Works matching IS 00086363 AND DT 2000 AND VI 48 AND IP 1
Results: 20
Myofibroblasts in reperfused myocardial infarcts express the embryonic form of smooth muscle myosin heavy chain (SMemb).
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 89, doi. 10.1016/S0008-6363(00)00158-9
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Cardiac gap junctions: good or bad?
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 8, doi. 10.1016/S0008-6363(00)00189-9
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Myocyte contractile function is intact in the post-infarct remodeled rat heart despite molecular alterations.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 77, doi. 10.1016/S0008-6363(00)00160-7
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Cell-to-cell interaction prevents cell death in cultured neonatal rat ventricular myocytes.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 68, doi. 10.1016/S0008-6363(00)00145-0
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The role of L-type calcium current in the generation of repolarization-induced contraction in cardiac myocytes.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 59, doi. 10.1016/S0008-6363(00)00148-6
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Comparative effects of azimilide and ambasilide on the human ether-a-go-go-related gene (HERG) potassium channel.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 44, doi. 10.1016/S0008-6363(00)00155-3
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Cardiac work and efficiency.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 4, doi. 10.1016/S0008-6363(00)00176-0
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Delayed rectifier K currents have reduced amplitudes and altered kinetics in myocytes from infarcted canine ventricle.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 34, doi. 10.1016/S0008-6363(00)00159-0
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Pathophysiology and treatment of haemodynamic instability in acute pulmonary embolism: the pivotal role of pulmonary vasoconstriction.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 23, doi. 10.1016/S0008-6363(00)00168-1
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Temporal regulation of endothelial ET-1 and eNOS expression in intact human conduit vessels exposed to different intraluminal pressure levels at physiological shear stress.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 168, doi. 10.1016/S0008-6363(00)00174-7
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Blockade of endothelin receptors markedly reduces atherosclerosis in LDL receptor deficient mice: role of endothelin in macrophage foam cell formation.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 158, doi. 10.1016/S0008-6363(00)00169-3
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Growth factor-induced phosphoinositide 3-OH kinase/Akt phosphorylation in smooth muscle cells: induction of cell proliferation and inhibition of cell death.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 148, doi. 10.1016/S0008-6363(00)00152-8
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DNA–RNA chimeric hammerhead ribozyme to transforming growth factor-β1 mRNA inhibits the exaggerated growth of vascular smooth muscle cells from spontaneously hypertensive rats.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 138, doi. 10.1016/S0008-6363(00)00157-7
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Calcium signalling in endothelial cells.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 13, doi. 10.1016/S0008-6363(00)00172-3
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Effects of lipids on the functional and metabolic recovery from global myocardial stunning in isolated rabbit hearts.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 129, doi. 10.1016/S0008-6363(00)00151-6
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Regional electrophysiological effects of left ventricular hypertrophy in isolated rabbit hearts under normal and ischaemic conditions.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 120, doi. 10.1016/S0008-6363(00)00149-8
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Effects of aging on the work output and efficiency of rat papillary muscle.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 111, doi. 10.1016/S0008-6363(00)00144-9
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- Article
Decrease of delayed rectifier currents in the subacute phase of infarction.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 11, doi. 10.1016/S0008-6363(00)00188-7
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Cellular FLIP is expressed in cardiomyocytes and down-regulated in TUNEL-positive grafted cardiac tissues.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 101, doi. 10.1016/S0008-6363(00)00154-1
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Reviewer's recommendations predict impact: why you have to send us your very best work.
- Published in:
- Cardiovascular Research, 2000, v. 48, n. 1, p. 1, doi. 10.1016/S0008-6363(00)00190-5
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