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Inhibition of BACH1 (FANCJ) helicase by backbone discontinuity is overcome by increased motor ATPase or length of loading strand.
- Published in:
- Nucleic Acids Research, 2006, v. 34, n. 22, p. 6673, doi. 10.1093/nar/gkl964
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- Article
Single-molecule imaging reveals replication fork coupled formation of G-quadruplex structures hinders local replication stress signaling.
- Published in:
- Nature Communications, 2021, v. 12, n. 1, p. 1, doi. 10.1038/s41467-021-22830-9
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- Article
FANCJ promotes PARP1 activity during DNA replication that is essential in BRCA1 deficient cells.
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- Nature Communications, 2024, v. 15, n. 1, p. 1, doi. 10.1038/s41467-024-46824-5
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- Article
Molecular and cellular functions of the FANCJ DNA helicase defective in cancer and in Fanconi anemia.
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- Frontiers in Genetics, 2014, v. 5, p. 1, doi. 10.3389/fgene.2014.00372
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- Article
Crosstalk between BRCA- Fanconi anemia and mismatch repair pathways prevents MSH2-dependent aberrant DNA damage responses.
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- EMBO Journal, 2014, v. 33, n. 15, p. 1698, doi. 10.15252/embj.201387530
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- Article
Interaction between the helicases genetically linked to Fanconi anemia group J and Bloom's syndrome.
- Published in:
- EMBO Journal, 2011, v. 30, n. 4, p. 692, doi. 10.1038/emboj.2010.362
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- Article
The FANCJ/MutLα interaction is required for correction of the cross-link response in FA-J cells.
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- EMBO Journal, 2007, v. 26, n. 13, p. 3238, doi. 10.1038/sj.emboj.7601754
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- Article
Assessing the Link Between BACH1/FANCJ and MLH1 in DNA Crosslink Repair.
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- Environmental & Molecular Mutagenesis, 2010, v. 51, n. 6, p. 500, doi. 10.1002/em.20568
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- Article
FANCJ compensates for RAP80 deficiency and suppresses genomic instability induced by interstrand cross-links.
- Published in:
- Nucleic Acids Research, 2020, v. 48, n. 16, p. 9161, doi. 10.1093/nar/gkaa660
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- Article
FANCJ/BACH1 Acetylation at Lysine 1249 Regulates the DNA Damage Response.
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- PLoS Genetics, 2012, v. 8, n. 7, p. 1, doi. 10.1371/journal.pgen.1002786
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- Article