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Latent Epstein-Barr virus infection collaborates with Myc over-expression in normal human B cells to induce Burkitt-like Lymphomas in mice.
- Published in:
- PLoS Pathogens, 2024, v. 20, n. 4, p. 1, doi. 10.1371/journal.ppat.1012132
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- Article
Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cells.
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- PLoS Pathogens, 2022, v. 18, n. 4, p. 1, doi. 10.1371/journal.ppat.1010453
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- Article
ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells.
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- PLoS Pathogens, 2021, v. 17, n. 11, p. 1, doi. 10.1371/journal.ppat.1010045
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- Article
Hippo signaling effectors YAP and TAZ induce Epstein-Barr Virus (EBV) lytic reactivation through TEADs in epithelial cells.
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- PLoS Pathogens, 2021, v. 17, n. 8, p. 1, doi. 10.1371/journal.ppat.1009783
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- Article
An EBNA3C-deleted Epstein-Barr virus (EBV) mutant causes B-cell lymphomas with delayed onset in a cord blood-humanized mouse model.
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- PLoS Pathogens, 2018, v. 14, n. 8, p. 1, doi. 10.1371/journal.ppat.1007221
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- Article
A cancer-associated Epstein-Barr virus BZLF1 promoter variant enhances lytic infection.
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- PLoS Pathogens, 2018, v. 14, n. 7, p. 1, doi. 10.1371/journal.ppat.1007179
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- Article
EBNA2-deleted Epstein-Barr virus (EBV) isolate, P3HR1, causes Hodgkin-like lymphomas and diffuse large B cell lymphomas with type II and Wp-restricted latency types in humanized mice.
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- PLoS Pathogens, 2020, v. 16, n. 6, p. 1, doi. 10.1371/journal.ppat.1008590
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- Article
B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infection.
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- PLoS Pathogens, 2020, v. 16, n. 2, p. 1, doi. 10.1371/journal.ppat.1008365
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- Article
Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes.
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- PLoS Pathogens, 2022, v. 18, n. 10, p. 1, doi. 10.1371/journal.ppat.1010868
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- Article