Found: 17
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Hereditary Hemochromatosis Predisposes Mice to Yersinia pseudotuberculosis Infection Even in the Absence of the Type III Secretion System.
- Published in:
- Frontiers in Cellular & Infection Microbiology, 2016, v. 6, p. 1, doi. 10.3389/fcimb.2016.00069
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- Article
The cell biology of Listeria monocytogenes infection: the intersection of bacterial pathogenesis and cell-mediated immunity.
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- Journal of Cell Biology, 2002, v. 158, n. 3, p. 409, doi. 10.1083/jcb.200205009
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- Article
Pivotal role of VASP in Arp2/3 complex-mediated actin nucleation, actin branch-formation, and....
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- Journal of Cell Biology, 2001, v. 155, n. 1, p. 89, doi. 10.1083/jcb.200106061
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- Article
Ena/VASP proteins contribute to Listeria monocytogenes pathogenesis by controlling temporal and spatial persistence of bacterial actin-based motility.
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- Molecular Microbiology, 2003, v. 49, n. 5, p. 1361, doi. 10.1046/j.1365-2958.2003.03639.x
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- Article
Y ersinia pseudotuberculosis YopD mutants that genetically separate effector protein translocation from host membrane disruption.
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- Molecular Microbiology, 2015, v. 96, n. 4, p. 764, doi. 10.1111/mmi.12970
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- Article
Microbial pathogenesis and the eukaryotic cytoskeleton at the American Society for Cell Biology annual meeting.
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- Cellular Microbiology, 2002, v. 4, n. 3, p. 187, doi. 10.1046/j.1462-5822.2002.00187.x
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- Article
Stability of the Listeria monocytogenes ActA protein in mammalian cells is regulated by the N-end rule pathway.
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- Cellular Microbiology, 1999, v. 1, n. 3, p. 249, doi. 10.1046/j.1462-5822.1999.00020.x
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- Article
Bacterial Ligands Generated in a Phagosome Are Targets of the Cytosolic Innate Immune System.
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- PLoS Pathogens, 2007, v. 3, n. 3, p. 0431, doi. 10.1371/journal.ppat.0030051
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- Article
An Experimental Pipeline for Initial Characterization of Bacterial Type III Secretion System Inhibitor Mode of Action Using Enteropathogenic Yersinia.
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- Frontiers in Cellular & Infection Microbiology, 2018, v. 8, p. N.PAG, doi. 10.3389/fcimb.2018.00404
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- Article
Control of hmu Heme Uptake Genes in Yersinia pseudotuberculosis in Response to Iron Sources.
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- Frontiers in Cellular & Infection Microbiology, 2018, v. 8, p. 1, doi. 10.3389/fcimb.2018.00047
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- Article
Bacterial iron–sulfur cluster sensors in mammalian pathogens.
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- Metallomics, 2015, v. 7, n. 6, p. 943, doi. 10.1039/c5mt00012b
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- Article
IscR Is Essential for <i>Yersinia pseudotuberculosis</i> Type III Secretion and Virulence.
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- PLoS Pathogens, 2014, v. 10, n. 6, p. 1, doi. 10.1371/journal.ppat.1004194
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- Article
Innate Immune Recognition of Yersinia pseudo tuberculosis Type III Secretion.
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- PLoS Pathogens, 2009, v. 5, n. 12, p. 1, doi. 10.1371/journal.ppat.1000686
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- Article
Bacterial internalization is required to trigger NIK-dependent NF-κB activation in response to the bacterial type three secretion system.
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- PLoS ONE, 2017, v. 12, n. 2, p. 1, doi. 10.1371/journal.pone.0171406
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- Article
Editorial: The Pathogenic Yersiniae–Advances in the Understanding of Physiology and Virulence, Second Edition.
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- 2019
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- Editorial
Iron availability and oxygen tension regulate the Yersinia Ysc type III secretion system to enable disseminated infection.
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- PLoS Pathogens, 2019, v. 15, n. 12, p. N.PAG, doi. 10.1371/journal.ppat.1008001
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- Article
Repression by the H-NS/YmoA histone-like protein complex enables IscR dependent regulation of the Yersinia T3SS.
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- PLoS Genetics, 2022, v. 18, n. 7, p. 1, doi. 10.1371/journal.pgen.1010321
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- Article