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Mitochondrial Phenotypes in Parkinson's Diseases—A Focus on Human iPSC-Derived Dopaminergic Neurons.
- Published in:
- Cells (2073-4409), 2021, v. 10, n. 12, p. 3436, doi. 10.3390/cells10123436
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- Publication type:
- Article
Dissecting the role of the mitochondrial chaperone mortalin in Parkinson's disease: functional impact of disease-related variants on mitochondrial homeostasis.
- Published in:
- Human Molecular Genetics, 2010, v. 19, n. 22, p. 4437, doi. 10.1093/hmg/ddq370
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- Publication type:
- Article
A patient-based model of RNA mis-splicing uncovers treatment targets in Parkinson's disease.
- Published in:
- Science Translational Medicine, 2020, v. 12, n. 560, p. 1, doi. 10.1126/scitranslmed.aau3960
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- Publication type:
- Article
A modulator of wild-type glucocerebrosidase improves pathogenic phenotypes in dopaminergic neuronal models of Parkinson's disease.
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- Science Translational Medicine, 2019, v. 11, n. 514, p. 1, doi. 10.1126/scitranslmed.aau6870
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- Article
Iron overload is accompanied by mitochondrial and lysosomal dysfunction in WDR45 mutant cells.
- Published in:
- 2018
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- Publication type:
- journal article
Human Neuron Cultures: Micropatterning Facilitates the Long-Term Growth and Analysis of iPSC-Derived Individual Human Neurons and Neuronal Networks (Adv. Healthcare Mater. 15/2016).
- Published in:
- Advanced Healthcare Materials, 2016, v. 5, n. 15, p. 1893, doi. 10.1002/adhm.201670079
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- Publication type:
- Article
Micropatterning Facilitates the Long-Term Growth and Analysis of iPSC-Derived Individual Human Neurons and Neuronal Networks.
- Published in:
- Advanced Healthcare Materials, 2016, v. 5, n. 15, p. 1894, doi. 10.1002/adhm.201500900
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- Publication type:
- Article
Activation of β-Glucocerebrosidase Reduces Pathological α-Synuclein and Restores Lysosomal Function in Parkinson's Patient Midbrain Neurons.
- Published in:
- Journal of Neuroscience, 2016, v. 36, n. 29, p. 7693, doi. 10.1523/JNEUROSCI.0628-16.2016
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- Publication type:
- Article
Knockdown of <i>Hsc70-5/mortalin</i> Induces Loss of Synaptic Mitochondria in a <i>Drosophila</i> Parkinson’s Disease Model.
- Published in:
- PLoS ONE, 2013, v. 8, n. 12, p. 1, doi. 10.1371/journal.pone.0083714
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- Publication type:
- Article
Reduced Basal Autophagy and Impaired Mitochondrial Dynamics Due to Loss of Parkinson's Disease-Associated Protein DJ-1.
- Published in:
- PLoS ONE, 2010, v. 5, n. 2, p. 1, doi. 10.1371/journal.pone.0009367
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- Publication type:
- Article
Balance is the challenge - The impact of mitochondrial dynamics in Parkinson's disease.
- Published in:
- European Journal of Clinical Investigation, 2010, v. 40, n. 11, p. 1048, doi. 10.1111/j.1365-2362.2010.02354.x
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- Publication type:
- Article
Modeling Brain Pathology of Niemann‐Pick Disease Type C Using Patient‐Derived Neurons.
- Published in:
- Movement Disorders, 2021, v. 36, n. 4, p. 1022, doi. 10.1002/mds.28463
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- Publication type:
- Article
Variants in Miro1 Cause Alterations of ER-Mitochondria Contact Sites in Fibroblasts from Parkinson's Disease Patients.
- Published in:
- Journal of Clinical Medicine, 2019, v. 8, n. 12, p. 2226, doi. 10.3390/jcm8122226
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- Publication type:
- Article
Identification of ASCL1 as a determinant for human iPSC-derived dopaminergic neurons.
- Published in:
- Scientific Reports, 2021, v. 11, n. 1, p. 1, doi. 10.1038/s41598-021-01366-4
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- Publication type:
- Article
Acid ceramidase inhibition ameliorates α-synuclein accumulation upon loss of GBA1 function.
- Published in:
- Human Molecular Genetics, 2018, v. 27, n. 11, p. 1972, doi. 10.1093/hmg/ddy105
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- Publication type:
- Article