Found: 22
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Combat phytopathogenic bacteria employing Argirium-SUNCs: limits and perspectives.
- Published in:
- Applied Microbiology & Biotechnology, 2024, v. 108, n. 1, p. 1, doi. 10.1007/s00253-024-13189-0
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- Article
Silver nanoclusters with Ag<sup>2+/3+</sup> oxidative states are a new highly effective tool against phytopathogenic bacteria.
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- Applied Microbiology & Biotechnology, 2023, v. 107, n. 14, p. 4519, doi. 10.1007/s00253-023-12596-z
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- Article
The membrane depolarization and increase intracellular calcium level produced by silver nanoclusters are responsible for bacterial death.
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- Scientific Reports, 2021, v. 11, n. 1, p. 1, doi. 10.1038/s41598-021-00545-7
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- Article
Structure and Properties of Electrochemically Synthesized Silver Nanoparticles in Aqueous Solution by High-Resolution Techniques.
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- Molecules, 2021, v. 26, n. 17, p. 5155, doi. 10.3390/molecules26175155
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- Article
Antimicrobial and Antibiofilm Activities of New Synthesized Silver Ultra-NanoClusters (SUNCs) Against Helicobacter pylori.
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- Frontiers in Microbiology, 2020, v. 11, p. N.PAG, doi. 10.3389/fmicb.2020.01705
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- Article
Simple Determination of Silver Nanoparticles Concentration as Ag<sup>+</sup> by Using ISE as Potential Alternative to ICP Optical Emission Spectrometry.
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- ChemistrySelect, 2019, v. 4, n. 32, p. 9501, doi. 10.1002/slct.201902336
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- Article
Exploring the Mitochondrial Degradome by the TAILS Proteomics Approach in a Cellular Model of Parkinson's Disease.
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- Frontiers in Aging Neuroscience, 2019, p. 1, doi. 10.3389/fnagi.2019.00195
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- Article
Proteomic Characterization of a New asymmetric Cellulose Triacetate Membrane for Hemodialysis.
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- Proteomics - Clinical Applications, 2018, v. 12, n. 6, p. N.PAG, doi. 10.1002/prca.201700140
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- Article
Electrochemically Synthesized Silver Nanoparticles Are Active Against Planktonic and Biofilm Cells of <italic>Pseudomonas aeruginosa</italic> and Other Cystic Fibrosis-Associated Bacterial Pathogens.
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- Frontiers in Microbiology, 2018, p. N.PAG, doi. 10.3389/fmicb.2018.01349
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- Article
Calcium Binding Promotes Prion Protein Fragment 90- 231 Conformational Change toward a Membrane Destabilizing and Cytotoxic Structure.
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- PLoS ONE, 2012, v. 7, n. 7, p. 1, doi. 10.1371/journal.pone.0038314
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- Article
Efficacy of Novel Acridine Derivatives in the Inhibition of hPrP90-231 Prion Protein Fragment Toxicity.
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- Neurotoxicity Research, 2011, v. 19, n. 4, p. 556, doi. 10.1007/s12640-010-9189-8
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- Article
Dual Modulation of ERK1/2 and p38 MAP Kinase Activities Induced by Minocycline Reverses the Neurotoxic Effects of the Prion Protein Fragment 90–231.
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- Neurotoxicity Research, 2009, v. 15, n. 2, p. 138, doi. 10.1007/s12640-009-9015-3
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- Article
Dual Modulation of ERK1/2 and p38 MAP Kinase Activities Induced by Minocycline Reverses the Neurotoxic Effects of the Prion Protein Fragment 90-231.
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- Neurotoxicity Research, 2009, v. 15, n. 1, p. 1
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- Article
Different structural stability and toxicity of PrP<sup>ARR</sup> and PrP<sup>ARQ</sup> sheep prion protein variants.
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- Journal of Neurochemistry, 2007, v. 103, n. 6, p. 2291, doi. 10.1111/j.1471-4159.2007.04934.x
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- Article
Intracellular accumulation of a mild-denatured monomer of the human PrP fragment 90–231, as possible mechanism of its neurotoxic effects.
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- Journal of Neurochemistry, 2007, v. 103, n. 6, p. 2597, doi. 10.1111/j.1471-4159.2007.04965.x
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- Article
ERK1/2 and p38 MAP kinases control prion protein fragment 90-231-induced astrocyte proliferation and microglia activation.
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- Glia, 2007, v. 55, n. 14, p. 1469, doi. 10.1002/glia.20559
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- Article
Amino-Terminally Truncated Prion Protein PrP90-231 Induces Microglial Activation in Vitro.
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- Annals of the New York Academy of Sciences, 2007, v. 1096, p. 258, doi. 10.1196/annals.1397.092
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- Article
Characterization of the Proapoptotic Intracellular Mechanisms Induced by a Toxic Conformer of the Recombinant Human Prion Protein Fragment 90–231.
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- Annals of the New York Academy of Sciences, 2006, v. 1090, n. 1, p. 276, doi. 10.1196/annals.1378.030
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- Article
P4-151: The intracellular accumulation of the recombinant PRP90-231 fragment is responsible for its cytotoxicity in vitro
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- 2006
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- Abstract
P4-151: The intracellular accumulation of the recombinant PRP90-231 fragment is responsible for its cytotoxicity in vitro
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- 2006
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- Publication type:
- Abstract
Contribution of two conserved glycine residues to fibrillogenesis of the 106–126 prion protein fragment. Evidence that a soluble variant of the 106–126 peptide is neurotoxic.
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- Journal of Neurochemistry, 2003, v. 85, n. 1, p. 62, doi. 10.1046/j.1471-4159.2003.01664.x
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- Article
Structural characterization of human glyoxalase II as probed by limited proteolysis.
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- IUBMB Life, 1998, v. 44, n. 4, p. 761, doi. 10.1080/15216549800201802
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- Article