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CCR4-dependent reduction in the number and suppressor function of CD4<sup>+</sup>Foxp3<sup>+</sup> cells augments IFN-γ-mediated pulmonary inflammation and aggravates tuberculosis pathogenesis.
- Published in:
- Cell Death & Disease, 2019, v. 10, n. 1, p. 1, doi. 10.1038/s41419-018-1240-3
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- Article
The Severity of COVID-19 Affects the Plasma Soluble Levels of the Immune Checkpoint HLA-G Molecule.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 17, p. 9736, doi. 10.3390/ijms23179736
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- Article
Detrimental Effect of Fungal 60-kDa Heat Shock Protein on Experimental Paracoccidioides brasiliensis Infection.
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- PLoS ONE, 2016, v. 11, n. 9, p. 1, doi. 10.1371/journal.pone.0162486
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- Article
CD11c<sup>+</sup> CD103<sup>+</sup> cells of Mycobacterium tuberculosis-infected C57 BL/6 but not of BALB/c mice induce a high frequency of interferon- γ- or interleukin-17-producing CD4<sup>+</sup> cells.
- Published in:
- Immunology, 2015, v. 144, n. 4, p. 574, doi. 10.1111/imm.12411
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- Article
Inulin prebiotic ameliorates type 1 diabetes dictating regulatory T cell homing via CCR4 to pancreatic islets and butyrogenic gut microbiota in murine model.
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- Journal of Leukocyte Biology, 2024, v. 115, n. 3, p. 483, doi. 10.1093/jleuko/qiad132
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- Article
A DNA vaccine against tuberculosis based on the 65 kDa heat-shock protein differentially activates human macrophages and dendritic cells.
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- Genetic Vaccines & Therapy, 2008, v. 6, p. 1, doi. 10.1186/1479-0556-6-3
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- Article
Comparison of different delivery systems of DNA vaccination for the induction of protection against tuberculosis in mice and guinea pigs.
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- Genetic Vaccines & Therapy, 2007, v. 5, p. 2, doi. 10.1186/1479-0556-5-2
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- Article
NOD2 Deficiency Promotes Intestinal CD4+ T Lymphocyte Imbalance, Metainflammation, and Aggravates Type 2 Diabetes in Murine Model.
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- Frontiers in Immunology, 2020, v. 11, p. 1, doi. 10.3389/fimmu.2020.01265
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- Article
Different bacterial cargo in apoptotic cells drive distinct macrophage phenotypes.
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- Apoptosis, 2024, v. 29, n. 3/4, p. 321, doi. 10.1007/s10495-023-01899-1
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- Article