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The spleen of patients with myelofibrosis harbors defective mesenchymal stromal cells.
- Published in:
- American Journal of Hematology, 2018, v. 93, n. 5, p. 615, doi. 10.1002/ajh.25047
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- Article
Increased plasma nicotinamide phosphoribosyltransferase is associated with a hyperproliferative phenotype and restrains disease progression in MPN-associated myelofibrosis.
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- American Journal of Hematology, 2016, v. 91, n. 7, p. 709, doi. 10.1002/ajh.24388
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- Article
Circulating Polymorphonuclear Myeloid-Derived Suppressor Cells (PMN-MDSCs) Have a Biological Role in Patients with Primary Myelofibrosis.
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- Cancers, 2024, v. 16, n. 14, p. 2556, doi. 10.3390/cancers16142556
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- Article
Type 1 CALR mutation allele frequency correlates with CD34/CXCR4 expression in myelofibrosis-type megakaryocyte dysplasia: A mechanism of disease progression?
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- Blood Cancer Journal, 2024, v. 14, n. 1, p. 1, doi. 10.1038/s41408-024-00991-2
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- Article
Synergistic inhibition of human neuroblastoma-related angiogenesis by vinblastine and rapamycin.
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- Oncogene, 2005, v. 24, n. 45, p. 6785, doi. 10.1038/sj.onc.1208829
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- Article
Clonal Megakaryocyte Dysplasia with Isolated Thrombocytosis Is a Distinct Myeloproliferative Neoplasm Phenotype.
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- Acta Haematologica, 2023, v. 146, n. 1, p. 14, doi. 10.1159/000527284
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- Article
Clonal Megakaryocyte Dysplasia with Normal Blood Values Is a Distinct Myeloproliferative Neoplasm.
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- Acta Haematologica, 2022, v. 145, n. 1, p. 30, doi. 10.1159/000517207
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- Article
Circulating Mesenchymal Stromal Cells in Patients with Infantile Hemangioma: Evaluation of Their Functional Capacity and Gene Expression Profile.
- Published in:
- Cells (2073-4409), 2024, v. 13, n. 3, p. 254, doi. 10.3390/cells13030254
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- Article
Apolipoprotein E in idiopathic nephrotic syndrome and focal segmental glomerulosclerosis.
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- Kidney International, 2003, v. 63, n. 2, p. 686, doi. 10.1046/j.1523-1755.2003.00777.x
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- Article
Methods in Molecular Biology, vol. 1768.
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- European Journal of Histochemistry, 2019, v. 63, n. 4, p. 1
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- Article
Clinical Relevance of VEGFA (rs3025039) +936 C>T Polymorphism in Primary Myelofibrosis: Susceptibility, Clinical Co-Variates, and Outcomes.
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- Genes, 2021, v. 12, n. 8, p. 1271, doi. 10.3390/genes12081271
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- Article
Constitutive STAT5 phosphorylation in CD34<sup>+</sup> cells of patients with primary myelofibrosis: Correlation with driver mutation status and disease severity.
- Published in:
- PLoS ONE, 2019, v. 14, n. 8, p. 1, doi. 10.1371/journal.pone.0220189
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- Article
Altered fibronectin expression and deposition by myeloproliferative neoplasm-derived mesenchymal stromal cells.
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- British Journal of Haematology, 2016, v. 172, n. 1, p. 140, doi. 10.1111/bjh.13471
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- Article
JAK2 Exon 14 Skipping in Patients with Primary Myelofibrosis: A Minor Splice Variant Modulated by the JAK2-V617F Allele Burden.
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- PLoS ONE, 2015, v. 10, n. 1, p. 1, doi. 10.1371/journal.pone.0116636
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- Article
<i>JAK2</i> V617F Genotype Is a Strong Determinant of Blast Transformation in Primary Myelofibrosis.
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- PLoS ONE, 2013, v. 8, n. 3, p. 1, doi. 10.1371/journal.pone.0059791
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- Article
Evidence that Prefibrotic Myelofibrosis Is Aligned along a Clinical and Biological Continuum Featuring Primary Myelofibrosis.
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- PLoS ONE, 2012, v. 7, n. 4, p. 1, doi. 10.1371/journal.pone.0035631
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- Article
VEGFA rs3025039 is associated with phenotype severity of myelofibrosis‐type megakaryocyte dysplasia.
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- EJHaem, 2023, v. 4, n. 3, p. 756, doi. 10.1002/jha2.708
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- Article
Angiotensin-converting enzyme (ACE) haplotypes and cyclosporine A (CsA) response: a model of the complex relationship between ACE quantitative trait locus and pathological phenotypes.
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- Human Molecular Genetics, 2005, v. 14, n. 16, p. 2357, doi. 10.1093/hmg/ddi238
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- Article
Impact of erythropoietin on the effects of irradiation under hypoxia.
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- Journal of Cancer Research & Clinical Oncology, 2009, v. 135, n. 11, p. 1615, doi. 10.1007/s00432-009-0609-1
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- Article
Tie2 Expressing Monocytes in the Spleen of Patients with Primary Myelofibrosis.
- Published in:
- PLoS ONE, 2016, v. 11, n. 6, p. 1, doi. 10.1371/journal.pone.0156990
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- Article