Works matching AU Luo, Cheng-Feng
Results: 9
Comprehensive transcriptomics and metabolomics analyses reveal that hyperhomocysteinemia is a high risk factor for coronary artery disease in a chinese obese population aged 40–65: a prospective cross-sectional study.
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- Cardiovascular Diabetology, 2023, v. 22, n. 1, p. 1, doi. 10.1186/s12933-023-01942-0
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- Article
Puerarin-7- O-glucuronide, a water-soluble puerarin metabolite, prevents angiotensin II-induced cardiomyocyte hypertrophy by reducing oxidative stress.
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- Naunyn-Schmiedeberg's Archives of Pharmacology, 2017, v. 390, n. 5, p. 535, doi. 10.1007/s00210-017-1353-8
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- Article
Human UDP-Glucuronosyltransferase 2B4 and 2B7 Are Responsible for Naftopidil Glucuronidation in Vitro.
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- Frontiers in Pharmacology, 2018, p. 1, doi. 10.3389/fphar.2017.00984
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- Article
Impact of Timing following Acute Myocardial Infarction on Efficacy and Safety of Bone Marrow Stem Cells Therapy: A Network Meta-Analysis.
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- Thrombosis, 2015, p. 1, doi. 10.1155/2016/1031794
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- Article
Impact of Timing following Acute Myocardial Infarction on Efficacy and Safety of Bone Marrow Stem Cells Therapy: A Network Meta-Analysis.
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- Stem Cells International, 2015, p. 1, doi. 10.1155/2016/1031794
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- Article
Azilsartan ameliorates ventricular hypertrophy in rats suffering from pressure overload-induced cardiac hypertrophy by activating the Keap1–Nrf2 signalling pathway.
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- Journal of Pharmacy & Pharmacology, 2021, v. 73, n. 12, p. 1715, doi. 10.1093/jpp/rgab097
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- Article
UDP-glucuronosyltransferase 1A1 is the principal enzyme responsible for puerarin metabolism in human liver microsomes.
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- Archives of Toxicology, 2012, v. 86, n. 11, p. 1681, doi. 10.1007/s00204-012-0874-7
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- Article
Erratum to: UDP-glucuronosyltransferase 1A1 is the principal enzyme responsible for puerarin metabolism in human liver microsomes.
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- 2012
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- Correction Notice
Atrial Fibrillation Types and Chronic Kidney Disease are Independent Predictors of Atrial Fibrillation Recurrence After Radiofrequency Ablation.
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- Therapeutics & Clinical Risk Management, 2024, v. 20, p. 817, doi. 10.2147/TCRM.S492265
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- Article