Works matching IS 10966080 AND DT 2014 AND VI 139 AND IP 1
Results: 21
Lead Exposure Disrupts Global DNA Methylation in Human Embryonic Stem Cells and Alters Their Neuronal Differentiation.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 142, doi. 10.1093/toxsci/kfu028
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Fishing for Teratogens: A Consortium Effort for a Harmonized Zebrafish Developmental Toxicology Assay.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 210, doi. 10.1093/toxsci/kfu017
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Testicular Effects Following In Utero Exposure to the Antivirals Acyclovir and Ganciclovir in Rats.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 220, doi. 10.1093/toxsci/kfu024
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Sex Differences in the Acute Nasal Antioxidant/Antielectrophilic Response of the Rat to Inhaled Naphthalene.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 234, doi. 10.1093/toxsci/kfu031
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Comparison of Life-Stage-Dependent Internal Dosimetry for Bisphenol A, Ethinyl Estradiol, a Reference Estrogen, and Endogenous Estradiol to Test an Estrogenic Mode of Action in Sprague Dawley Rats.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 4, doi. 10.1093/toxsci/kfu021
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Integrated Preclinical Photosafety Testing Strategy for Systemically Applied Pharmaceuticals.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 245, doi. 10.1093/toxsci/kfu026
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The Ah Receptor Recruits IKKα to Its Target Binding Motifs to Phosphorylate Serine-10 in Histone H3 Required for Transcriptional Activation.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 121, doi. 10.1093/toxsci/kfu027
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Identification of Hepatic Phospholipidosis Inducers in Sandwich-Cultured Rat Hepatocytes, a Physiologically Relevant Model, Reveals Altered Basolateral Uptake and Biliary Excretion of Anionic Probe Substrates.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 99, doi. 10.1093/toxsci/kfu033
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Comparative Time Course Profiles of Phthalate Stereoisomers in Mice.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 21, doi. 10.1093/toxsci/kfu025
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Halogenated Bisphenol-A Analogs Act as Obesogens in Zebrafish Larvae (Danio rerio).
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 48, doi. 10.1093/toxsci/kfu036
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Are Structural Analogues to Bisphenol A Safe Alternatives?
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 35, doi. 10.1093/toxsci/kfu030
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Catechol-O-Methyltransferase and 3,4-(±)-Methylenedioxymethamphetamine Toxicity.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 162, doi. 10.1093/toxsci/kfu035
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Improving Reproducibility in Toxicology.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 1, doi. 10.1093/toxsci/kfu050
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Integrated Redox Proteomics and Metabolomics of Mitochondria to Identify Mechanisms of Cd Toxicity.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 59, doi. 10.1093/toxsci/kfu018
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Modulation of Mucin mRNA (MUC5AC and MUC5B) Expression and Protein Production and Secretion in Caco-2/HT29-MTX Co-cultures Following Exposure to Individual and Combined Fusarium Mycotoxins.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 83, doi. 10.1093/toxsci/kfu019
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The Thiocarbamate Disulphide Drug, Disulfiram Induces Osteopenia in Rats by Inhibition of Osteoblast Function Due to Suppression of Acetaldehyde Dehydrogenase Activity.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 257, doi. 10.1093/toxsci/kfu020
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Toxicity Evaluation of Bisphenol A Administered by Gavage to Sprague Dawley Rats From Gestation Day 6 Through Postnatal Day 90.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 174, doi. 10.1093/toxsci/kfu022
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AroER Tri-Screen Is a Biologically Relevant Assay for Endocrine Disrupting Chemicals Modulating the Activity of Aromatase and/or the Estrogen Receptor.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 198, doi. 10.1093/toxsci/kfu023
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Cisplatin Plus Sodium Arsenite and Hyperthermia Induces Pseudo-G1 Associated Apoptotic Cell Death in Ovarian Cancer Cells.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 74, doi. 10.1093/toxsci/kfu029
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Andrographolide Sensitizes the Cytotoxicity of Human Colorectal Carcinoma Cells Toward Cisplatin via Enhancing Apoptosis Pathways In Vitro and In Vivo.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 108, doi. 10.1093/toxsci/kfu032
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Tempol Protects Cardiomyocytes from Nucleoside Reverse Transcriptase Inhibitor-Induced Mitochondrial Toxicity.
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- Toxicological Sciences, 2014, v. 139, n. 1, p. 133, doi. 10.1093/toxsci/kfu034
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