Works matching AU Coffey, Nathan J.

Results: 8

Bleomycin Induces Drug Efflux in Lungs: A Pitfall for Pharmacological Studies of Pulmonary Fibrosis.

Published in:

American Journal of Respiratory Cell & Molecular Biology, 2020, v. 62, n. 2, p. 178, doi. 10.1165/rcmb.2018-0147OC

By:

Park, Joshua K.;
Coffey, Nathan J.;
Bodine, Steven P.;
Zawatsky, Charles N.;
Jay, Lindsey;
Gahl, William A.;
Kunos, George;
Gochuico, Bernadette R.;
Malicdan, May Christine V.;
Cinar, Resat

Publication type:

Article

Synthesis of 13C6‐labeled, dual‐target inhibitor of cannabinoid‐1 receptor (CB1R) and inducible nitric oxide synthase (iNOS).

Published in:

Journal of Labelled Compounds & Radiopharmaceuticals, 2018, v. 61, n. 10, p. 773, doi. 10.1002/jlcr.3639

By:

Iyer, Malliga R.;
Cinar, Resat;
Coffey, Nathan J.;
Kunos, George

Publication type:

Article

Cover Image.

Published in:

Journal of Labelled Compounds & Radiopharmaceuticals, 2018, v. 61, n. 10, p. i, doi. 10.1002/jlcr.3676

By:

Iyer, Malliga R.;
Cinar, Resat;
Coffey, Nathan J.;
Kunos, George

Publication type:

Article

Synthesis of S-2-(( S)-3-(4-chlorophenyl)- N'-((4-chlorophenyl)sulfonyl)-4-phenyl-4,5-dihydro-1 H-pyrazole-1-carboximidamido)-3-(methyl- d3)butanamide-d5, octadeuterated JD5037.

Published in:

Journal of Labelled Compounds & Radiopharmaceuticals, 2017, v. 60, n. 10, p. 460, doi. 10.1002/jlcr.3521

By:

Iyer, Malliga R.;
Cinar, Resat;
Coffey, Nathan J.;
Chorvat, Robert J.;
Kunos, George

Publication type:

Article

CB1R and iNOS are distinct players promoting pulmonary fibrosis in Hermansky–Pudlak syndrome.

Published in:

Clinical & Translational Medicine, 2021, v. 11, n. 7, p. 1, doi. 10.1002/ctm2.471

By:

Cinar, Resat;
Park, Joshua K.;
Zawatsky, Charles N.;
Coffey, Nathan J.;
Bodine, Steven P.;
Abdalla, Jasmina;
Yokoyama, Tadafumi;
Jourdan, Tony;
Jay, Lindsey;
Zuo, Mei Xing G.;
O'Brien, Kevin J.;
Huang, Junfeng;
Mackie, Ken;
Alimardanov, Asaf;
Iyer, Malliga R.;
Gahl, William A.;
Kunos, George;
Gochuico, Bernadette R.;
Malicdan, May Christine V.

Publication type:

Article

Cholesterol as a modulator of cannabinoid receptor CB2 signaling.

Published in:

Scientific Reports, 2021, v. 11, n. 1, p. 1, doi. 10.1038/s41598-021-83245-6

By:

Yeliseev, Alexei;
Iyer, Malliga R.;
Joseph, Thomas T.;
Coffey, Nathan J.;
Cinar, Resat;
Zoubak, Lioudmila;
Kunos, George;
Gawrisch, Klaus

Publication type:

Article

Ephrin-A3 promotes and maintains slow muscle fiber identity during postnatal development and reinnervation.

Published in:

Journal of Cell Biology, 2015, v. 211, n. 5, p. 1077, doi. 10.1083/jcb.201502036

By:

Stark, Danny A.;
Coffey, Nathan J.;
Pancoast, Hannah R.;
Arnold, Laura L.;
Walker, J. Peyton D.;
Vallée, Joanne;
Robitaille, Richard;
Garcia, Michael L.;
Cornelison, D. D. W.

Publication type:

Article

Cannabinoid‐1 receptor deletion in podocytes mitigates both glomerular and tubular dysfunction in a mouse model of diabetic nephropathy.

Published in:

Diabetes, Obesity & Metabolism, 2018, v. 20, n. 3, p. 698, doi. 10.1111/dom.13150

By:

Jourdan, Tony;
Park, Joshua K.;
Varga, Zoltán V.;
Pálóczi, János;
Coffey, Nathan J.;
Rosenberg, Avi Z.;
Godlewski, Grzegorz;
Cinar, Resat;
Mackie, Ken;
Pacher, Pal;
Kunos, George

Publication type:

Article

Works matching AU Coffey, Nathan J.

Bleomycin Induces Drug Efflux in Lungs: A Pitfall for Pharmacological Studies of Pulmonary Fibrosis.

Synthesis of <sup>13</sup>C<sub>6</sub>‐labeled, dual‐target inhibitor of cannabinoid‐1 receptor (CB<sub>1</sub>R) and inducible nitric oxide synthase (iNOS).

Cover Image.

Synthesis of S-2-(( S)-3-(4-chlorophenyl)- N'-((4-chlorophenyl)sulfonyl)-4-phenyl-4,5-dihydro-1 H-pyrazole-1-carboximidamido)-3-(methyl- d<sub>3</sub>)butanamide-d<sub>5</sub>, octadeuterated JD5037.

CB<sub>1</sub>R and iNOS are distinct players promoting pulmonary fibrosis in Hermansky–Pudlak syndrome.

Cholesterol as a modulator of cannabinoid receptor CB2 signaling.

Ephrin-A3 promotes and maintains slow muscle fiber identity during postnatal development and reinnervation.

Cannabinoid‐1 receptor deletion in podocytes mitigates both glomerular and tubular dysfunction in a mouse model of diabetic nephropathy.