risky Choice and Decreased Dopaminergic and Cholinergic Neuron Markers in Adult Rats.
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- International Journal of Neuropsychopharmacology, 2015, v. 18, n. 2, p. 1, doi. 10.1093/ijnp/pyu003
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- Article
Long-term suppression of forebrain neurogenesis and loss of neuronal progenitor cells following prolonged alcohol dependence in rats.
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- International Journal of Neuropsychopharmacology, 2010, v. 13, n. 5, p. 583, doi. 10.1017/S1461145710000246
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- Article
Stress and Alcohol Priming of Brain Toll-Like Receptor Signaling in Alcohol Use Disorder.
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- Alcohol & Alcoholism, 2018, v. 53, n. 6, p. 639, doi. 10.1093/alcalc/agy061
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- Article
Mechanisms of Neurodegeneration and Regeneration in Alcoholism.
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- Alcohol & Alcoholism, 2009, v. 44, n. 2, p. 115, doi. 10.1093/alcalc/agn079
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- Article
Loss of Basal Forebrain Cholinergic Neurons Following Adolescent Binge Ethanol Exposure: Recovery With the Cholinesterase Inhibitor Galantamine.
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- Frontiers in Behavioral Neuroscience, 2021, v. 14, p. N.PAG, doi. 10.3389/fnbeh.2021.652494
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- Article
TRAIL Mediates Neuronal Death in AUD: A Link between Neuroinflammation and Neurodegeneration.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 5, p. 2547, doi. 10.3390/ijms22052547
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- Article
Acceleration of β-receptor desensitization in combined administration of antidepressants and phenoxybenzamine.
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- Nature, 1981, v. 290, n. 5809, p. 787, doi. 10.1038/290787a0
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- Article
Persistent Loss of Hippocampal Neurogenesis and Increased Cell Death following Adolescent, but Not Adult, Chronic Ethanol Exposure.
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- Developmental Neuroscience, 2014, v. 36, n. 3/4, p. 297, doi. 10.1159/000362874
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- Article
Persistent Adult Neuroimmune Activation and Loss of Hippocampal Neurogenesis Following Adolescent Ethanol Exposure: Blockade by Exercise and the Anti-inflammatory Drug Indomethacin.
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- Frontiers in Neuroscience, 2018, p. 1, doi. 10.3389/fnins.2018.00200
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- Article
Chronic alcohol exposure reduces hippocampal neurogenesis and dendritic growth of newborn neurons.
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- European Journal of Neuroscience, 2005, v. 21, n. 10, p. 2711, doi. 10.1111/j.1460-9568.2005.04120.x
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- Article
Extracellular microvesicles promote microglia‐mediated pro‐inflammatory responses to ethanol.
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- Journal of Neuroscience Research, 2021, v. 99, n. 8, p. 1940, doi. 10.1002/jnr.24813
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- Article
Adolescent binge ethanol-induced loss of basal forebrain cholinergic neurons and neuroimmune activation are prevented by exercise and indomethacin.
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- PLoS ONE, 2018, v. 13, n. 10, p. 1, doi. 10.1371/journal.pone.0204500
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- Article
HMGB1/IL-1β complexes in plasma microvesicles modulate immune responses to burn injury.
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- PLoS ONE, 2018, v. 13, n. 3, p. 1, doi. 10.1371/journal.pone.0195335
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- Article