Works matching AU Takada, Yuzo
Results: 8
Common dysfunctional variants of ABCG2 have stronger impact on hyperuricemia progression than typical environmental risk factors.
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- Scientific Reports, 2014, p. 1, doi. 10.1038/srep05227
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- Article
ABCG2 dysfunction causes hyperuricemia due to both renal urate underexcretion and renal urate overload.
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- Scientific Reports, 2014, p. 1, doi. 10.1038/srep03755
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- Article
Common dysfunctional variants in ABCG2 are a major cause of early-onset gout.
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- Scientific Reports, 2013, p. 1, doi. 10.1038/srep02014
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- Article
Common variants of a urate-associated gene LRP2 are not associated with gout susceptibility.
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- Rheumatology International, 2014, v. 34, n. 4, p. 473, doi. 10.1007/s00296-013-2924-8
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- Article
ABCG2 Dysfunction Increases the Risk of Renal Overload Hyperuricemia.
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- Nucleosides, Nucleotides & Nucleic Acids, 2014, v. 33, n. 4-6, p. 266, doi. 10.1080/15257770.2013.866679
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- Article
A common variant of leucine-rich repeat-containing 16A (LRRC16A) gene is associated with gout susceptibility.
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- Human Cell, 2014, v. 27, n. 2, p. 1, doi. 10.1007/s13577-013-0081-8
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- Article
A common missense variant of monocarboxylate transporter 9 (<i>MCT9/SLC16A9</i>) gene is associated with renal overload gout, but not with all gout susceptibility.
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- Human Cell, 2013, v. 26, n. 4, p. 133, doi. 10.1007/s13577-013-0073-8
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- Article
Common Defects of ABCG2, a High-Capacity Urate Exporter, Cause Gout: A Function-Based Genetic Analysis in a Japanese Population.
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- Science Translational Medicine, 2009, v. 1, n. 5, p. 1, doi. 10.1126/scitranslmed.3000237
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- Article