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Systems biology identifies preserved integrity but impaired metabolism of mitochondria due to a glycolytic defect in Alzheimer's disease neurons.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12924
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Tau underlies synaptic and cognitive deficits for type 1, but not type 2 diabetes mouse models.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12919
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Effects of IGF‐1 isoforms on muscle growth and sarcopenia.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12954
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Noncoding variations in Cyp24a1 gene are associated with Klotho‐mediated aging phenotypes in different strains of mice.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12949
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The exceptional longevity of the naked mole‐rat may be explained by mitochondrial antioxidant defenses.
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- Aging Cell, 2019, v. 18, n. 3, p. N.PAG, doi. 10.1111/acel.12916
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Announcement.
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- Aging Cell, 2019, v. 18, n. 3, p. N.PAG, doi. 10.1111/acel.12963
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- Article
Corrigendum.
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- Aging Cell, 2019, v. 18, n. 3, p. N.PAG, doi. 10.1111/acel.12962
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- Article
NMNAT2‐mediated NAD<sup>+</sup> generation is essential for quality control of aged oocytes.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12955
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Glycine supplementation extends lifespan of male and female mice.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12953
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1,25‐Dihydroxyvitamin D exerts an antiaging role by activation of Nrf2‐antioxidant signaling and inactivation of p16/p53‐senescence signaling.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12951
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Targeting senescent cells alleviates obesity‐induced metabolic dysfunction.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12950
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Long‐term caloric restriction ameliorates deleterious effects of aging on white and brown adipose tissue plasticity.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12948
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SLC1A5 glutamine transporter is a target of MYC and mediates reduced mTORC1 signaling and increased fatty acid oxidation in long‐lived Myc hypomorphic mice.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12947
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IL‐1α cleavage by inflammatory caspases of the noncanonical inflammasome controls the senescence‐associated secretory phenotype.
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- Aging Cell, 2019, v. 18, n. 3, p. N.PAG, doi. 10.1111/acel.12946
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Pharmacological clearance of senescent cells improves survival and recovery in aged mice following acute myocardial infarction.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12945
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Hyperadrenocorticism of calorie restriction contributes to its anti‐inflammatory action in mice.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12944
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mTORC1 underlies age‐related muscle fiber damage and loss by inducing oxidative stress and catabolism.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12943
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Cardiolipin remodeling by ALCAT1 links mitochondrial dysfunction to Parkinson's diseases.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12941
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KCNQ1OT1 promotes autophagy by regulating miR‐200a/FOXO3/ATG7 pathway in cerebral ischemic stroke.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12940
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Hormone replacement therapy attenuates hearing loss: Mechanisms involving estrogen and the IGF‐1 pathway.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12939
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Decoy fitness peaks, tumor suppression, and aging.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12938
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Vascular smooth muscle cell‐specific progerin expression in a mouse model of Hutchinson–Gilford progeria syndrome promotes arterial stiffness: Therapeutic effect of dietary nitrite.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12936
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Astrocyte senescence: Evidence and significance.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12937
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NAD<sup>+</sup> supplementation rejuvenates aged gut adult stem cells.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12935
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Tendinosis develops from age‐ and oxygen tension‐dependent modulation of Rac1 activity.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12934
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An early‐senescence state in aged mesenchymal stromal cells contributes to hematopoietic stem and progenitor cell clonogenic impairment through the activation of a pro‐inflammatory program.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12933
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Aged‐senescent cells contribute to impaired heart regeneration.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12931
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Age‐associated cholesterol reduction triggers brain insulin resistance by facilitating ligand‐independent receptor activation and pathway desensitization.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12932
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Tau overexpression impairs neuronal endocytosis by decreasing the GTPase dynamin 1 through the miR‐132/MeCP2 pathway.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12929
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Short‐term starvation stress at young adult stages enhances meiotic activity of germ cells to maintain spermatogenesis in aged male Caenorhabditis elegans.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12930
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Systemic clearance of p16<sup>INK4a</sup>‐positive senescent cells mitigates age‐associated intervertebral disc degeneration.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12927
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Increased intron retention is a post‐transcriptional signature associated with progressive aging and Alzheimer's disease.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12928
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Nontuberculous mycobacterium M. avium infection predisposes aged mice to cardiac abnormalities and inflammation.
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- Aging Cell, 2019, v. 18, n. 3, p. N.PAG, doi. 10.1111/acel.12926
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Elimination of senescent osteoclast progenitors has no effect on the age‐associated loss of bone mass in mice.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12923
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Caloric restriction controls stationary phase survival through Protein Kinase A (PKA) and cytosolic pH.
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- Aging Cell, 2019, v. 18, n. 3, p. N.PAG, doi. 10.1111/acel.12921
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The p53/miRNAs/Ccna2 pathway serves as a novel regulator of cellular senescence: Complement of the canonical p53/p21 pathway.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12918
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The angiotensin‐(1‐7)/Mas receptor axis protects from endothelial cell senescence via klotho and Nrf2 activation.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12913
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Accelerated bio‐cognitive aging in Down syndrome: State of the art and possible deceleration strategies.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12903
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Endothelial toll‐like receptor 4 maintains lung integrity via epigenetic suppression of p16<sup>INK4a</sup>.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12914
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Dynamin‐like protein 1 cleavage by calpain in Alzheimer's disease.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12912
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KIN‐4/MAST kinase promotes PTEN‐mediated longevity of Caenorhabditis elegans via binding through a PDZ domain.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12906
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Genetically heterogeneous mice exhibit a female survival advantage that is age‐ and site‐specific: Results from a large multi‐site study.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12905
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DAF‐16 stabilizes the aging transcriptome and is activated in mid‐aged Caenorhabditis elegans to cope with internal stress.
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- Aging Cell, 2019, v. 18, n. 3, p. N.PAG, doi. 10.1111/acel.12896
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PKR knockout in the 5xFAD model of Alzheimer's disease reveals beneficial effects on spatial memory and brain lesions.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12887
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The leptin sensitizer celastrol reduces age‐associated obesity and modulates behavioral rhythms.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12874
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JNK modifies neuronal metabolism to promote proteostasis and longevity.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12849
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Issue Information.
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- Aging Cell, 2019, v. 18, n. 3, p. N.PAG, doi. 10.1111/acel.12805
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- Article