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Evaluation of cerebrospinal fluid glycoprotein NMB (GPNMB) as a potential biomarker for Alzheimer's disease.
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- Alzheimer's Research & Therapy, 2021, v. 13, n. 1, p. 1, doi. 10.1186/s13195-021-00828-1
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- Article
Evaluation of cerebrospinal fluid glycoprotein NMB (GPNMB) as a potential biomarker for Alzheimer's disease.
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- Alzheimer's Research & Therapy, 2021, v. 13, n. 1, p. 1, doi. 10.1186/s13195-021-00828-1
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- Article
Evaluation of the putative lymphoma-associated point mutation D427H in the STAT3 transcription factor.
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- BMC Molecular & Cell Biology, 2022, v. 23, n. 1, p. 1, doi. 10.1186/s12860-022-00422-9
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- Article
Axonal degeneration in an Alzheimer mouse model is PS1 gene dose dependent and linked to intraneuronal Aβ accumulation.
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- Frontiers in Aging Neuroscience, 2014, v. 6, p. 1, doi. 10.3389/fnagi.2014.00139
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- Article
N-truncated Aβ<sub>4-x</sub> peptides in sporadic Alzheimer's disease cases and transgenic Alzheimer mouse models.
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- Alzheimer's Research & Therapy, 2017, v. 9, p. 1, doi. 10.1186/s13195-017-0309-z
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- Article
Blood plasma biomarkers for Alzheimer's disease: Aβ1–42/1–40 vs. AβX–42/X–40.
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- Clinical Chemistry & Laboratory Medicine, 2024, v. 62, n. 2, p. e56, doi. 10.1515/cclm-2023-0990
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- Article
Blood plasma biomarkers for Alzheimer's disease: Aβ1–42/1–40 vs. AβX–42/X–40.
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- Clinical Chemistry & Laboratory Medicine, 2024, v. 62, n. 2, p. e56, doi. 10.1515/cclm-2023-0990
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- Article
A Combination of Caffeine Supplementation and Enriched Environment in an Alzheimer's Disease Mouse Model.
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- International Journal of Molecular Sciences, 2023, v. 24, n. 3, p. 2155, doi. 10.3390/ijms24032155
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- Article
Characterization of a Mouse Model of Alzheimer's Disease Expressing Aβ4-42 and Human Mutant Tau.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 10, p. 5191, doi. 10.3390/ijms22105191
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- Article
Neuron Loss in Alzheimer's Disease: Translation in Transgenic Mouse Models.
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- International Journal of Molecular Sciences, 2020, v. 21, n. 21, p. 8144, doi. 10.3390/ijms21218144
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- Article
Development and Technical Validation of an Immunoassay for the Detection of APP669–711 (Aβ−3–40) in Biological Samples.
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- International Journal of Molecular Sciences, 2020, v. 21, n. 18, p. 6564, doi. 10.3390/ijms21186564
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- Article
Chronic Memantine Treatment Ameliorates Behavioral Deficits, Neuron Loss, and Impaired Neurogenesis in a Model of Alzheimer's Disease.
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- Molecular Neurobiology, 2021, v. 58, n. 1, p. 204, doi. 10.1007/s12035-020-02120-z
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- Article
Limited Effects of Prolonged Environmental Enrichment on the Pathology of 5XFAD Mice.
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- Molecular Neurobiology, 2017, v. 54, n. 8, p. 6542, doi. 10.1007/s12035-016-0167-x
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- Article
Endogenous Apolipoprotein E (ApoE) Fragmentation Is Linked to Amyloid Pathology in Transgenic Mouse Models of Alzheimer's Disease.
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- Molecular Neurobiology, 2017, v. 54, n. 1, p. 319, doi. 10.1007/s12035-015-9674-4
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- Article
The metalloprotease ADAMTS4 generates N-truncated Aβ4-x species and marks oligodendrocytes as a source of amyloidogenic peptides in Alzheimer's disease.
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- Acta Neuropathologica, 2019, v. 137, n. 2, p. 239, doi. 10.1007/s00401-018-1929-5
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- Article
Phosphorylation of the amyloid β-peptide at Ser26 stabilizes oligomeric assembly and increases neurotoxicity.
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- Acta Neuropathologica, 2016, v. 131, n. 4, p. 525, doi. 10.1007/s00401-016-1546-0
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- Article
Focusing the amyloid cascade hypothesis on N-truncated Abeta peptides as drug targets against Alzheimer's disease.
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- Acta Neuropathologica, 2014, v. 127, n. 6, p. 787, doi. 10.1007/s00401-014-1287-x
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- Article
N-truncated amyloid β (Aβ) 4-42 forms stable aggregates and induces acute and long-lasting behavioral deficits.
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- Acta Neuropathologica, 2013, v. 126, n. 2, p. 189, doi. 10.1007/s00401-013-1129-2
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- Article
Early intraneuronal accumulation and increased aggregation of phosphorylated Abeta in a mouse model of Alzheimer's disease.
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- Acta Neuropathologica, 2013, v. 125, n. 5, p. 699, doi. 10.1007/s00401-013-1107-8
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- Article
Accumulation of intraneuronal Aβ correlates with ApoE4 genotype.
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- Acta Neuropathologica, 2010, v. 119, n. 5, p. 555, doi. 10.1007/s00401-010-0666-1
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- Article
Intraneuronal pyroglutamate-Abeta 3–42 triggers neurodegeneration and lethal neurological deficits in a transgenic mouse model.
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- Acta Neuropathologica, 2009, v. 118, n. 4, p. 487, doi. 10.1007/s00401-009-0557-5
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- Article
APP/PS1KI bigenic mice develop early synaptic deficits and hippocampus atrophy.
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- Acta Neuropathologica, 2009, v. 117, n. 6, p. 677, doi. 10.1007/s00401-009-0539-7
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- Article
Transient intraneuronal Aβ rather than extracellular plaque pathology correlates with neuron loss in the frontal cortex of APP/PS1KI mice.
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- Acta Neuropathologica, 2008, v. 116, n. 6, p. 647, doi. 10.1007/s00401-008-0451-6
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- Article
Axonopathy in an APP/PS1 transgenic mouse model of Alzheimer’s disease.
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- Acta Neuropathologica, 2006, v. 111, n. 4, p. 312, doi. 10.1007/s00401-006-0041-4
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- Article
Deposition of C-terminally truncated Aβ species Aβ37 and Aβ39 in Alzheimer's disease and transgenic mouse models.
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- Acta Neuropathologica Communications, 2016, v. 4, p. 1, doi. 10.1186/s40478-016-0294-7
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- Article
A microRNA signature that correlates with cognition and is a target against cognitive decline.
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- EMBO Molecular Medicine, 2021, v. 13, n. 11, p. 1, doi. 10.15252/emmm.202013659
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- Article
Abundance of Aβ<sub>5-x</sub> like immunoreactivity in transgenic 5XFAD, APP/PS1KI and 3xTG mice, sporadic and familial Alzheimer's disease.
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- Molecular Neurodegeneration, 2014, v. 9, n. 1, p. 1, doi. 10.1186/1750-1326-9-13
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- Article
Intraneuronal β-Amyloid Is a Major Risk Factor – Novel Evidence from the APP/PS1KI Mouse Model.
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- Neurodegenerative Diseases, 2008, v. 5, n. 3/4, p. 140, doi. 10.1159/000113684
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- Article
A modifiedβ-amyloid hypothesis: intraneuronal accumulation of theβ-amyloid peptide– the first step of a fatal cascade.
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- Journal of Neurochemistry, 2004, v. 91, n. 3, p. 513, doi. 10.1111/j.1471-4159.2004.02737.x
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- Article
An inhibitory effect on the nuclear accumulation of phospho-STAT1 by its unphosphorylated form.
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- Cell Communication & Signaling, 2022, v. 20, n. 1, p. 1, doi. 10.1186/s12964-022-00841-3
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- Article
Loss of Hippocampal Calretinin and Parvalbumin Interneurons in the 5XFAD Mouse Model of Alzheimer's Disease.
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- ASN Neuro (Sage Publications, Ltd.), 2020, v. 12, p. 1, doi. 10.1177/1759091420925356
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- Article
Loss of Hippocampal Calretinin and Parvalbumin Interneurons in the 5XFAD Mouse Model of Alzheimer's Disease.
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- ASN Neuro (Sage Publications, Ltd.), 2020, v. 12, p. 1, doi. 10.1177/1759091420925356
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- Article
Physical Activity Ameliorates Impaired Hippocampal Neurogenesis in the Tg4-42 Mouse Model of Alzheimer's Disease.
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- ASN Neuro (Sage Publications, Ltd.), 2019, v. 11, p. N.PAG, doi. 10.1177/1759091419892692
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- Article
Combined long-term enriched environment and caffeine supplementation improve memory function in C57Bl6 mice.
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- European Archives of Psychiatry & Clinical Neuroscience, 2023, v. 273, n. 1, p. 269, doi. 10.1007/s00406-022-01431-7
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- Article
The Cannabinoid CB1/CB2 Agonist WIN55212.2 Promotes Oligodendrocyte Differentiation In Vitro and Neuroprotection During the Cuprizone-Induced Central Nervous System Demyelination.
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- CNS Neuroscience & Therapeutics, 2016, v. 22, n. 5, p. 387, doi. 10.1111/cns.12506
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- Article
Die modifizierte Amyloid-Hypothese der Alzheimer-Demenz - intraneuronales Abeta induziert Neurodegeneration.
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- Neuroforum, 2009, v. 15, n. 3, p. 76, doi. 10.1515/nf-2009-0302
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Intracellular Aβ and neuronal death
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- 2011
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- Abstract
Treatment of 5XFAD transgenic mice with Ibuprofen
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- 2011
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- Abstract
P1-038: Inflammatory changes are tightly associated with neurodegeneration in the brain and spinal cord of the APP/PS1KI mouse model of Alzheimer's disease
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- 2008
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- Abstract
P1-026: Intracellular Aβ triggers neuron loss in the cholinergic system of the APP/PS1KI mouse model of Alzheimer's disease
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- 2008
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- Abstract
O3-01-02: Effect of enriched environmental on motor an memory functions in the APP/PS1KI mouse model of Alzheimer's disease
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- 2008
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- Abstract
P1-042: Neuronal loss and changes in metal homeostasis in the APP751<sup>SL</sup>PS1ki mouse model of Alzheimer’s disease
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- 2006
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- Abstract
P1-028: Axonopathy in APP/PS1 transgenic mouse models of Alzheimer’s disease
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- 2006
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- Abstract
O2-04-06: New lessons form the APP/PS1 transgenic mouse model with neuron loss: Metal biology, axonal degeneration and intraneuronal N-modified Aβ aggregation
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- 2006
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- Abstract
P1-042: Neuronal loss and changes in metal homeostasis in the APP751<sup>SL</sup>PS1ki mouse model of Alzheimer’s disease
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- 2006
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- Publication type:
- Abstract
P1-028: Axonopathy in APP/PS1 transgenic mouse models of Alzheimer’s disease
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- 2006
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- Publication type:
- Abstract
O2-04-06: New lessons form the APP/PS1 transgenic mouse model with neuron loss: Metal biology, axonal degeneration and intraneuronal N-modified Aβ aggregation
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- 2006
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- Abstract
Synergistic Effect on Neurodegeneration by N-Truncated Aβ<sub>4-42</sub> and Pyroglutamate Aβ<sub>3-42</sub> in a Mouse Model of Alzheimer's Disease.
- Published in:
- Frontiers in Aging Neuroscience, 2018, p. 1, doi. 10.3389/fnagi.2018.00064
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- Article
Neuron Loss in Transgenic Mouse Models of Alzheimer's Disease.
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- International Journal of Alzheimer's Disease, 2010, v. 2010, p. 1, doi. 10.4061/2010/723782
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- Article
Amino‐terminally elongated Aβ peptides are generated by the secreted metalloprotease ADAMTS4 and deposit in a subset of Alzheimer's disease brains.
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- Neuropathology & Applied Neurobiology, 2024, v. 50, n. 3, p. 1, doi. 10.1111/nan.12991
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- Article