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Cell adhesion molecule CD44 is dispensable for reactive astrocyte activation during prion disease.
- Published in:
- Scientific Reports, 2024, v. 14, n. 1, p. 1, doi. 10.1038/s41598-024-63464-3
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- Article
Microglia deficiency accelerates prion disease but does not enhance prion accumulation in the brain.
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- Glia, 2022, v. 70, n. 11, p. 2169, doi. 10.1002/glia.24244
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- Article
Innate Immune Tolerance in Microglia Does Not Impact on Central Nervous System Prion Disease.
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- Frontiers in Cellular Neuroscience, 2022, v. 16, p. 1, doi. 10.3389/fncel.2022.918883
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- Article
The Effects of Immune System Modulation on Prion Disease Susceptibility and Pathogenesis.
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- International Journal of Molecular Sciences, 2020, v. 21, n. 19, p. 7299, doi. 10.3390/ijms21197299
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- Article
Accelerated onset of CNS prion disease in mice co-infected with a gastrointestinal helminth pathogen during the preclinical phase.
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- Scientific Reports, 2020, v. 10, n. 1, p. 1, doi. 10.1038/s41598-020-61483-4
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- Article
Unaltered intravenous prion disease pathogenesis in the temporary absence of marginal zone B cells.
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- Scientific Reports, 2019, v. 9, n. 1, p. 1, doi. 10.1038/s41598-019-55772-w
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- Article
Discrimination of Prion Strain Targeting in the Central Nervous System via Reactive Astrocyte Heterogeneity in CD44 Expression.
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- Frontiers in Cellular Neuroscience, 2019, p. 1, doi. 10.3389/fncel.2019.00411
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- Article
Effect of co-infection with a small intestine-restricted helminth pathogen on oral prion disease pathogenesis in mice.
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- Scientific Reports, 2019, v. 9, n. 1, p. N.PAG, doi. 10.1038/s41598-019-42900-9
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- Article
Increased susceptibility to oral Trichuris muris infection in the specific absence of CXCR5<sup>+</sup> CD11c<sup>+</sup> cells.
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- Parasite Immunology, 2018, v. 40, n. 8, p. 1, doi. 10.1111/pim.12566
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- Article
Human Prion Diseases and the Risk of Their Transmission During Anatomical Dissection.
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- Clinical Anatomy, 2014, v. 27, n. 6, p. 821, doi. 10.1002/ca.22403
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- Article
Peripheral prion disease pathogenesis is unaltered in the absence of sialoadhesin (Siglec-1/ CD169).
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- Immunology, 2014, v. 143, n. 1, p. 120, doi. 10.1111/imm.12294
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- Article
Prion Disease and the Innate Immune System.
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- Viruses (1999-4915), 2012, v. 4, n. 12, p. 3389, doi. 10.3390/v4123389
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- Article
Follicular Dendritic Cell-Specific Prion Protein (PrP<sup>c</sup>) Expression Alone Is Sufficient to Sustain Prion Infection in the Spleen.
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- PLoS Pathogens, 2011, v. 7, n. 12, p. 1, doi. 10.1371/journal.ppat.1002402
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- Article
Dramatic Reduction of PrP<sup>C</sup> Level and Glycosylation in Peripheral Nerves following PrP Knock-Out from Schwann Cells Does Not Prevent Transmissible Spongiform Encephalopathy Neuroinvasion.
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- Journal of Neuroscience, 2009, v. 29, n. 49, p. 15445, doi. 10.1523/JNEUROSCI.4195-09.2009
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- Article