Works matching DE "FRAMINGHAM Heart Study (Organization)"
Results: 102
Bulletin Board.
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- Journal of Women's Health (15409996), 2004, v. 13, n. 5, p. 465, doi. 10.1089/1540999041281061
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Comparison of testosterone fractions between Framingham Heart Study participants and Japanese participants.
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- International Journal of Urology, 2014, v. 21, n. 7, p. 689, doi. 10.1111/iju.12393
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Is Obesity Contagious?
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- JOPERD: The Journal of Physical Education, Recreation & Dance, 2008, v. 79, n. 4, p. 3
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- Article
Classification tree for detection of single-nucleotide polymorphism (SNP)-by-SNP interactions related to heart disease: Framingham Heart Study.
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- BMC Proceedings, 2009, v. 3, p. 1, doi. 10.1186/1753-6561-3-S7-S83
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Copy number variation in the Framingham Heart Study.
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- BMC Proceedings, 2009, v. 3, p. 1, doi. 10.1186/1753-6561-3-S7-S133
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Region-based analysis in genome-wide association study of Framingham Heart Study blood lipid phenotypes.
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- BMC Proceedings, 2009, v. 3, p. 1, doi. 10.1186/1753-6561-3-S7-S127
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Growth mixture modelling in families of the Framingham Heart Study.
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- BMC Proceedings, 2009, v. 3, p. 1, doi. 10.1186/1753-6561-3-S7-S114
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Detection of imprinting and heterogeneous maternal effects on high blood pressure using Framingh am Heart Study data.
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- BMC Proceedings, 2009, v. 3, p. 1, doi. 10.1186/1753-6561-3-S7-S125
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Testing for genetic association taking into account phenotypic information of relatives.
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- BMC Proceedings, 2009, v. 3, p. 1
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A genome-wide association analysis of Framingham Heart Study longitudinal data using multivariate adaptive splines.
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- BMC Proceedings, 2009, v. 3, p. 1, doi. 10.1186/1753-6561-3-S7-S119
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Evaluation of population impact of candidate polymorphisms for coronary heart disease in the Framingham Heart Study Offspring Cohort.
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- BMC Proceedings, 2009, v. 3, p. 1, doi. 10.1186/1753-6561-3-S7-S118
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Longitudinal trends in the association of metabolic syndrome with 550 k single-nucleotide polymorphisms in the Framingham Heart Study.
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- BMC Proceedings, 2009, v. 3, p. 1, doi. 10.1186/1753-6561-3-S7-S116
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Genome-wide association analysis of cardiovascular-related quantitative traits in the Framingham Heart Study.
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- BMC Proceedings, 2009, v. 3, p. 1, doi. 10.1186/1753-6561-3-S7-S117
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- Article
Is obesity contagious?
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- Expert Review of Endocrinology & Metabolism, 2008, v. 3, n. 1, p. 21, doi. 10.1586/17446651.3.1.21
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A three-stage approach for genome-wide association studies with family data for quantitative traits.
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- BMC Genetics, 2010, v. 11, p. 40, doi. 10.1186/1471-2156-11-40
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Quantitative trait linkage analysis of longitudinal change in body weight.
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- BMC Genetics, 2003, v. 4, p. S7, doi. 10.1186/1471-2156-4-S1-S7
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Multivariate sib-pair linkage analysis of longitudinal phenotypes by three step-wise analysis approaches.
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- BMC Genetics, 2003, v. 4, p. S68, doi. 10.1186/1471-2156-4-S1-S68
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Risk factors for coronary artery disease and the use of neural networks to predict the presence or absence of high blood pressure.
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- BMC Genetics, 2003, v. 4, p. S67, doi. 10.1186/1471-2156-4-S1-S67
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Use of tree-based models to identify subgroups and increase power to detect linkage to cardiovascular disease traits.
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- BMC Genetics, 2003, v. 4, p. S66, doi. 10.1186/1471-2156-4-S1-S66
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Screening the genome to detect an association with hypertension.
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- BMC Genetics, 2003, v. 4, p. S63, doi. 10.1186/1471-2156-4-S1-S63
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Bivariate linkage analysis of cholesterol and triglyceride levels in the Framingham Heart Study.
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- BMC Genetics, 2003, v. 4, p. S62, doi. 10.1186/1471-2156-4-S1-S62
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Evidence for bivariate linkage of obesity and HDL-C levels in the Framingham Heart Study.
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- BMC Genetics, 2003, v. 4, p. S52, doi. 10.1186/1471-2156-4-S1-S52
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Susceptibility scoring in family-based association testing.
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- BMC Genetics, 2003, v. 4, p. S49, doi. 10.1186/1471-2156-4-S1-S49
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Transmission ratio distortion in families from the Framingham Heart Study.
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- BMC Genetics, 2003, v. 4, p. S48, doi. 10.1186/1471-2156-4-S1-S48
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Interaction of gender and body mass index (BMI) reveals evidence of linkage for hypertension in the Framingham Heart Study.
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- BMC Genetics, 2003, v. 4, p. S45, doi. 10.1186/1471-2156-4-S1-S45
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Pedigree and genotype errors in the Framingham Heart Study.
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- BMC Genetics, 2003, v. 4, p. S41, doi. 10.1186/1471-2156-4-S1-S41
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Variance components linkage analysis for adjusted systolic blood pressure in the Framingham Heart Study.
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- BMC Genetics, 2003, v. 4, p. S4, doi. 10.1186/1471-2156-4-S1-S4
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Bootstrap calibration of TRANSMIT for informative missingness of parental genotype data.
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- BMC Genetics, 2003, v. 4, p. S39, doi. 10.1186/1471-2156-4-S1-S39
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Stability of exploratory multivariate data modeling in longitudinal data.
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- BMC Genetics, 2003, v. 4, p. S38, doi. 10.1186/1471-2156-4-S1-S38
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Lack of reproducibility of linkage results in serially measured blood pressure data.
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- BMC Genetics, 2003, v. 4, p. S37, doi. 10.1186/1471-2156-4-S1-S37
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Comparison of year-of-exam- and age-matched estimates of heritability in the Framingham Heart Study data.
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- BMC Genetics, 2003, v. 4, p. S36, doi. 10.1186/1471-2156-4-S1-S36
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Genome-wide linkage analysis using cross-sectional and longitudinal traits for body mass index in a subsample of the Framingham Heart Study.
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- BMC Genetics, 2003, v. 4, p. S35, doi. 10.1186/1471-2156-4-S1-S35
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Strategy and model building in the fourth dimension: a null model for genotype x age interaction as a Gaussian stationary stochastic process.
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- BMC Genetics, 2003, v. 4, p. 1
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Age-stratified heritability estimation in the Framingham Heart Study families.
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- BMC Genetics, 2003, v. 4, p. S32, doi. 10.1186/1471-2156-4-S1-S32
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Age-Stratified QTL Genome Scan Analyses for Anthropometric Measures.
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- BMC Genetics, 2003, v. 4, p. S31, doi. 10.1186/1471-2156-4-S1-S31
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Consistency of linkage results across exams and methods in the Framingham Heart Study.
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- BMC Genetics, 2003, v. 4, p. S30, doi. 10.1186/1471-2156-4-S1-S30
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Genetic Analysis Workshop 13: Simulated longitudinal data on families for a system of oligogenic traits.
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- BMC Genetics, 2003, v. 4, p. S3, doi. 10.1186/1471-2156-4-S1-S3
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Power of linkage analysis using traits generated from simulated longitudinal data of the Framingham Heart Study.
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- BMC Genetics, 2003, v. 4, p. S28, doi. 10.1186/1471-2156-4-S1-S28
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Genetic linkage analysis of longitudinal hypertension phenotypes using three summary measures.
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- BMC Genetics, 2003, v. 4, p. S24, doi. 10.1186/1471-2156-4-S1-S24
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Longitudinal variance-components analysis of the Framingham Heart Study data.
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- BMC Genetics, 2003, v. 4, p. S22, doi. 10.1186/1471-2156-4-S1-S22
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Segregation and linkage analysis for longitudinal measurements of a quantitative trait.
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- BMC Genetics, 2003, v. 4, p. S21, doi. 10.1186/1471-2156-4-S1-S21
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Comparison of the linkage results of two phenotypic constructs from longitudinal data in the Framingham Heart Study: analyses on data measured at three time points and on the average of three measurements.
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- BMC Genetics, 2003, v. 4, p. S20, doi. 10.1186/1471-2156-4-S1-S20
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Multilevel modeling for the analysis of longitudinal blood pressure data in the Framingham Heart Study pedigrees.
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- BMC Genetics, 2003, v. 4, p. S19, doi. 10.1186/1471-2156-4-S1-S19
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Empirically derived phenotypic subgroups - qualitative and quantitative trait analyses.
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- BMC Genetics, 2003, v. 4, p. 1
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Genome-wide linkage analysis of longitudinal phenotypes using σ²<sub>A</sub> random effects (SSARs) fitted by Gibbs sampling.
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- BMC Genetics, 2003, v. 4, p. 1
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Genome-wide screen for heavy alcohol consumption.
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- BMC Genetics, 2003, v. 4, p. S106, doi. 10.1186/1471-2156-4-S1-S106
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A genome-wide scan to identify loci for smoking rate in the Framingham Heart Study population.
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- BMC Genetics, 2003, v. 4, p. S103, doi. 10.1186/1471-2156-4-S1-S103
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Multiple genome-wide analyses of smoking behavior in the Framingham Heart Study.
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- BMC Genetics, 2003, v. 4, p. S102, doi. 10.1186/1471-2156-4-S1-S102
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Genomic regions linked to alcohol consumption in the Framingham Heart Study.
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- BMC Genetics, 2003, v. 4, p. S101, doi. 10.1186/1471-2156-4-S1-S101
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Obesity and the Risk of New-Onset Atrial Fibrillation.
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- JAMA: Journal of the American Medical Association, 2004, v. 292, n. 20, p. 2471, doi. 10.1001/jama.292.20.2471
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