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Grape Seed Polyphenolic Extract Specifically Decreases Aβ*56 in the Brains of Tg2576 Mice.
- Published in:
- Journal of Alzheimer's Disease, 2011, v. 26, n. 4, p. 657, doi. 10.3233/JAD-2011-110383
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- Article
Molecular basis of memory loss in the Tg2576 mouse model of Alzheimer's disease.
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- Journal of Alzheimer's Disease, 2006, v. 9, n. 3, p. 123, doi. 10.3233/JAD-2006-9S314
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- Publication type:
- Article
Targeting the ensemble of heterogeneous tau oligomers in cells: A novel small molecule screening platform for tauopathies.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2019, v. 15, n. 11, p. 1489, doi. 10.1016/j.jalz.2019.06.4954
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- Article
P3‐195: A TAU CLEAVAGE PRODUCT, ΔTAU314, IS INCREASED IN ALZHEIMER'S DISEASE AND PARKINSON'S DISEASE WITH DEMENTIA.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2018, v. 14, p. P1141, doi. 10.1016/j.jalz.2018.06.1553
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- Article
β-Amyloid oligomers in aging and Alzheimer's disease.
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- Frontiers in Aging Neuroscience, 2013, p. 1, doi. 10.3389/fnagi.2013.00028
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- Publication type:
- Article
Cellular prion protein and class-specific Aβ oligomers mediate plaque-associated cytopathology in a mouse model of Alzheimer’s disease.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2015, v. 11, n. 7, p. P504, doi. 10.1016/j.jalz.2015.06.592
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- Publication type:
- Article
Quantitative comparison of dense-core amyloid plaque burdens in Alzheimer’s disease patients and amyloid precursor protein transgenic mice.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2015, v. 11, n. 7, p. P353, doi. 10.1016/j.jalz.2015.06.236
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- Publication type:
- Article
Extended results of the Alzheimer’s disease anti-inflammatory prevention trial
- Published in:
- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2011, v. 7, n. 4, p. 402, doi. 10.1016/j.jalz.2010.12.014
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- Publication type:
- Article
Grape seed polyphenolic extract specifically decreases Aβ<sup>∗</sup>56 in the brains of Tg2576 mice
- Published in:
- 2010
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- Publication type:
- Abstract
The mislocalization of tau to dendritic spines mediates synaptic dysfunction independent of neurodegeneration
- Published in:
- 2010
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- Publication type:
- Abstract
Transmission of tau-induced cognitive dysfunction
- Published in:
- 2010
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- Publication type:
- Abstract
P1-095: CNS stem cell lines from tau transgenic mice as models for frontotemporal dementia
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- 2008
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- Abstract
P4-370: Long-term survival of human CNS stem cells and their progeny in immunodeficient, plaque-producing APP transgenic mice
- Published in:
- 2006
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- Publication type:
- Abstract
S5-03-03: A specific amyloid-β assembly in the brain impairs memory
- Published in:
- 2006
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- Publication type:
- Abstract
P4-370: Long-term survival of human CNS stem cells and their progeny in immunodeficient, plaque-producing APP transgenic mice
- Published in:
- 2006
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- Publication type:
- Abstract
S5-03-03: A specific amyloid-β assembly in the brain impairs memory
- Published in:
- 2006
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- Publication type:
- Abstract
Characterization of a Novel Mouse Model of Alzheimer’s Disease—Amyloid Pathology and Unique β-Amyloid Oligomer Profile.
- Published in:
- PLoS ONE, 2015, v. 10, n. 5, p. 1, doi. 10.1371/journal.pone.0126317
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- Publication type:
- Article
Genotype-Specific Differences between Mouse CNS Stem Cell Lines Expressing Frontotemporal Dementia Mutant or Wild Type Human Tau.
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- PLoS ONE, 2012, v. 7, n. 6, p. 1, doi. 10.1371/journal.pone.0039328
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- Article
A soluble truncated tau species related to cognitive dysfunction and caspase-2 is elevated in the brain of Huntington's disease patients.
- Published in:
- Acta Neuropathologica Communications, 2019, v. 7, n. 1, p. N.PAG, doi. 10.1186/s40478-019-0764-9
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- Publication type:
- Article
A soluble tau fragment generated by caspase-2 is associated with dementia in Lewy body disease.
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- Acta Neuropathologica Communications, 2019, v. 7, n. 1, p. N.PAG, doi. 10.1186/s40478-019-0765-8
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- Publication type:
- Article
Factors other than hTau overexpression that contribute to tauopathy-like phenotype in rTg4510 mice.
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- Nature Communications, 2019, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41467-019-10428-1
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- Article
In Vivo Imaging Reveals Dissociation between Caspase Activation and Acute Neuronal Death in Tangle-Bearing Neurons.
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- Journal of Neuroscience, 2008, v. 28, n. 4, p. 862, doi. 10.1523/JNEUROSCI.3072-08.2008
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- Publication type:
- Article
Age-Dependent Neurofibrillary Tangle Formation, Neuron Loss, and Memory Impairment in a Mouse Model of Human Tauopathy (P301L).
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- Journal of Neuroscience, 2005, v. 25, n. 46, p. 10637, doi. 10.1523/JNEUROSCI.3279-05.2005
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- Publication type:
- Article
A Cautionary Tale: Endogenous Biotinylated Proteins and Exogenously-Introduced Protein A Cause Antibody-Independent Artefacts in Western Blot Studies of Brain-Derived Proteins.
- Published in:
- Biological Procedures Online, 2019, v. 21, n. 1, p. N.PAG, doi. 10.1186/s12575-019-0095-z
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- Article
Characterization of caspase‐2 inhibitors based on specific sites of caspase‐2‐mediated proteolysis.
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- Archiv der Pharmazie, 2022, v. 355, n. 9, p. 1, doi. 10.1002/ardp.202200095
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- Publication type:
- Article
A specific amyloid-β protein assembly in the brain impairs memory.
- Published in:
- Nature, 2006, v. 440, n. 7082, p. 352, doi. 10.1038/nature04533
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- Article
Correlation of Specific Amyloid-β Oligomers With Tau in Cerebrospinal Fluid From Cognitively Normal Older Adults.
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- JAMA Neurology, 2013, v. 70, n. 5, p. 594, doi. 10.1001/jamaneurol.2013.48
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- Publication type:
- Article
A soluble truncated tau species related to cognitive dysfunction is elevated in the brain of cognitively impaired human individuals.
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- Scientific Reports, 2020, v. 10, n. 1, p. 1, doi. 10.1038/s41598-020-60777-x
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- Article
Brain amyloid-β oligomers in ageing and Alzheimer's disease.
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- 2013
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- Publication type:
- journal article
Brain amyloid-β oligomers in ageing and Alzheimer’s disease.
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- Brain: A Journal of Neurology, 2013, v. 136, n. 5, p. 1383, doi. 10.1093/brain/awt062
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- Publication type:
- Article
Cyclooxygenase-2 inhibition improves amyloid-{beta}-mediated suppression of memory and synaptic plasticity.
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- Brain: A Journal of Neurology, 2008, v. 131, n. 3, p. 651, doi. 10.1093/brain/awn008
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- Article
The Accumulation of Tau in Postsynaptic Structures: A Common Feature in Multiple Neurodegenerative Diseases?
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- Neuroscientist, 2020, v. 26, n. 5/6, p. 503, doi. 10.1177/1073858420916696
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- Article
Phosphorylation in two discrete tau domains regulates a stepwise process leading to postsynaptic dysfunction.
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- Journal of Physiology, 2021, v. 599, n. 9, p. 2483, doi. 10.1113/JP277459
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- Article
Natural oligomers of the amyloid-ßprotein specifically disrupt cognitive function.
- Published in:
- Nature Neuroscience, 2005, v. 8, n. 1, p. 79, doi. 10.1038/nn1372
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- Publication type:
- Article
Blocking Site-Specific Cleavage of Human Tau Delays Progression of Disease-Related Phenotypes in Genetically Matched Tau-Transgenic Mice Modeling Frontotemporal Dementia.
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- Journal of Neuroscience, 2022, v. 42, n. 23, p. 4737, doi. 10.1523/JNEUROSCI.0543-22.2022
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- Publication type:
- Article
Developmental Pathogenicity of 4-Repeat Human Tau Is Lost with the P301L Mutation in Genetically Matched Tau-Transgenic Mice.
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- Journal of Neuroscience, 2020, v. 40, n. 1, p. 220, doi. 10.1523/JNEUROSCI.1256-19.2019
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- Article
Human cerebrospinal fluid 6E10-immunoreactive protein species contain amyloid precursor protein fragments.
- Published in:
- PLoS ONE, 2019, v. 14, n. 2, p. 1, doi. 10.1371/journal.pone.0212815
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- Article