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Combinatorial actions of IL-22 and IL-17 drive optimal immunity to oral candidiasis through SPRRs.
Published in:
PLoS Pathogens, 2024, v. 0, n. 7, p. 1, doi. 10.1371/journal.ppat.1012302
By:
- Aggor, Felix E. Y.;
- Bertolini, Martinna;
- Coleman, Bianca M.;
- Taylor, Tiffany C.;
- Ponde, Nicole O.;
- Gaffen, Sarah L.
Publication type:
Article
IL-17 integrates multiple self-reinforcing, feed-forward mechanisms through the RNA binding protein Arid5a.
Published in:
Science Signaling, 2018, v. 11, n. 551, p. 1, doi. 10.1126/scisignal.aat4617
By:
- Amatya, Nilesh;
- Childs, Erin E.;
- Cruz, J. Agustin;
- Aggor, Felix E. Y.;
- Garg, Abhishek V.;
- Berman, Andrea J.;
- Gudjonsson, Johann E.;
- Atasoy, Ulus;
- Gaffen, Sarah L.
Publication type:
Article
The m<sup>6</sup>A reader IMP2 directs autoimmune inflammation through an IL-17– and TNFα-dependent C/EBP transcription factor axis.
Published in:
Science Immunology, 2021, v. 6, n. 61, p. 1, doi. 10.1126/sciimmunol.abd1287
By:
- Bechara, Rami;
- Amatya, Nilesh;
- Bailey, Rachel D.;
- Li, Yang;
- Aggor, Felix E. Y.;
- Li, De-Dong;
- Jawale, Chetan V.;
- Coleman, Bianca M.;
- Dai, Ning;
- Gokhale, Nandan S.;
- Taylor, Tiffany C.;
- Horner, Stacy M.;
- Poholek, Amanda C.;
- Bansal, Anita;
- Biswas, Partha S.;
- Gaffen, Sarah L.
Publication type:
Article
Oral epithelial IL-22/STAT3 signaling licenses IL-17–mediated immunity to oral mucosal candidiasis.
Published in:
Science Immunology, 2020, v. 5, n. 48, p. 1, doi. 10.1126/sciimmunol.aba0570
By:
- Aggor, Felix E. Y.;
- Break, Timothy J.;
- Trevejo-Nuñez, Giraldina;
- Whibley, Natasha;
- Coleman, Bianca M.;
- Bailey, Rachel D.;
- Kaplan, Daniel H.;
- Naglik, Julian R.;
- Shan, Wei;
- Shetty, Amol C.;
- McCracken, Carrie;
- Durum, Scott K.;
- Biswas, Partha S.;
- Bruno, Vincent M.;
- Kolls, Jay K.;
- Lionakis, Michail S.;
- Gaffen, Sarah L.
Publication type:
Article
The Interleukin (IL) 17R/IL-22R Signaling Axis Is Dispensable for Vulvovaginal Candidiasis Regardless of Estrogen Status.
Published in:
Journal of Infectious Diseases, 2020, v. 221, n. 9, p. 1554, doi. 10.1093/infdis/jiz649
By:
- Peters, Brian M.;
- Coleman, Bianca M.;
- Willems, Hubertine M. E.;
- Barker, Katherine S.;
- Aggor, Felix E. Y.;
- Cipolla, Ellyse;
- Verma, Akash H.;
- Bishu, Srinivas;
- Huppler, Anna H.;
- Bruno, Vincent M.;
- Gaffen, Sarah L.
Publication type:
Article

Found: 5

Combinatorial actions of IL-22 and IL-17 drive optimal immunity to oral candidiasis through SPRRs.

IL-17 integrates multiple self-reinforcing, feed-forward mechanisms through the RNA binding protein Arid5a.

The m<sup>6</sup>A reader IMP2 directs autoimmune inflammation through an IL-17– and TNFα-dependent C/EBP transcription factor axis.

Oral epithelial IL-22/STAT3 signaling licenses IL-17–mediated immunity to oral mucosal candidiasis.

The Interleukin (IL) 17R/IL-22R Signaling Axis Is Dispensable for Vulvovaginal Candidiasis Regardless of Estrogen Status.