Found: 17
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Hypoxia-induced alternative splicing: the 11th Hallmark of Cancer.
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- Journal of Experimental & Clinical Cancer Research (17569966), 2020, v. 39, n. 1, p. 1, doi. 10.1186/s13046-020-01616-9
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Discovery, characterization and potential roles of a novel NF-YAx splice variant in human neuroblastoma.
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- Journal of Experimental & Clinical Cancer Research (17569966), 2019, v. 38, n. 1, p. N.PAG, doi. 10.1186/s13046-019-1481-8
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- Article
Doxorubicin-Induced TrkAIII Activation: A Selection Mechanism for Resistant Dormant Neuroblastoma Cells.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 18, p. N.PAG, doi. 10.3390/ijms231810895
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Multidisciplinary Treatment, Including Locoregional Chemotherapy, for Merkel-Polyomavirus-Positive Merkel Cell Carcinomas: Perspectives for Patients Exhibiting Oncogenic Alternative Δ exon 6–7 TrkAIII Splicing of Neurotrophin Receptor Tropomyosin-Related Kinase A
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- International Journal of Molecular Sciences, 2020, v. 21, n. 21, p. 8222, doi. 10.3390/ijms21218222
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Nutraceutical Preventative and Therapeutic Potential in Neuroblastoma: From Pregnancy to Early Childhood.
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- Life (2075-1729), 2022, v. 12, n. 11, p. 1762, doi. 10.3390/life12111762
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- Article
Retinoic acid-enhanced invasion through reconstituted basement membrane by human SK-N-SH neuroblastoma cells involves membrane-associated tissue-type plasminogen activator.
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- International Journal of Cancer, 1997, v. 73, n. 5, p. 740, doi. 10.1002/(SICI)1097-0215(19971127)73:5<740::AID-IJC22>3.0.CO;2-8
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- Article
A pilot study of alternative TrkAIII splicing in Merkel cell carcinoma: a potential oncogenic mechanism and novel therapeutic target.
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- Journal of Experimental & Clinical Cancer Research (17569966), 2019, v. 38, n. 1, p. N.PAG, doi. 10.1186/s13046-019-1425-3
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- Article
The oncogenic neurotrophin receptor tropomyosin-related kinase variant, TrkAIII.
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- Journal of Experimental & Clinical Cancer Research (17569966), 2018, v. 37, n. 1, p. N.PAG, doi. 10.1186/s13046-018-0786-3
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- Article
The TrkAIII Oncoprotein Inhibits Mitochondrial Free Radical ROS-Induced Death of SH-SY5Y Neuroblastoma Cells by Augmenting SOD2 Expression and Activity at the Mitochondria, within the Context of a Tumour Stem Cell-like Phenotype.
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- PLoS ONE, 2014, v. 9, n. 4, p. 1, doi. 10.1371/journal.pone.0094568
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- Article
In vivo analysis of the model tyrosine aminotransferase gene reveals multiple sequential steps in glucocorticoid receptor action.
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- Oncogene, 2001, v. 20, n. 24, p. 3028, doi. 10.1038/sj.onc.1204327
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- Article
A Study of Alternative TrkA Splicing Identifies TrkAIII as a Novel Potentially Targetable Participant in PitNET Progression.
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- Biology (2079-7737), 2024, v. 13, n. 3, p. 171, doi. 10.3390/biology13030171
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- Article
The Alternative TrkAIII Splice Variant, a Targetable Oncogenic Participant in Human Cutaneous Malignant Melanoma.
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- Cells (2073-4409), 2023, v. 12, n. 2, p. 237, doi. 10.3390/cells12020237
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- Article
Constitutive autotaxin transcription by Nmyc-amplified and non-amplified neuroblastoma cells is regulated by a novel AP-1 and SP-mediated mechanism and abrogated by curcumin
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- FEBS Letters, 2012, v. 586, n. 20, p. 3681, doi. 10.1016/j.febslet.2012.08.026
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Alendronate promotes plasmin-mediated MMP-9 inactivation by exposing cryptic plasmin degradation sites within the MMP-9 catalytic domain
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- FEBS Letters, 2012, v. 586, n. 16, p. 2366, doi. 10.1016/j.febslet.2012.05.048
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- Article
Thioredoxin stimulates MMP-9 expression, de-regulates the MMP-9/TIMP-1 equilibrium and promotes MMP-9 dependent invasion in human MDA-MB-231 breast cancer cells
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- FEBS Letters, 2011, v. 585, n. 20, p. 3328, doi. 10.1016/j.febslet.2011.09.023
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Molecular Characterization and Inhibition of a Novel Stress-Induced Mitochondrial Protecting Role for Misfolded TrkAIII in Human SH-SY5Y Neuroblastoma Cells.
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- International Journal of Molecular Sciences, 2024, v. 25, n. 10, p. 5475, doi. 10.3390/ijms25105475
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TrkAIII Promotes Microtubule Nucleation and Assembly at the Centrosome in SH-SY5Y Neuroblastoma Cells, Contributing to an Undifferentiated Anaplastic Phenotype.
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- BioMed Research International, 2013, v. 2013, p. 1, doi. 10.1155/2013/740187
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- Article