The article "Author Reply" responds to feedback on a study about in utero aspirin exposure and child neurocognitive development. The study acknowledged limitations in not having information on aspirin dose and duration, but used propensity score-matching and marginal structural models to address potential biases. While the results were consistent with other studies showing a reduced risk of abnormal child neurodevelopment with antenatal aspirin exposure, the authors noted limitations in generalizing the findings due to differences in maternal characteristics and aspirin usage. The authors also discussed potential mechanisms for the observed effects and called for further research to establish causality.