Works matching AU TEPIKIN, Alexei V.
Results: 31
Intracellular rupture, exocytosis and actin interaction of endocytic vacuoles in pancreatic acinar cells: initiating events in acute pancreatitis.
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- Journal of Physiology, 2018, v. 596, n. 13, p. 2547, doi. 10.1113/JP275879
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Endoplasmic reticulum-plasma membrane junctions: structure, function and dynamics.
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- Journal of Physiology, 2016, v. 594, n. 11, p. 2837, doi. 10.1113/JP271142
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Transformation of local Ca<sup>2+</sup> spikes to global Ca<sup>2+</sup> transients: the combinatorial roles of multiple Ca<sup>2+</sup> releasing messengers.
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- EMBO Journal, 2002, v. 21, n. 5, p. 909, doi. 10.1093/emboj/21.5.909
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Perinuclear, perigranular and sub-plasmalemmal mitochondria have distinct functions in the regulation of cellular calcium transport.
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- EMBO Journal, 2001, v. 20, n. 8, p. 1863, doi. 10.1093/emboj/20.8.1863
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The endoplasmic reticulum as one continuous Ca<sup>2+</sup> pool: visualization of rapid Ca<sup>2+</sup> movements and equilibration.
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- EMBO Journal, 2000, v. 19, n. 21, p. 5729, doi. 10.1093/emboj/19.21.5729
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Two different but converging messenger pathways to intracellular Ca<sup>2+</sup> release: the roles of nicotinic acid adenine dinucleotide phosphate, cyclic ADP-ribose and inositol trisphosphate.
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- EMBO Journal, 2000, v. 19, n. 11, p. 2549, doi. 10.1093/emboj/19.11.2549
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Active mitochondria surrounding the pancreatic acinar granule region prevent spreading of inositol trisphosphate-evoked local cytosolic Ca<sup>2+</sup> signals.
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- EMBO Journal, 1999, v. 18, n. 18, p. 4999, doi. 10.1093/emboj/18.18.4999
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Termination of cytosolic Ca<sup>2+</sup> signals: Ca<sup>2+</sup> reuptake into intracellular stores is regulated by the free Ca<sup>2+</sup> concentration in the store lumen.
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- EMBO Journal, 1998, v. 17, n. 2, p. 435, doi. 10.1093/emboj/17.2.435
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ATP depletion induces translocation of STIM1 to puncta and formation of STIM1–ORAI1 clusters: translocation and re-translocation of STIM1 does not require ATP.
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- Pflügers Archiv: European Journal of Physiology, 2008, v. 457, n. 2, p. 505, doi. 10.1007/s00424-008-0529-y
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ATP depletion inhibits Ca<sup>2+</sup> release, influx and extrusion in pancreatic acinar cells but not pathological Ca<sup>2+</sup> responses induced by bile.
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- Pflügers Archiv: European Journal of Physiology, 2008, v. 455, n. 6, p. 1025, doi. 10.1007/s00424-007-0360-x
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What can we learn about cell signalling by combining optical imaging and patch clamp techniques?
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- Pflügers Archiv: European Journal of Physiology, 2002, v. 444, n. 3, p. 305, doi. 10.1007/s00424-002-0832-y
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Altered Bioenergetics of Blood Cell Sub-Populations in Acute Pancreatitis Patients.
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- Journal of Clinical Medicine, 2019, v. 8, n. 12, p. 2201, doi. 10.3390/jcm8122201
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Protective Effects of Necrostatin-1 in Acute Pancreatitis: Partial Involvement of Receptor Interacting Protein Kinase 1.
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- Cells (2073-4409), 2021, v. 10, n. 5, p. 1035, doi. 10.3390/cells10051035
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Knockout of the Mitochondrial Calcium Uniporter Strongly Suppresses Stimulus-Metabolism Coupling in Pancreatic Acinar Cells but Does not Reduce Severity of Experimental Acute Pancreatitis.
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- Cells (2073-4409), 2020, v. 9, n. 6, p. 1407, doi. 10.3390/cells9061407
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Calcium-dependent release of NO from intracellular S-nitrosothiols.
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- EMBO Journal, 2006, v. 25, n. 13, p. 3024, doi. 10.1038/sj.emboj.7601207
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Non-uniform distribution of mitochondria in pancreatic acinar cells.
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- Cell & Tissue Research, 2003, v. 313, n. 1, p. 37, doi. 10.1007/s00441-003-0741-1
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Epithelial-mesenchymal transition, IP3 receptors and ER-PM junctions: translocation of Ca<sup>2+</sup> signalling complexes and regulation of migration.
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- Biochemical Journal, 2016, v. 473, n. 6, p. 757, doi. 10.1042/BJ20150364
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The role of Ca<sup>2+</sup> influx in endocytic vacuole formation in pancreatic acinar cells.
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- Biochemical Journal, 2015, v. 465, n. 3, p. 405, doi. 10.1042/BJ20140398
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Saltatory formation, sliding and dissolution of ER-PM junctions in migrating cancer cells.
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- Biochemical Journal, 2013, v. 451, n. 1, p. 25, doi. 10.1042/BJ20121864
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Ca<sup>2+</sup> signalling and bioenergetics of pancreatic diseases.
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- Journal of Cellular Neuroscience & Oxidative Stress, 2018, v. 10, n. 2, p. 669
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Calcium binding capacity of the cytosol and endoplasmic reticulum of mouse pancreatic acinar cells.
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- Journal of Physiology, 1999, v. 518, n. 2, p. 463, doi. 10.1111/j.1469-7793.1999.0463p.x
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Mitochondrial junctions with cellular organelles: Ca<sup>2+</sup> signalling perspective.
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- Pflügers Archiv: European Journal of Physiology, 2018, v. 470, n. 8, p. 1181, doi. 10.1007/s00424-018-2179-z
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Mitochondrial signalling, physiology and pathophysiology.
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- Pflügers Archiv: European Journal of Physiology, 2018, v. 470, n. 8, p. 1139, doi. 10.1007/s00424-018-2172-6
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Traffic of Kv4 K<sup>+</sup> channels mediated by KChlP1 is via a novel post-ER vesicular pathway.
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- Journal of Cell Biology, 2005, v. 171, n. 3, p. 458, doi. 10.1083/jcb.200506005
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NAADP mobilizes Ca[SUP2+] from a thapsigargin-sensitive store in the nuclear envelope by activating ryanodine receptors.
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- Journal of Cell Biology, 2003, v. 163, n. 2, p. 271, doi. 10.1083/jcb.200306134
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Localized Ca[sup2+] uncaging reveals polarized distribution of Ca[sup2+]-sensitive Ca[sup2+] release sites: mechanism of unidirectional Ca[sup2+] waves.
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- Journal of Cell Biology, 2002, v. 158, n. 2, p. 283, doi. 10.1083/jcb.200112025
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F<sub>1</sub>F<sub>0</sub>-ATP Synthase Inhibitory Factor 1 in the Normal Pancreas and in Pancreatic Ductal Adenocarcinoma: Effects on Bioenergetics, Invasion and Proliferation.
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- Frontiers in Physiology, 2018, p. N.PAG, doi. 10.3389/fphys.2018.00833
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Mitochondrial Targeting of Antioxidants Alters Pancreatic Acinar Cell Bioenergetics and Determines Cell Fate.
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- International Journal of Molecular Sciences, 2019, v. 20, n. 7, p. 1700, doi. 10.3390/ijms20071700
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Effects of the Mitochondria-Targeted Antioxidant Mitoquinone in Murine Acute Pancreatitis.
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- Mediators of Inflammation, 2015, v. 2015, p. 1, doi. 10.1155/2015/901780
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Deletion of the WD40 domain of ATG16L1 exacerbates acute pancreatitis, abolishes LAP-like non-canonical autophagy and slows trypsin degradation.
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- Autophagy, 2025, v. 21, n. 1, p. 210, doi. 10.1080/15548627.2024.2392478
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LAP-like non-canonical autophagy and evolution of endocytic vacuoles in pancreatic acinar cells.
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- Autophagy, 2020, v. 16, n. 7, p. 1314, doi. 10.1080/15548627.2019.1679514
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