Found: 44
Select item for more details and to access through your institution.
AAV-mediated upregulation of VDAC1 rescues the mitochondrial respiration and sirtuins expression in a SOD1 mouse model of inherited ALS.
- Published in:
- Cell Death Discovery, 2024, v. 10, n. 1, p. 1, doi. 10.1038/s41420-024-01949-w
- By:
- Publication type:
- Article
Epigenetic Changes in Alzheimer's Disease: DNA Methylation and Histone Modification.
- Published in:
- Cells (2073-4409), 2024, v. 13, n. 8, p. 719, doi. 10.3390/cells13080719
- By:
- Publication type:
- Article
Anti-Biofilm Strategies: A Focused Review on Innovative Approaches.
- Published in:
- Microorganisms, 2024, v. 12, n. 4, p. 639, doi. 10.3390/microorganisms12040639
- By:
- Publication type:
- Article
Generation of a helper phage for the fluorescent detection of peptide-target interactions by dual-display phages.
- Published in:
- Scientific Reports, 2023, v. 13, n. 1, p. 1, doi. 10.1038/s41598-023-45087-2
- By:
- Publication type:
- Article
Selective Noradrenaline Depletion in the Neocortex and Hippocampus Induces Working Memory Deficits and Regional Occurrence of Pathological Proteins.
- Published in:
- Biology (2079-7737), 2023, v. 12, n. 9, p. 1264, doi. 10.3390/biology12091264
- By:
- Publication type:
- Article
Physiologic Response Evaluation of Human Foetal Osteoblast Cells within Engineered 3D-Printed Polylactic Acid Scaffolds.
- Published in:
- Biology (2079-7737), 2023, v. 12, n. 3, p. 424, doi. 10.3390/biology12030424
- By:
- Publication type:
- Article
The Role of the Transcription Factor Nrf2 in Alzheimer's Disease: Therapeutic Opportunities.
- Published in:
- Biomolecules (2218-273X), 2023, v. 13, n. 3, p. 549, doi. 10.3390/biom13030549
- By:
- Publication type:
- Article
Applications of CRISPR-Cas9 in Alzheimer's Disease and Related Disorders.
- Published in:
- International Journal of Molecular Sciences, 2022, v. 23, n. 15, p. 8714, doi. 10.3390/ijms23158714
- By:
- Publication type:
- Article
Small Hexokinase 1 Peptide against Toxic SOD1 G93A Mitochondrial Accumulation in ALS Rescues the ATP-Related Respiration.
- Published in:
- Biomedicines, 2021, v. 9, n. 8, p. 948, doi. 10.3390/biomedicines9080948
- By:
- Publication type:
- Article
Temporal and regional progression of Alzheimer's disease‐like pathology in 3xTg‐AD mice.
- Published in:
- Aging Cell, 2019, v. 18, n. 1, p. 1, doi. 10.1111/acel.12873
- By:
- Publication type:
- Article
S3‐02‐01: NECROPTOSIS ACTIVATION IN ALZHEIMER'S DISEASE.
- Published in:
- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2018, v. 14, p. P995, doi. 10.1016/j.jalz.2018.06.2735
- By:
- Publication type:
- Article
Genetic reduction of Nrf2 exacerbates cognitive deficits in a mouse model of Alzheimer's disease.
- Published in:
- Human Molecular Genetics, 2017, v. 26, n. 24, p. 4823, doi. 10.1093/hmg/ddx361
- By:
- Publication type:
- Article
Dyrk1 inhibition improves Alzheimer's disease-like pathology.
- Published in:
- Aging Cell, 2017, v. 16, n. 5, p. 1146, doi. 10.1111/acel.12648
- By:
- Publication type:
- Article
INCREASED SUSCEPTIBILITY TO NECROPTOSIS IN APP/PS1 MICE.
- Published in:
- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2017, v. 13, p. P667, doi. 10.1016/j.jalz.2017.06.807
- By:
- Publication type:
- Article
NECROPTOSIS AS A NOVEL MECHANISM UNDERLYING NEURONAL LOSS IN ALZHEIMER'S DISEASE.
- Published in:
- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2017, v. 13, p. P674, doi. 10.1016/j.jalz.2017.06.828
- By:
- Publication type:
- Article
Pim1 inhibition as a novel therapeutic strategy for Alzheimer's disease.
- Published in:
- Molecular Neurodegeneration, 2016, v. 11, p. 1, doi. 10.1186/s13024-016-0118-z
- By:
- Publication type:
- Article
Reducing Ribosomal Protein S6 Kinase 1 Expression Improves Spatial Memory and Synaptic Plasticity in a Mouse Model of Alzheimer's Disease.
- Published in:
- Journal of Neuroscience, 2015, v. 35, n. 41, p. 14042, doi. 10.1523/JNEUROSCI.2781-15.2015
- By:
- Publication type:
- Article
Reduced protein turnover mediates functional deficits in transgenic mice expressing the 25 kDa C-terminal fragment of TDP-43.
- Published in:
- Human Molecular Genetics, 2015, v. 24, n. 16, p. 4625, doi. 10.1093/hmg/ddv193
- By:
- Publication type:
- Article
Reducing ribosomal protein s6 kinase 1 ameliorates Alzheimer’s disease-like cognitive and synaptic deficits by reducing bace-1 translation.
- Published in:
- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2015, v. 11, n. 7, p. P203, doi. 10.1016/j.jalz.2015.07.196
- By:
- Publication type:
- Article
Chemogenetic facilitation of neuronal depolarization ameliorates Alzheimer’s disease–like cognitive deficits.
- Published in:
- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2015, v. 11, n. 7, p. P634, doi. 10.1016/j.jalz.2015.06.909
- By:
- Publication type:
- Article
Genetic reduction of mtor signaling ameliorates alzheimer's disease-related cognitive deficits and amyloid-b pathology by restoring hippocampal gene expression signature.
- Published in:
- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2014, v. 10, p. P192, doi. 10.1016/j.jalz.2014.04.224
- By:
- Publication type:
- Article
Genetic Reduction of Mammalian Target of Rapamycin Ameliorates Alzheimer's Disease-Like Cognitive and Pathological Deficits by Restoring Hippocampal Gene Expression Signature.
- Published in:
- Journal of Neuroscience, 2014, v. 34, n. 23, p. 7988, doi. 10.1523/JNEUROSCI.0777-14.2014
- By:
- Publication type:
- Article
Oxidative damage and amyloid-β metabolism in brain regions of the longest-lived rodents.
- Published in:
- Journal of Neuroscience Research, 2014, v. 92, n. 2, p. 195, doi. 10.1002/jnr.23320
- By:
- Publication type:
- Article
mTOR regulates tau phosphorylation and degradation: implications for Alzheimer's disease and other tauopathies.
- Published in:
- Aging Cell, 2013, v. 12, n. 3, p. 370, doi. 10.1111/acel.12057
- By:
- Publication type:
- Article
Glucocorticoids Exacerbate Cognitive Deficits in TDP-25 Transgenic Mice via a Glutathione-Mediated Mechanism: Implications for Aging, Stress and TDP-43 Proteinopathies.
- Published in:
- Journal of Neuroscience, 2013, v. 33, n. 3, p. 906, doi. 10.1523/JNEUROSCI.3314-12.2013
- By:
- Publication type:
- Article
Inducing autophagy by rapamycin before, but not after, the formation of plaques and tangles ameliorates cognitive deficits
- Published in:
- 2012
- By:
- Publication type:
- Abstract
Lifelong rapamycin administration ameliorates age-dependent cognitive deficits by reducing IL-1β and enhancing NMDA signaling.
- Published in:
- Aging Cell, 2012, v. 11, n. 2, p. 326, doi. 10.1111/j.1474-9726.2011.00791.x
- By:
- Publication type:
- Article
Methylene Blue Reduces Aβ Levels and Rescues Early Cognitive Deficit by Increasing Proteasome Activity.
- Published in:
- Brain Pathology, 2011, v. 21, n. 2, p. 140, doi. 10.1111/j.1750-3639.2010.00430.x
- By:
- Publication type:
- Article
Rapamycin rescues TDP-43 mislocalization and the associated low molecular weight neurofilament instability
- Published in:
- 2010
- By:
- Publication type:
- Abstract
Role of M1 receptors in Alzheimer's disease
- Published in:
- 2010
- By:
- Publication type:
- Abstract
Age-dependent changes in TDP-43 levels in a mouse model of Alzheimer disease are linked to Aβ oligomers accumulation.
- Published in:
- Molecular Neurodegeneration, 2010, v. 5, p. 51, doi. 10.1186/1750-1326-5-51
- By:
- Publication type:
- Article
Genetically Altering Aβ Distribution from the Brain to the Vasculature Ameliorates Tau Pathology.
- Published in:
- Brain Pathology, 2009, v. 19, n. 3, p. 421, doi. 10.1111/j.1750-3639.2008.00194.x
- By:
- Publication type:
- Article
Blocking Aβ42 Accumulation Delays the Onset and Progression of Tau Pathology via the C Terminus of Heat Shock Protein70-Interacting Protein: A Mechanistic Link between Aβ and Tau Pathology.
- Published in:
- Journal of Neuroscience, 2008, v. 28, n. 47, p. 12163, doi. 10.1523/JNEUROSCI.2464-08.2008
- By:
- Publication type:
- Article
O2-02-06: Blocking Aβ42 accumulation delays the onset and progression of Tau pathology and cognitive decline via CHIP: A mechanistic link between Aβ and Tau pathology
- Published in:
- 2008
- By:
- Publication type:
- Abstract
Genetically augmenting tau levels does not modulate the onset or progression of Aβ pathology in transgenic mice.
- Published in:
- Journal of Neurochemistry, 2007, v. 102, n. 4, p. 1053, doi. 10.1111/j.1471-4159.2007.04607.x
- By:
- Publication type:
- Article
Enhanced Ryanodine-Mediated Calcium Release in Mutant PS1-Expressing Alzheimer's Mouse Models.
- Published in:
- Annals of the New York Academy of Sciences, 2007, v. 1097, p. 265, doi. 10.1196/annals.1379.025
- By:
- Publication type:
- Article
O1-06-04: Reduction of both soluble Aβ and tau, but not soluble Aβ alone, ameliorates cognitive decline in transgenic mice with plaques and tangles
- Published in:
- 2006
- By:
- Publication type:
- Abstract
P1-125: Enhanced ryanodine receptor recruitment contributes to Ca<sup>2+</sup> disruptions in young, adult and aged Alzheimer disease mice
- Published in:
- 2006
- By:
- Publication type:
- Abstract
P1-451: M1 receptors play a central role in modulating AD-like pathology in transgenic mice
- Published in:
- 2006
- By:
- Publication type:
- Abstract
O1-06-04: Reduction of both soluble Aβ and tau, but not soluble Aβ alone, ameliorates cognitive decline in transgenic mice with plaques and tangles
- Published in:
- 2006
- By:
- Publication type:
- Abstract
P1-125: Enhanced ryanodine receptor recruitment contributes to Ca<sup>2+</sup> disruptions in young, adult and aged Alzheimer disease mice
- Published in:
- 2006
- By:
- Publication type:
- Abstract
P1-451: M1 receptors play a central role in modulating AD-like pathology in transgenic mice
- Published in:
- 2006
- By:
- Publication type:
- Abstract
Enhanced Ryanodine Receptor Recruitment Contributes to Ca<sup>2+</sup> Disruptions in Young, Adult, and Aged Alzheimer's Disease Mice.
- Published in:
- Journal of Neuroscience, 2006, v. 26, n. 19, p. 5180, doi. 10.1523/JNEUROSCI.0739-06.2006
- By:
- Publication type:
- Article
Dysregulated IP[sub3] Signaling in Cortical Neurons of Knock-In Mice Expressing an Alzheimer's-Linked Mutation in Presenilin1 Results in Exaggerated Ca<sup>2+</sup> Signals and Altered Membrane Excitability.
- Published in:
- Journal of Neuroscience, 2004, v. 24, n. 2, p. 508, doi. 10.1523/JNEUROSCI.4386-03.2004
- By:
- Publication type:
- Article