Works matching AU Ten, Vadim
Results: 26
Mechanical Ventilation Causes Pulmonary Mitochondrial Dysfunction and Delayed Alveolarization in Neonatal Mice.
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- American Journal of Respiratory Cell & Molecular Biology, 2013, v. 49, n. 6, p. 943, doi. 10.1165/rcmb.2012-0172OC
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- Article
Mitochondrial Dysfunction Contributes to Alveolar Developmental Arrest in Hyperoxia-Exposed Mice.
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- American Journal of Respiratory Cell & Molecular Biology, 2009, v. 40, n. 5, p. 511, doi. 10.1165/rcmb.2008-0341RC
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- Article
Mitochondrial ROS prime the hyperglycemic shift from apoptosis to necroptosis.
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- Cell Death Discovery, 2020, v. 6, n. 1, p. N.PAG, doi. 10.1038/s41420-020-00370-3
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- Article
Mitochondrial Dysfunction and Permeability Transition in Neonatal Brain and Lung Injuries.
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- Cells (2073-4409), 2021, v. 10, n. 3, p. 569, doi. 10.3390/cells10030569
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- Article
The Contribution of Intermittent Hypoxemia to Late Neurological Handicap in Mice with Hyperoxia-Induced Lung Injury.
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- Neonatology (16617800), 2007, v. 92, n. 1, p. 50, doi. 10.1159/000100086
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- Article
DHA but Not EPA Emulsions Preserve Neurological and Mitochondrial Function after Brain Hypoxia-Ischemia in Neonatal Mice.
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- PLoS ONE, 2016, v. 11, n. 8, p. 1, doi. 10.1371/journal.pone.0160870
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- Article
Isoflurane Anesthesia Initiated at the Onset of Reperfusion Attenuates Oxidative and Hypoxic-Ischemic Brain Injury.
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- PLoS ONE, 2015, v. 10, n. 3, p. 1, doi. 10.1371/journal.pone.0120456
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- Article
Isolation of brain mitochondria from neonatal mice.
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- Journal of Neurochemistry, 2011, v. 119, n. 6, p. 1253, doi. 10.1111/j.1471-4159.2011.07525.x
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- Article
Hyperglycemia potentiates a shift from apoptosis to RIP1-dependent necroptosis.
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- Cell Death Discovery, 2018, v. 4, n. 1, p. N.PAG, doi. 10.1038/s41420-018-0058-1
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- Article
Reverse electron transfer results in a loss of flavin from mitochondrial complex I: Potential mechanism for brain ischemia reperfusion injury.
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- Journal of Cerebral Blood Flow & Metabolism, 2017, v. 37, n. 12, p. 3649, doi. 10.1177/0271678X17730242
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- Article
Mild hypoxemia during initial reperfusion alleviates the severity of secondary energy failure and protects brain in neonatal mice with hypoxic-ischemic injury.
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- Journal of Cerebral Blood Flow & Metabolism, 2012, v. 32, n. 2, p. 232, doi. 10.1038/jcbfm.2011.164
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- Article
Temporal pattern of C1q deposition after transient focal cerebral ischemia.
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- Journal of Neuroscience Research, 2006, v. 83, n. 5, p. 883, doi. 10.1002/jnr.20775
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- Article
Cyclophilin D-dependent oligodendrocyte mitochondrial ion leak contributes to neonatal white matter injury.
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- 2020
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- journal article
Cyclophilin D-dependent oligodendrocyte mitochondrial ion leak contributes to neonatal white matter injury.
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- Journal of Clinical Investigation, 2020, v. 130, n. 10, p. 5536, doi. 10.1172/jci133082
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- Article
Acute Injection of Omega-3 Triglyceride Emulsion Provides Very Similar Protection as Hypothermia in a Neonatal Mouse Model of Hypoxic-Ischemic Brain Injury.
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- Frontiers in Neurology, 2021, v. 11, p. N.PAG, doi. 10.3389/fneur.2020.618419
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- Article
Systemic Inflammation following Hind-Limb Ischemia-Reperfusion Affects Brain in Neonatal Mice.
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- Developmental Neuroscience, 2008, v. 30, n. 6, p. 367, doi. 10.1159/000164686
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- Article
An Isolation Method for Assessment of Brain Mitochondria Function in Neonatal Mice with Hypoxic-Ischemic Brain Injury.
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- Developmental Neuroscience, 2008, v. 30, n. 5, p. 319, doi. 10.1159/000121416
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- Article
Hypoxic Stress Exacerbates HyperoxiaInduced Lung Injury in a Neonatal Mouse Model of Bronchopulmonary Dysplasia.
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- Neonatology (16617800), 2008, v. 95, n. 4, p. 299, doi. 10.1159/000178798
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- Article
Mitochondrial calcium buffering depends upon temperature and is associated with hypothermic neuroprotection against hypoxia-ischemia injury.
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- PLoS ONE, 2022, v. 17, n. 8, p. 1, doi. 10.1371/journal.pone.0273677
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- Article
Activation of TNFR1 ectodomain shedding by mitochondrial Ca2+ determines the severity of inflammation in mouse lung microvessels.
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- 2011
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- journal article
Prematurity alters the progenitor cell program of the upper respiratory tract of neonates.
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- Scientific Reports, 2021, v. 11, n. 1, p. 1, doi. 10.1038/s41598-021-90093-x
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- Article
The Oxygen Free Radicals Originating from Mitochondrial Complex I Contribute to Oxidative Brain Injury Following Hypoxia-Ischemia in Neonatal Mice.
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- Journal of Neuroscience, 2012, v. 32, n. 9, p. 3235, doi. 10.1523/JNEUROSCI.6303-11.2012
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- Article
Complement Component C1q Mediates Mitochondria-Driven Oxidative Stress in Neonatal Hypoxic—Ischemic Brain Injury.
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- Journal of Neuroscience, 2010, v. 30, n. 6, p. 2077, doi. 10.1523/JNEUROSCI.5249-09.2010
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- Article
The dependence of brain mitochondria reactive oxygen species production on oxygen level is linear, except when inhibited by antimycin A.
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- Journal of Neurochemistry, 2019, v. 148, n. 6, p. 731, doi. 10.1111/jnc.14654
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- Article
Nelfinavir Inhibits Intra-Mitochondrial Calcium Influx and Protects Brain against Hypoxic-Ischemic Injury in Neonatal Mice.
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- PLoS ONE, 2013, v. 8, n. 4, p. 1, doi. 10.1371/journal.pone.0062448
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- Article
N-3 Fatty Acid Rich Triglyceride Emulsions Are Neuroprotective after Cerebral Hypoxic-Ischemic Injury in Neonatal Mice.
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- PLoS ONE, 2013, v. 8, n. 2, p. 1, doi. 10.1371/journal.pone.0056233
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- Article