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ELIMINATION DIRECTE DES CELLULES SENESCENTES INDUITES PAR LA THÉRAPIE EN CIBLANT LES PROTÉINES ANTI-APOPTOTIQUES DANS LE CONTEXTE DE DLBCL.
- Published in:
- Lebanese Medical Journal, 2019, v. 67, p. S28
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- Publication type:
- Article
DIRECTED ELIMINATION OF THERAPY-INDUCED SENESCENT CELLS BY TARGETING ANTI-APOPTOTIC PROTEINS IN DLBCL.
- Published in:
- Lebanese Medical Journal, 2019, v. 67, p. S29
- By:
- Publication type:
- Article
DIRECTED ELIMINATION OF THERAPY-INDUCED SENESCENT CELLS BY TARGETING ANTI-APOPTOTIC PROTEINS IN DLBCL.
- Published in:
- Lebanese Medical Journal, 2019, v. 67, n. 1, p. S29
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- Publication type:
- Article
Identification of Lactobacillus strains from human mother milk and cottage cheese revealed potential probiotic properties with enzymatic activity.
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- Scientific Reports, 2022, v. 12, n. 1, p. 1, doi. 10.1038/s41598-022-27003-2
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- Publication type:
- Article
Selective regulation of IP<sub>3</sub>-receptor-mediated Ca<sup>2+</sup> signaling and apoptosis by the BH4 domain of Bcl-2 versus Bcl-Xl.
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- Cell Death & Differentiation, 2012, v. 19, n. 2, p. 295, doi. 10.1038/cdd.2011.97
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- Publication type:
- Article
HTLV-I infection of WE17/10 CD4<sup>+</sup> cell line leads to progressive alteration of Ca<sup>2+</sup> influx that eventually results in loss of CD7 expression and activation of an antiapoptotic pathway involving AKT and BAD which paves the way for malignant transformation
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- 2009
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- Correction Notice
HTLV-I infection of WE17/10 CD4<sup>+</sup> cell line leads to progressive alteration of Ca<sup>2+</sup> influx that eventually results in loss of CD7 expression and activation of an antiapoptotic pathway involving AKT and BAD which paves the way for malignant transformation
- Published in:
- Leukemia (08876924), 2007, v. 21, n. 4, p. 788, doi. 10.1038/sj.leu.2404585
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- Publication type:
- Article
IP<sub>3</sub>R2 levels dictate the apoptotic sensitivity of diffuse large B-cell lymphoma cells to an IP<sub>3</sub>R-derived peptide targeting the BH4 domain of Bcl-2.
- Published in:
- Cell Death & Disease, 2013, v. 4, n. 5, p. 1, doi. 10.1038/cddis.2013.140
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- Publication type:
- Article