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Genetics, Pathophysiology, and Current Challenges in Von Hippel–Lindau Disease Therapeutics.
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- Diagnostics (2075-4418), 2024, v. 14, n. 17, p. 1909, doi. 10.3390/diagnostics14171909
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Autophagy: A Key Regulator of Homeostasis and Disease: An Overview of Molecular Mechanisms and Modulators.
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- Cells (2073-4409), 2022, v. 11, n. 15, p. 2262, doi. 10.3390/cells11152262
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Alzheimer's Disease: An Updated Overview of Its Genetics.
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- International Journal of Molecular Sciences, 2023, v. 24, n. 4, p. 3754, doi. 10.3390/ijms24043754
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Oxidative Stress and Mitochondrial Complex I Dysfunction Correlate with Neurodegeneration in an α-Synucleinopathy Animal Model.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 19, p. 11394, doi. 10.3390/ijms231911394
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Mechanistic Insight from Preclinical Models of Parkinson's Disease Could Help Redirect Clinical Trial Efforts in GDNF Therapy.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 21, p. 11702, doi. 10.3390/ijms222111702
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P3‐192: ACIDIC PH ACTIVATES AUTOPHAGY PATHWAY IN HUMAN HIPPOCAMPAL NEURAL PRECURSOR CELLS AND INDUCES THE NEURONAL PHENOTYPE.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2019, v. 15, p. P1003, doi. 10.1016/j.jalz.2019.06.3221
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P3‐157: SYNTHETIC PEPTIDES DERIVED FROM THE ACTIVE SITE OF BDNF AND SELENOAMIDES ENHANCE CELL SURVIVAL AND DIFFERENTIATION OF DENTAL PULP STEM CELLS (DPSCS) INTO NEURONAL PHENOTYPE: POTENTIAL THERAPEUTIC APPLICATION.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2019, v. 15, p. P991, doi. 10.1016/j.jalz.2019.06.3186
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P2‐144: MOLECULAR STUDY OF DEGENERATION IN PRESENILIN‐1 MUTATED (M146L OR A246E) FIBROBLAST ASSOCIATED WITH FAMILIAR ALZHEIMER'S DISEASE.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2019, v. 15, p. P628, doi. 10.1016/j.jalz.2019.06.2551
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INDUCTION OF NEURONAL DIFFERENTIATION, TAU EXPRESSION, AGGREGATION AND PHOSPHORYLATION BY GROWTH FACTOR REMOVAL AND ACIDIC PH CORRELATES WITH AN ENHANCED PROTEIN CLEARANCE AND RECYCLING IN HUMAN HIPPOCAMPAL NEURAL PRECURSOR CELLS (HHIPPNPCS).
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2016, v. 12, p. P871, doi. 10.1016/j.jalz.2016.06.1798
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Alzheimer's disease research in Ibero America.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2016, v. 12, n. 6, p. 749, doi. 10.1016/j.jalz.2016.04.007
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Growth factor removal and acidic changes affect the major proteolytic systems in human neural precursor cells (NPCs) and promote tau expression and oligomerization.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2015, v. 11, n. 7, p. P639, doi. 10.1016/j.jalz.2015.06.922
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Soluble amyloid-beta promotes inflammation: Caspase-5 characterizes the brains with Alzheimer's disease.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2015, v. 11, n. 7, p. P775, doi. 10.1016/j.jalz.2015.06.1731
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Evaluation of the effect of amyloid beta oligomers on neurotrophins and GSK-3/creb signal transduction pathways.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2015, v. 11, n. 7, p. P364, doi. 10.1016/j.jalz.2015.06.263
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Growth factor removal and acidic treatment induce neuronal differentiation in human neural precursor cells (hNPCs).
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2015, v. 11, n. 7, p. P634, doi. 10.1016/j.jalz.2015.06.906
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Tau splicing factor expression pattern in Alzheimer’s disease brain.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2015, v. 11, n. 7, p. P499, doi. 10.1016/j.jalz.2015.06.582
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Analysis of tau protein and amyloid-b expression in the olfactory bulb of the triple transgenic mouse model of Alzheimer’s disease.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2015, v. 11, n. 7, p. P636, doi. 10.1016/j.jalz.2015.06.913
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Biomarker Candidates for Alzheimer's Disease Unraveled through In Silico Differential Gene Expression Analysis.
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- Diagnostics (2075-4418), 2022, v. 12, n. 5, p. N.PAG, doi. 10.3390/diagnostics12051165
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Neurogenic and Neurotrophic Effects of BDNF Peptides in Mouse Hippocampal Primary Neuronal Cell Cultures.
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- PLoS ONE, 2013, v. 8, n. 1, p. 1, doi. 10.1371/journal.pone.0053596
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Patient-Derived Fibroblasts With Presenilin-1 Mutations, That Model Aspects of Alzheimer's Disease Pathology, Constitute a Potential Object for Early Diagnosis.
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- Frontiers in Aging Neuroscience, 2022, v. 14, p. 1, doi. 10.3389/fnagi.2022.921573
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Immunological and Functional Characterization of RhoGDI3 and Its Molecular Targets RhoG and RhoB in Human Pancreatic Cancerous and Normal Cells.
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- PLoS ONE, 2016, v. 11, n. 11, p. 1, doi. 10.1371/journal.pone.0166370
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Novel Nuclear Protein Complexes of Dystrophin 71 Isoforms in Rat Cultured Hippocampal GABAergic and Glutamatergic Neurons.
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- PLoS ONE, 2015, v. 10, n. 9, p. 1, doi. 10.1371/journal.pone.0137328
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Sera from Children with Autism Induce Autistic Features Which Can Be Rescued with a CNTF Small Peptide Mimetic in Rats.
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- PLoS ONE, 2015, v. 10, n. 3, p. 1, doi. 10.1371/journal.pone.0118627
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Dysregulated processes and genes associated with Progressive Supranuclear Palsy: A transcriptomics analysis.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.078060
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- Article
Autophagy dysregulation in human fibroblasts from Niemann‐Pick type C patients.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.079446
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Establishment of Cerebral Organoid‐like Structures from Dental Pulp Stem Cells (DPSCs) to Model Neurodegenerative Diseases.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.066380
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Discovery of potential Alzheimer's disease key biomarkers based on bioinformatics analysis.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.064366
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