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Spatial Memory Consolidation is Associated with Induction of Several Lysine-Acetyltransferase (Histone Acetyltransferase) Expression Levels and H2B/H4 Acetylation-Dependent Transcriptional Events in the Rat Hippocampus.
- Published in:
- Neuropsychopharmacology, 2010, v. 35, n. 13, p. 2521, doi. 10.1038/npp.2010.117
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- Publication type:
- Article
Deficiency of the Circadian Clock Gene Bmal1 Reduces Microglial Immunometabolism.
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- Frontiers in Immunology, 2020, v. 11, p. N.PAG, doi. 10.3389/fimmu.2020.586399
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- Publication type:
- Article
Caspase-dependent cleavage of the retinoblastoma protein is an early step in neuronal apoptosis.
- Published in:
- Oncogene, 2000, v. 19, n. 18, p. 2171, doi. 10.1038/sj.onc.1203532
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- Article
The retinoblastoma susceptibility gene product/Sp1 signalling pathway is modulated by Ca<sup>2+</sup>/calmodulin kinases II and IV activity.
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- Oncogene, 1999, v. 18, n. 17, p. 2762, doi. 10.1038/sj.onc.1202634
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- Publication type:
- Article
Pituitary Adenyl Cyclase-Activating Peptide: A Hypophysiotropic Factor That Stimulates Proopiomelanocortin Gene Transcription, and Proopio-melanocortin-Derived Peptide Secretion in Corticotropic Cells.
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- Neuroendocrinology, 1994, v. 60, n. 5, p. 493, doi. 10.1159/000126786
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- Publication type:
- Article
Depolarization regulates cyclin D1 degradation and neuronal apoptosis: a hypothesis about the role of the ubiquitin/proteasome signalling pathway.
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- European Journal of Neuroscience, 1999, v. 11, n. 2, p. 441, doi. 10.1046/j.1460-9568.1999.00451.x
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- Article
Sodium Valproate Exerts Neuroprotective Effects In Vivo through CREB-Binding Protein-Dependent Mechanisms But Does Not Improve Survival in an Amyotrophic Lateral Sclerosis Mouse Model.
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- Journal of Neuroscience, 2007, v. 27, n. 21, p. 5535, doi. 10.1523/JNEUROSCI.1139-07.2007
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- Publication type:
- Article
Selective E2F-dependent gene transcription is controlled by histone deacetylase activity during neuronal apoptosis.
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- Journal of Neurochemistry, 2003, v. 84, n. 4, p. 814, doi. 10.1046/j.1471-4159.2003.01581.x
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- Publication type:
- Article
Brain-Derived Neurotrophic Factor Exerts Opposing Effects on β<sub>2</sub>-Adrenergic Receptor According to Depolarization Status of Cerebellar Neurons.
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- Journal of Neurochemistry, 1999, v. 73, n. 4, p. 1467, doi. 10.1046/j.1471-4159.1999.0731467.x
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- Publication type:
- Article
Detection of Histone Acetylation Levels in the Dorsal Hippocampus Reveals Early Tagging on Specific Residues of H2B and H4 Histones in Response to Learning.
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- PLoS ONE, 2013, v. 8, n. 3, p. 1, doi. 10.1371/journal.pone.0057816
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- Publication type:
- Article
HP1α guides neuronal fate by timing E2F-targeted genes silencing during terminal differentiation.
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- EMBO Journal, 2007, v. 26, n. 15, p. 3616, doi. 10.1038/sj.emboj.7601789
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- Publication type:
- Article
Ventral midline thalamus lesion prevents persistence of new (learning-triggered) hippocampal spines, delayed neocortical spinogenesis, and spatial memory durability.
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- Brain Structure & Function, 2019, v. 224, n. 4, p. 1659, doi. 10.1007/s00429-019-01865-1
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- Article
Altered enhancer transcription underlies Huntington's disease striatal transcriptional signature.
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- Scientific Reports, 2017, p. 42875, doi. 10.1038/srep42875
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- Article
Neuronal identity genes regulated by super-enhancers are preferentially down-regulated in the striatum of Huntington's disease mice.
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- Human Molecular Genetics, 2015, v. 24, n. 12, p. 3481, doi. 10.1093/hmg/ddv099
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- Article
Age-related and disease locus-specific mechanisms contribute to early remodelling of chromatin structure in Huntington's disease mice.
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- Nature Communications, 2021, v. 12, n. 1, p. 1, doi. 10.1038/s41467-020-20605-2
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- Article
A metabolic switch toward lipid use in glycolytic muscle is an early pathologic event in a mouse model of amyotrophic lateral sclerosis.
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- EMBO Molecular Medicine, 2015, v. 7, n. 5, p. 526, doi. 10.15252/emmm.201404433
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- Publication type:
- Article
The CBP KIX domain regulates long-term memory and circadian activity.
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- BMC Biology, 2020, v. 18, n. 1, p. N.PAG, doi. 10.1186/s12915-020-00886-1
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- Article
Brain network remodelling reflects tau-related pathology prior to memory deficits in Thy-Tau22 mice.
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- 2020
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- Publication type:
- journal article
Constitutive Repression of E2F1 Transcriptional Activity through HDAC Proteins Is Essential for Neuronal Survival.
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- Annals of the New York Academy of Sciences, 2002, v. 973, n. 1, p. 438, doi. 10.1111/j.1749-6632.2002.tb04679.x
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- Article
Régulations épigénétiques et plasticitécérébrale : vers de nouvelles thérapies dans les maladies neurodégénératives ?
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- Biologie Aujourd'hui, 2016, v. 210, n. 4, p. 297, doi. 10.1051/jbio/2017002
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- Article
Infectious Agents as Potential Drivers of α‐Synucleinopathies.
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- Movement Disorders, 2022, v. 37, n. 3, p. 464, doi. 10.1002/mds.28925
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- Publication type:
- Article
PCAF-dependent epigenetic changes promote axonal regeneration in the central nervous system.
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- Nature Communications, 2014, v. 5, n. 4, p. 3527, doi. 10.1038/ncomms4527
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- Publication type:
- Article
Late-Life Environmental Enrichment Induces Acetylation Events and Nuclear Factor κB-Dependent Regulations in the Hippocampus of Aged Rats Showing Improved Plasticity and Learning.
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- Journal of Neuroscience, 2016, v. 36, n. 15, p. 4351, doi. 10.1523/JNEUROSCI.3239-15.2016
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- Publication type:
- Article
A Novel Activator of CBP/p300 Acetyltransferases Promotes Neurogenesis and Extends Memory Duration in Adult Mice.
- Published in:
- Journal of Neuroscience, 2013, v. 33, n. 26, p. 10698, doi. 10.1523/JNEUROSCI.5772-12.2013
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- Publication type:
- Article
Transcriptional Activation of the Proopiomelanocortin Gene by Cyclic AMP-responsive Element Binding Protein.
- Published in:
- Pituitary, 1998, v. 1, n. 1, p. 33, doi. 10.1023/A:1009966808106
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- Publication type:
- Article
Lesions and Genes: On the Edge of Improved Isomorphic Models for Alzheimer’s Disease?
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- Neurodegenerative Diseases, 2008, v. 5, n. 5, p. 318, doi. 10.1159/000135616
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- Publication type:
- Article
Sp3 and Sp4 Transcription Factor Levels Are Increased in Brains of Patients with Alzheimer’s Disease.
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- Neurodegenerative Diseases, 2007, v. 4, n. 6, p. 413, doi. 10.1159/000107701
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- Article