Works matching AU Konrad, Csaba
Results: 20
Forward operation of adenine nucleotide translocase during F<sub>0</sub>F<sub>1</sub>-ATPase reversal: critical role of matrix substrate-level phosphorylation.
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- FASEB Journal, 2010, v. 24, n. 7, p. 2405, doi. 10.1096/fj.09-149898
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Fibroblast bioenergetics to classify amyotrophic lateral sclerosis patients.
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- Molecular Neurodegeneration, 2017, v. 12, p. 1, doi. 10.1186/s13024-017-0217-5
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Mutant TDP-43 does not impair mitochondrial bioenergetics in vitro and in vivo.
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- Molecular Neurodegeneration, 2017, v. 12, p. 1, doi. 10.1186/s13024-017-0180-1
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Modulation of F.
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- FEBS Journal, 2011, v. 278, n. 7, p. 1112, doi. 10.1111/j.1742-4658.2011.08026.x
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A distinct sequence in the adenine nucleotide translocase from Artemia franciscana embryos is associated with insensitivity to bongkrekate and atypical effects of adenine nucleotides on Ca.
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- FEBS Journal, 2011, v. 278, n. 5, p. 822, doi. 10.1111/j.1742-4658.2010.08001.x
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Better understanding the neurobiology of primary lateral sclerosis.
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- Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration, 2020, v. 21, p. 35, doi. 10.1080/21678421.2020.1837175
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Divalent cation chelators citrate and EDTA unmask an intrinsic uncoupling pathway in isolated mitochondria.
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- Journal of Bioenergetics & Biomembranes, 2017, v. 49, n. 1, p. 3, doi. 10.1007/s10863-016-9656-x
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S1P controls endothelial sphingolipid homeostasis via ORMDL.
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- FASEB Journal, 2022, v. 36, p. N.PAG, doi. 10.1096/fasebj.2022.36.S1.R5883
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Mitochondrial diaphorases as NAD<sup>+</sup> donors to segments of the citric acid cycle that support substrate-level phosphorylation yielding ATP during respiratory inhibition.
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- FASEB Journal, 2014, v. 28, n. 4, p. 1682, doi. 10.1096/fj.13-243030
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The negative impact of α-ketoglutarate dehydrogenase complex deficiency on matrix substrate-level phosphorylation.
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- FASEB Journal, 2013, v. 27, n. 6, p. 2392, doi. 10.1096/fj.12-220202
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Reverse electron transfer results in a loss of flavin from mitochondrial complex I: Potential mechanism for brain ischemia reperfusion injury.
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- Journal of Cerebral Blood Flow & Metabolism, 2017, v. 37, n. 12, p. 3649, doi. 10.1177/0271678X17730242
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Prohibitin S-Nitrosylation Is Required for the Neuroprotective Effect of Nitric Oxide in Neuronal Cultures.
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- Journal of Neuroscience, 2020, v. 40, n. 16, p. 3142, doi. 10.1523/jneurosci.1804-19.2020
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Sphingosine‐1‐phosphate controls endothelial sphingolipid homeostasis via ORMDL.
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- EMBO Reports, 2023, v. 24, n. 1, p. 1, doi. 10.15252/embr.202254689
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Gene expression profiles in sporadic ALS fibroblasts define disease subtypes and the metabolic effects of the investigational drug EH301.
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- Human Molecular Genetics, 2022, v. 31, n. 20, p. 3458, doi. 10.1093/hmg/ddac118
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Deactivation of mitochondrial complex I after hypoxia–ischemia in the immature brain.
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- Journal of Cerebral Blood Flow & Metabolism, 2019, v. 39, n. 9, p. 1790, doi. 10.1177/0271678X18770331
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A re-evaluation of the role of matrix acidification in uncoupler-induced Ca<sup>2+</sup> release from mitochondria.
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- FEBS Journal, 2009, v. 276, n. 10, p. 2713, doi. 10.1111/j.1742-4658.2009.06995.x
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Parkin is a disease modifier in the mutant SOD1 mouse model of ALS.
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- EMBO Molecular Medicine, 2018, v. 10, n. 10, p. N.PAG, doi. 10.15252/emmm.201808888
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The dependence of brain mitochondria reactive oxygen species production on oxygen level is linear, except when inhibited by antimycin A.
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- Journal of Neurochemistry, 2019, v. 148, n. 6, p. 731, doi. 10.1111/jnc.14654
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Absence of Ca<sup>2+</sup>-Induced Mitochondrial Permeability Transition but Presence of Bongkrekate-Sensitive Nucleotide Exchange in C. crangon and P. serratus.
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- PLoS ONE, 2012, v. 7, n. 6, p. 1, doi. 10.1371/journal.pone.0039839
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Modulation of the IGF1R-MTOR pathway attenuates motor neuron toxicity of human ALS SOD1<sup>G93A</sup> astrocytes.
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- Autophagy, 2021, v. 17, n. 12, p. 4029, doi. 10.1080/15548627.2021.1899682
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