Found: 11
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Molecular bases of the antigens of the Lutheran blood group system.
- Published in:
- Transfusion, 2003, v. 43, n. 12, p. 1729, doi. 10.1111/j.0041-1132.2003.00600.x
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- Publication type:
- Article
Acquired and transient RBC CD55 deficiency (Inab phenotype) and anti-IFC.
- Published in:
- 2002
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- Publication type:
- journal article
The Molecular Basis of the Lutheran Blood Group Antigens.
- Published in:
- Vox Sanguinis, 2002, v. 83, p. 189, doi. 10.1111/j.1423-0410.2002.tb05298.x
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- Publication type:
- Article
A novel KEL c.1414-1G>T allele in a polish patient with anti-Ku antibody.
- Published in:
- 2022
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- Publication type:
- journal article
CROK (CROM19): A new high‐prevalence antigen in the Cromer blood group system.
- Published in:
- Transfusion, 2021, v. 61, n. 11, p. E85, doi. 10.1111/trf.16680
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- Publication type:
- Article
A large deletion spanning XG and GYG2 constitutes a genetic basis of the Xg<sub>null</sub> phenotype, underlying anti-Xg<sup>a</sup> production.
- Published in:
- 2019
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- Publication type:
- journal article
Two MER2-negative individuals with the same novel CD151 mutation and evidence for clinical significance of anti-MER2.
- Published in:
- Transfusion, 2008, v. 48, n. 9, p. 1912, doi. 10.1111/j.1537-2995.2008.01792.x
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- Publication type:
- Article
Different inactivating mutations in the LU genes of three individuals with the Lutheran-null phenotype.
- Published in:
- Transfusion, 2007, v. 47, n. 3, p. 492, doi. 10.1111/j.1537-2995.2006.01141.x
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- Publication type:
- Article
A KEL gene encoding serine at position 193 of the Kell glycoprotein results in expression of KEL1 antigen.
- Published in:
- Transfusion, 2006, v. 46, n. 11, p. 1879, doi. 10.1111/j.1537-2995.2006.00993.x
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- Publication type:
- Article
New mutations in C1GALT1C1 in individuals with Tn positive phenotype.
- Published in:
- British Journal of Haematology, 2008, v. 142, n. 4, p. 657, doi. 10.1111/j.1365-2141.2008.07215.x
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- Publication type:
- Article
Disruption of the tumour-associated EMP3 enhances erythroid proliferation and causes the MAM-negative phenotype.
- Published in:
- Nature Communications, 2020, v. 11, n. 1, p. 1, doi. 10.1038/s41467-020-17060-4
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- Publication type:
- Article