Found: 21
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Excision of 5-hydroxymethyluracil and 5-carboxylcytosine by the thymine DNA glycosylase domain: its structural basis and implications for active DNA demethylation.
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- Nucleic Acids Research, 2012, v. 40, n. 20, p. 10203, doi. 10.1093/nar/gks845
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- Article
Efficient deamination of 5-methylcytosines in DNA by human APOBEC3A, but not by AID or APOBEC3G.
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- Nucleic Acids Research, 2012, v. 40, n. 18, p. 9206, doi. 10.1093/nar/gks685
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- Article
Central base pair flipping and discrimination by PspGI.
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- Nucleic Acids Research, 2008, v. 36, n. 19, p. 6109, doi. 10.1093/nar/gkn622
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- Article
DNA base flipping by both members of the PspGI restriction–modification system.
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- Nucleic Acids Research, 2008, v. 36, n. 16, p. 5417, doi. 10.1093/nar/gkn528
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- Article
Sequence-dependent enhancement of hydrolytic deamination of cytosines in DNA by the restriction enzyme PspGI.
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- Nucleic Acids Research, 2006, v. 34, n. 13, p. 3762, doi. 10.1093/nar/gkl545
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- Article
An extended APOBEC3A mutation signature in cancer.
- Published in:
- Nature Communications, 2021, v. 12, n. 1, p. 1, doi. 10.1038/s41467-021-21891-0
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- Article
Importance of the tmRNA system for cell survival when transcription is blocked by DNA-protein cross-links H. K. Kuo, R. Krasich, A. S. Bhagwat and K. N. Kreuzer Transcription blockage and the tmRNA system.
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- Molecular Microbiology, 2010, v. 78, n. 3, p. 686, doi. 10.1111/j.1365-2958.2010.07355.x
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- Article
Cooperation and competition in mismatch repair: very short-patch repair and methyl-directed mismatch repair in Escherichia coli.
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- Molecular Microbiology, 2002, v. 44, n. 6, p. 1421, doi. 10.1046/j.1365-2958.2002.02989.x
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- Article
Escherichia coli DNA glycosylase Mug: a growth-regulated enzyme required for mutation avoidance in stationary-phase cells.
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- Molecular Microbiology, 2001, v. 41, n. 5, p. 1101, doi. 10.1046/j.1365-2958.2001.02559.x
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- Article
Distinguishing preferences of human APOBEC3A and APOBEC3B for cytosines in hairpin loops, and reflection of these preferences in APOBEC-signature cancer genome mutations.
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- Nature Communications, 2024, v. 15, n. 1, p. 1, doi. 10.1038/s41467-024-46231-w
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- Article
Phylogenomic identification of five new human homologs of the DNA repair enzyme AlkB.
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- BMC Genomics, 2003, v. 4, p. 48, doi. 10.1186/1471-2164-4-48
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- Article
Very short patch repair: reducing the cost of cytosine methylation.
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- Molecular Microbiology, 1996, v. 20, n. 3, p. 467, doi. 10.1046/j.1365-2958.1996.5291066.x
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- Article
Mechanism of expression of DNA repair gene vsr, an Escherichia coli gene that overlaps the DNA cytosine methylase gene, dcm.
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- Molecular Microbiology, 1993, v. 9, n. 4, p. 823, doi. 10.1111/j.1365-2958.1993.tb01741.x
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- Article
Human activation‐induced cytidine deaminase causes transcription‐dependent, strand‐biased C to U deaminations.
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- Nucleic Acids Research, 2003, v. 31, n. 12, p. 2990, doi. 10.1093/nar/gkg464
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- Article
A nomenclature for restriction enzymes, DNA methyltransferases, homing endonucleases and their genes.
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- Nucleic Acids Research, 2003, v. 31, n. 7, p. 1805, doi. 10.1093/nar/gkg274
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- Article
Delayed DNA joining at 3′ mismatches by human DNA ligases.
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- Nucleic Acids Research, 1999, v. 27, n. 20, p. 4028, doi. 10.1093/nar/27.20.4028
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- Article
Transcriptional pausing and stalling causes multiple clustered mutations by human activation-induced deaminase.
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- FASEB Journal, 2009, v. 23, n. 1, p. 34, doi. 10.1096/fj.08-115352
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- Article
Visualization of uracils created by APOBEC3A using UdgX shows colocalization with RPA at stalled replication forks.
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- Nucleic Acids Research, 2020, v. 48, n. 20, p. e118, doi. 10.1093/nar/gkaa845
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- Article
Base Damage within Single-Strand DNA Underlies In Vivo Hypermutability Induced by a Ubiquitous Environmental Agent.
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- PLoS Genetics, 2012, v. 8, n. 12, p. 1, doi. 10.1371/journal.pgen.1003149
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- Article
FAM72A degrades UNG2 through the GID/CTLH complex to promote mutagenic repair during antibody maturation.
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- Nature Communications, 2024, v. 15, n. 1, p. 1, doi. 10.1038/s41467-024-52009-x
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- Article
A novel class of chemicals that react with abasic sites in DNA and specifically kill B cell cancers.
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- PLoS ONE, 2017, v. 12, n. 9, p. 1, doi. 10.1371/journal.pone.0185010
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- Article