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Vitiligo non‐responding lesions to narrow band UVB have intriguing cellular and molecular abnormalities that may prevent epidermal repigmentation.
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- Pigment Cell & Melanoma Research, 2024, v. 37, n. 3, p. 378, doi. 10.1111/pcmr.13160
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- Article
Frontiers in pigment cell and melanoma research.
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- Pigment Cell & Melanoma Research, 2018, v. 31, n. 6, p. 728, doi. 10.1111/pcmr.12728
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Trends in Regenerative Medicine: Repigmentation in Vitiligo Through Melanocyte Stem Cell Mobilization.
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- Medicinal Research Reviews, 2017, v. 37, n. 4, p. 907, doi. 10.1002/med.21426
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Isolating RNA from precursor and mature melanocytes from human vitiligo and normal skin using laser capture microdissection.
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- Experimental Dermatology, 2016, v. 25, n. 10, p. 805, doi. 10.1111/exd.13072
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Narrow Band Ultraviolet B Treatment for Human Vitiligo Is Associated with Proliferation, Migration, and Differentiation of Melanocyte Precursors.
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- Journal of Investigative Dermatology, 2015, v. 135, n. 8, p. 2068, doi. 10.1038/jid.2015.126
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- Article
Epithelial stem cell mutations that promote squamous cell carcinoma metastasis.
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- Journal of Clinical Investigation, 2013, v. 123, n. 10, p. 4390, doi. 10.1172/JCI65856
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Association of Generalized Vitiligo with MHC Class II Loci in Patients from the Indian Subcontinent.
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- Journal of Investigative Dermatology, 2013, v. 133, n. 5, p. 1369, doi. 10.1038/jid.2012.501
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Genome-wide association analyses identify 13 new susceptibility loci for generalized vitiligo.
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- Nature Genetics, 2012, v. 44, n. 6, p. 676, doi. 10.1038/ng.2272
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Genome-Wide Analysis Identifies a Quantitative Trait Locus in the MHC Class II Region Associated with Generalized Vitiligo Age of Onset.
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- Journal of Investigative Dermatology, 2011, v. 131, n. 6, p. 1308, doi. 10.1038/jid.2011.12
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Comprehensive Association Analysis of Candidate Genes for Generalized Vitiligo Supports XBP1, FOXP3, and TSLP.
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- Journal of Investigative Dermatology, 2011, v. 131, n. 2, p. 371, doi. 10.1038/jid.2010.337
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- Article
Common variants in FOXP1 are associated with generalized vitiligo.
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- Nature Genetics, 2010, v. 42, n. 7, p. 576, doi. 10.1038/ng.602
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- Article
Genome-Wide Association Study of Generalized Vitiligo in an Isolated European Founder Population Identifies SMOC2, in Close Proximity to IDDM8.
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- Journal of Investigative Dermatology, 2010, v. 130, n. 3, p. 798, doi. 10.1038/jid.2009.347
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New insights on therapy with vitamin D analogs targeting the intracellular pathways that control repigmentation in human vitiligo.
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- Medicinal Research Reviews, 2009, v. 29, n. 3, p. 514, doi. 10.1002/med.20146
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CTLA4 and generalized vitiligo: two genetic association studies and a meta-analysis of published data.
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- Pigment Cell & Melanoma Research, 2009, v. 22, n. 2, p. 230, doi. 10.1111/j.1755-148X.2009.00543.x
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- Article
The PTPN22-1858C>T (R620W) functional polymorphism is associated with generalized vitiligo in the Romanian population.
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- Pigment Cell & Melanoma Research, 2008, v. 21, n. 2, p. 206, doi. 10.1111/j.1755-148X.2008.00443.x
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- Article
Genetic Variations in NALP1 Are Associated with Generalized Vitiligo in a Romanian Population.
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- Journal of Investigative Dermatology, 2007, v. 127, n. 11, p. 2558, doi. 10.1038/sj.jid.5700953
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Autoimmune Diseases and Vitamin D Receptor Apa-I Polymorphism are Associated with Vitiligo in a Small Inbred Romanian Community.
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- Acta Dermato-Venereologica, 2006, v. 86, n. 3, p. 209, doi. 10.2340/00015555-0093
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What is the mechanism for melasma that so commonly accompanies human pregnancy?
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- IUBMB Life, 2006, v. 58, n. 1, p. 55, doi. 10.1080/15216540500417020
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- Article