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Combinatorial actions of IL-22 and IL-17 drive optimal immunity to oral candidiasis through SPRRs.
- Published in:
- PLoS Pathogens, 2024, v. 0, n. 7, p. 1, doi. 10.1371/journal.ppat.1012302
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- Article
The m<sup>6</sup>A reader IMP2 directs autoimmune inflammation through an IL-17– and TNFα-dependent C/EBP transcription factor axis.
- Published in:
- Science Immunology, 2021, v. 6, n. 61, p. 1, doi. 10.1126/sciimmunol.abd1287
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- Article
Oral epithelial IL-22/STAT3 signaling licenses IL-17–mediated immunity to oral mucosal candidiasis.
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- Science Immunology, 2020, v. 5, n. 48, p. 1, doi. 10.1126/sciimmunol.aba0570
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- Article
The Interleukin (IL) 17R/IL-22R Signaling Axis Is Dispensable for Vulvovaginal Candidiasis Regardless of Estrogen Status.
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- Journal of Infectious Diseases, 2020, v. 221, n. 9, p. 1554, doi. 10.1093/infdis/jiz649
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- Article
IL-17 integrates multiple self-reinforcing, feed-forward mechanisms through the RNA binding protein Arid5a.
- Published in:
- Science Signaling, 2018, v. 11, n. 551, p. 1, doi. 10.1126/scisignal.aat4617
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- Article