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The interaction between C5a and both C5a R and C5 L2 receptors is required for production of G- CSF during acute inflammation.
- Published in:
- European Journal of Immunology, 2013, v. 43, n. 7, p. 1907, doi. 10.1002/eji.201243075
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- Article
Bidirectional Crosstalk between C5a Receptors and the NLRP3 Inflammasome in Macrophages and Monocytes.
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- Mediators of Inflammation, 2016, p. 1, doi. 10.1155/2016/1340156
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Functional roles for C5a receptors in sepsis.
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- Nature Medicine, 2008, v. 14, n. 5, p. 551, doi. 10.1038/nm1753
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Generation of C5a in the absence of C3: a new complement activation pathway.
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- Nature Medicine, 2006, v. 12, n. 6, p. 682, doi. 10.1038/nm1419
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- Article
Murine Complement Interactions with Pseudomonas aeruginosa and Their Consequences During Pneumonia.
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- American Journal of Respiratory Cell & Molecular Biology, 2003, v. 29, n. 4, p. 432, doi. 10.1165/rcmb.2002-0145OC
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- Article
Phagocyte-derived catecholamines enhance acute inflammatory injury.
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- Nature, 2007, v. 449, n. 7163, p. 721, doi. 10.1038/nature06185
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Harmful Roles of TLR3 and TLR9 in Cardiac Dysfunction Developing during Polymicrobial Sepsis.
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- BioMed Research International, 2018, v. 2018, p. 1, doi. 10.1155/2018/4302726
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- Article
Complement-induced activation of MAPKs and Akt during sepsis: role in cardiac dysfunction.
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- FASEB Journal, 2017, v. 31, n. 9, p. 4129, doi. 10.1096/fj.201700140R
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- Article
Complement-induced activation of the cardiac NLRP<sub>3</sub> inflammasome in sepsis.
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- FASEB Journal, 2016, v. 30, n. 12, p. 3997, doi. 10.1096/fj.201600728R
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- Article
Role of extracellular histones in the cardiomyopathy of sepsis.
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- FASEB Journal, 2015, v. 29, n. 5, p. 2185, doi. 10.1096/fj.14-268730
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- Article
Extracellular histones are essential effectors of C5aR- and C5L2-mediated tissue damage and inflammation in acute lung injury.
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- FASEB Journal, 2013, v. 27, n. 12, p. 5010, doi. 10.1096/fj.13-236380
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- Article
Anti-inflammatory effects of β<sub>2</sub> adrenergic receptor agonists in experimental acute lung injury.
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- FASEB Journal, 2012, v. 26, n. 5, p. 2137, doi. 10.1096/fj.11-201640
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- Article
Evidence for anti-inflammatory effects of C5a on the innate IL-17A/IL-23 axis.
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- FASEB Journal, 2012, v. 26, n. 4, p. 1640, doi. 10.1096/fj.11-199216
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- Article
MyD88-dependent production of IL-17F is modulated by the anaphylatoxin C5a via the Akt signaling pathway.
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- FASEB Journal, 2011, v. 25, n. 12, p. 4222, doi. 10.1096/fj.11-191205
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- Article
Complement dependency of cardiomyocyte release of mediators during sepsis.
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- FASEB Journal, 2011, v. 25, n. 7, p. 2500, doi. 10.1096/fj.11-183236
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- Article
Attenuation of IgG immune complex-induced acute lung injury by silencing C5aR in lung epithelial cells.
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- FASEB Journal, 2009, v. 23, n. 11, p. 3808, doi. 10.1096/fj.09-133694
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- Article
Functions of the complement components C3 and C5 during sepsis.
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- FASEB Journal, 2008, v. 22, n. 10, p. 3483, doi. 10.1096/fj.08-110595
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Adverse functions of IL-17A in experimental sepsis.
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- FASEB Journal, 2008, v. 22, n. 7, p. 2198, doi. 10.1096/fj.07-105221
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- Article
Mitogen -- Activated Protein Kinases and Septic Cardiomyopathy.
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- FASEB Journal, 2007, v. 21, n. 6, p. A1150, doi. 10.1096/fasebj.21.6.a1150-c
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Evidence for a functional role of the second C5a receptor C5L2.
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- FASEB Journal, 2005, v. 19, n. 8, p. 1003, doi. 10.1096/fj.04-3424fje
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Regulatory role of C5a in LPS-induced IL-6 production by neutrophils during sepsis.
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- FASEB Journal, 2004, v. 18, n. 2, p. 370, doi. 10.1096/fj.03-0708fje
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Neutrophil C5a receptor and the outcome in a rat model of sepsis.
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- FASEB Journal, 2003, v. 17, n. 13, p. 1889, doi. 10.1096/fj.03-0009fje
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Protection of innate immunity by C5aR antagonist in septic mice.
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- FASEB Journal, 2002, v. 16, n. 12, p. 1567, doi. 10.1096/fj.02-0209com
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- Article
Cross-Talk between TLR4 and FcγReceptorIII (CD16) Pathways.
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- PLoS Pathogens, 2009, v. 5, n. 6, p. 1, doi. 10.1371/journal.ppat.1000464
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Selective Biological Responses of Phagocytes and Lungs to Purified Histones.
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- Journal of Innate Immunity, 2017, v. 9, n. 3, p. 300, doi. 10.1159/000452951
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- Article
Induction of M2 Regulatory Macrophages through the β<sub>2</sub>-Adrenergic Receptor with Protection during Endotoxemia and Acute Lung Injury.
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- Journal of Innate Immunity, 2014, v. 6, n. 5, p. 607, doi. 10.1159/000358524
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Persistent Neutrophil Dysfunction and Suppression of Acute Lung Injury in Mice following Cecal Ligation and Puncture Sepsis.
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- Journal of Innate Immunity, 2014, v. 6, n. 5, p. 695, doi. 10.1159/000362554
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The Impact of Extracellular Histones and Absence of Toll-like Receptors on Cardiac Functional and Electrical Disturbances in Mouse Hearts.
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- International Journal of Molecular Sciences, 2024, v. 25, n. 16, p. 8653, doi. 10.3390/ijms25168653
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