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Ethanol and Stress Activate Catecholamine Synthesis in the Adrenal.
- Published in:
- Annals of the New York Academy of Sciences, 2008, v. 1148, p. 542, doi. 10.1196/annals.1410.021
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- Article
Regulation of Gene Expression of Catecholamine Biosynthetic Enzymes in Dopamine-β-Hydroxylase- and CRH-Knockout Mice Exposed to Stress.
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- Annals of the New York Academy of Sciences, 2008, v. 1148, p. 257, doi. 10.1196/annals.1410.033
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- Article
Muscarinic receptors are affected by corticotropin-releasing hormone and c- fos gene disruptions.
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- Journal of Molecular Neuroscience, 2006, v. 30, n. 1/2, p. 69, doi. 10.1385/JMN:30:1:69
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- Article
Localization and Regulation of Phenylethanolamine N-Methyltransferase Gene Expression in the Heart of Rats and Mice during Stress.
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- Annals of the New York Academy of Sciences, 2004, v. 1018, n. 1, p. 405, doi. 10.1196/annals.1296.050
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- Article
Hypertonic Saline and Immobilization Induce Fos Expression in Mouse Brain Catecholaminergic Cell Groups: Colocalization with Tyrosine Hydroxylase and Neuropeptide Y.
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- Annals of the New York Academy of Sciences, 2004, v. 1018, n. 1, p. 398, doi. 10.1196/annals.1296.049
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- Article
Repeated Immobilization Stress Decreases mRNA and Protein Levels of the Type 1 IP<sub>3</sub> Receptor in Rat Heart.
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- Annals of the New York Academy of Sciences, 2004, v. 1018, n. 1, p. 339, doi. 10.1196/annals.1296.042
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- Article
Alcohol Alters Rat Adrenomedullary Function and Stress Response.
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- Annals of the New York Academy of Sciences, 2004, v. 1018, n. 1, p. 173, doi. 10.1196/annals.1296.020
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- Article
Immobilization Stress-Induced Increase in Plasma Catecholamine Levels Is Inhibited by a Prolactoliberin (Salsolinol) Administration.
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- Annals of the New York Academy of Sciences, 2004, v. 1018, n. 1, p. 124, doi. 10.1196/annals.1296.014
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- Article
Social Hierarchy Affects Gene Expression for Catecholamine Biosynthetic Enzymes in Rat Adrenal Glands.
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- Neuroendocrinology, 2004, v. 80, n. 1, p. 42, doi. 10.1159/000080664
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- Article
HSP27 is a partner of JAK2-STAT5 and a potential therapeutic target in myelofibrosis.
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- Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-03627-9
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- Article