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Calcitriol increases frataxin levels and restores mitochondrial function in cell models of Friedreich Ataxia.
- Published in:
- Biochemical Journal, 2021, v. 478, n. 1, p. 1, doi. 10.1042/BCJ20200331
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- Article
Calpain-Inhibitors Protect Frataxin-Deficient Dorsal Root Ganglia Neurons from Loss of Mitochondrial Na+/Ca2+ Exchanger, NCLX, and Apoptosis.
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- Neurochemical Research, 2021, v. 46, n. 1, p. 108, doi. 10.1007/s11064-020-03020-3
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- Article
Mitochondrial iron and calcium homeostasis in Friedreich ataxia.
- Published in:
- IUBMB Life, 2021, v. 73, n. 3, p. 543, doi. 10.1002/iub.2457
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- Article
Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models.
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- Cellular & Molecular Life Sciences, 2024, v. 81, n. 1, p. 1, doi. 10.1007/s00018-023-05064-4
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- Article
Mice harboring the FXN I151F pathological point mutation present decreased frataxin levels, a Friedreich ataxia-like phenotype, and mitochondrial alterations.
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- Cellular & Molecular Life Sciences, 2022, v. 79, n. 2, p. 1, doi. 10.1007/s00018-021-04100-5
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- Article
Frataxin‐deficient neurons and mice models of Friedreich ataxia are improved by TAT‐MTScs‐FXN treatment.
- Published in:
- Journal of Cellular & Molecular Medicine, 2018, v. 22, n. 2, p. 834, doi. 10.1111/jcmm.13365
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- Article