Found: 7
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Disruption of cytokeratin-8 interaction with F508del-CFTR corrects its functional defect.
- Published in:
- Human Molecular Genetics, 2012, v. 21, n. 3, p. 623, doi. 10.1093/hmg/ddr496
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- Publication type:
- Article
Discovery of novel potent ΔF508- CFTR correctors that target the nucleotide binding domain.
- Published in:
- EMBO Molecular Medicine, 2013, v. 5, n. 10, p. 1484, doi. 10.1002/emmm.201302699
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- Publication type:
- Article
Keratin 8 is a scaffolding and regulatory protein of ERAD complexes.
- Published in:
- Cellular & Molecular Life Sciences, 2022, v. 79, n. 9, p. 1, doi. 10.1007/s00018-022-04528-3
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- Article
Author Correction: New insights into structure and function of bis-phosphinic acid derivatives and implications for CFTR modulation.
- Published in:
- 2021
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- Publication type:
- Correction Notice
New insights into structure and function of bis-phosphinic acid derivatives and implications for CFTR modulation.
- Published in:
- Scientific Reports, 2021, v. 11, n. 1, p. 1, doi. 10.1038/s41598-021-83240-x
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- Publication type:
- Article
Analysis of CLCNKB mutations at dimer‐interface, calcium‐binding site, and pore reveals a variety of functional alterations in ClC‐Kb channel leading to Bartter syndrome.
- Published in:
- Human Mutation, 2020, v. 41, n. 4, p. 774, doi. 10.1002/humu.23962
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- Publication type:
- Article
<italic>Cis</italic> variants identified in F508del complex alleles modulate CFTR channel rescue by small molecules.
- Published in:
- Human Mutation, 2018, v. 39, n. 4, p. 506, doi. 10.1002/humu.23389
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- Publication type:
- Article