Found: 2
Select item for more details and to access through your institution.
Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing human SOD1 gene mutation.
- Published in:
- Neuropathology, 2001, v. 21, n. 1, p. 67, doi. 10.1046/j.1440-1789.2001.00359.x
- By:
- Publication type:
- Article
Pathogenesis of two axonopathies does not require axonal neurofilaments.
- Published in:
- Nature, 1998, v. 391, n. 6667, p. 584, doi. 10.1038/35378
- By:
- Publication type:
- Article