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ELP1, the Gene Mutated in Familial Dysautonomia, Is Required for Normal Enteric Nervous System Development and Maintenance and for Gut Epithelium Homeostasis.
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- Journal of Neuroscience, 2024, v. 44, n. 37, p. 1, doi. 10.1523/JNEUROSCI.2253-23.2024
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- Article
Elongator and codon bias regulate protein levels in mammalian peripheral neurons.
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- Nature Communications, 2018, v. 9, p. 1, doi. 10.1038/s41467-018-03221-z
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- Article
Mouse models for Familial Dysautonomia reveal underlying cellular and molecular mechanisms that cause the human disease.
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- International Journal of Developmental Neuroscience, 2015, v. 47, p. 4, doi. 10.1016/j.ijdevneu.2015.04.021
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- Article
IKAP/Elp1 Is Required In Vivo for Neurogenesis and Neuronal Survival, but Not for Neural Crest Migration.
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- PLoS ONE, 2012, v. 7, n. 2, p. 1, doi. 10.1371/journal.pone.0032050
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- Article
Nociceptive sensory neurons derive from contralaterally migrating, fate-restricted neural crest cells.
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- Nature Neuroscience, 2007, v. 10, n. 10, p. 1287, doi. 10.1038/nn1962
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- Article
Reduction of retinal ganglion cell death in mouse models of familial dysautonomia using AAV-mediated gene therapy and splicing modulators.
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- Scientific Reports, 2023, v. 13, n. 1, p. 1, doi. 10.1038/s41598-023-45376-w
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- Article