Works matching IS 19330219 AND DT 2015 AND VI 8 AND IP 4
Results: 22
Intestinal barrier loss as a critical pathogenic link between inflammatory bowel disease and graft-versus-host disease.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 720, doi. 10.1038/mi.2015.40
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PTPN2 controls differentiation of CD4<sup>+</sup> T cells and limits intestinal inflammation and intestinal dysbiosis.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 918, doi. 10.1038/mi.2014.122
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Altered metabolism of gut microbiota contributes to chronic immune activation in HIV-infected individuals.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 760, doi. 10.1038/mi.2014.107
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Thrombospondin-1 restrains neutrophil granule serine protease function and regulates the innate immune response during Klebsiella pneumoniae infection.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 896, doi. 10.1038/mi.2014.120
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Neutrophils negatively regulate induction of mucosal IgA responses after sublingual immunization.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 735, doi. 10.1038/mi.2014.105
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- Article
IL-1β in eosinophil-mediated small intestinal homeostasis and IgA production.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 930, doi. 10.1038/mi.2014.123
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Distinct Tlr4-expressing cell compartments control neutrophilic and eosinophilic airway inflammation.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 863, doi. 10.1038/mi.2014.117
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Neurotrophic tyrosine kinase receptor 1 is a direct transcriptional and epigenetic target of IL-13 involved in allergic inflammation.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 785, doi. 10.1038/mi.2014.109
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- Article
Bystander immunotherapy as a strategy to control allergen-driven airway inflammation.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 841, doi. 10.1038/mi.2014.115
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Cholera toxin adjuvant promotes a balanced Th1/Th2/Th17 response independently of IL-12 and IL-17 by acting on Gsα in CD11b<sup>+</sup> DCs.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 815, doi. 10.1038/mi.2014.111
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- Article
Commensal enteric bacteria lipopolysaccharide impairs host defense against disseminated Candida albicans fungal infection.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 886, doi. 10.1038/mi.2014.119
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TNFR1-induced lethal inflammation is mediated by goblet and Paneth cell dysfunction.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 828, doi. 10.1038/mi.2014.112
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Divergent behavior of mucosal memory T cells.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 731, doi. 10.1038/mi.2015.52
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Activation of C3a receptor is required in cigarette smoke-mediated emphysema.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 874, doi. 10.1038/mi.2014.118
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New developments in goblet cell mucus secretion and function.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 712, doi. 10.1038/mi.2015.32
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- Article
Intestinal immunopathology is associated with decreased CD73-generated adenosine during lethal infection.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 773, doi. 10.1038/mi.2014.108
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- Article
In situ hematopoiesis: a regulator of T<sub>H</sub>2 cytokine-mediated immunity and inflammation at mucosal surfaces.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 701, doi. 10.1038/mi.2015.17
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Lactoferrin causes IgA and IgG2b isotype switching through betaglycan binding and activation of canonical TGF-β signaling.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 906, doi. 10.1038/mi.2014.121
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CpG DNA assists the whole inactivated H9N2 influenza virus in crossing the intestinal epithelial barriers via transepithelial uptake of dendritic cell dendrites.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 799, doi. 10.1038/mi.2014.110
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Lipoxin B<sub>4</sub> promotes the resolution of allergic inflammation in the upper and lower airways of mice.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 852, doi. 10.1038/mi.2014.116
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Control of pathogenic effector T-cell activities in situ by PD-L1 expression on respiratory inflammatory dendritic cells during respiratory syncytial virus infection.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 746, doi. 10.1038/mi.2014.106
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Intestinal neuroendocrine cells and goblet cells are mediators of IL-17A-amplified epithelial IL-17C production in human inflammatory bowel disease.
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- Mucosal Immunology (1933-0219), 2015, v. 8, n. 4, p. 943, doi. 10.1038/mi.2014.124
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