Found: 7
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Gender, Neuroendocrine-Immune Interactions and Neuron-Glial Plasticity: Role of Luteinizing Hormone-Releasing Hormone (LHRH).
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- Annals of the New York Academy of Sciences, 2000, v. 917, n. 1, p. 678, doi. 10.1111/j.1749-6632.2000.tb05434.x
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- Article
Luteinizing Hormone-Releasing Hormone Is a Primary Signaling Molecule in the Neuroimmune Network.
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- Annals of the New York Academy of Sciences, 1998, v. 840, n. 1, p. 205, doi. 10.1111/j.1749-6632.1998.tb09564.x
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- Article
Hormones Are Key Actors in Gene X Environment Interactions Programming the Vulnerability to Parkinson's Disease: Glia as a Common Final Pathway.
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- Annals of the New York Academy of Sciences, 2005, v. 1057, n. 1, p. 296, doi. 10.1196/annals.1356.023
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- Article
Basic fibroblast growth factor (bFGF) acts on both neurons and glia to mediate the neurotrophic effects of astrocytes on LHRH neurons in culture.
- Published in:
- Synapse, 2000, v. 36, n. 4, p. 233, doi. 10.1002/(SICI)1098-2396(20000615)36:4<233::AID-SYN1>3.0.CO;2-I
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- Article
Uncovering novel actors in astrocyte-neuron crosstalk in Parkinson's disease: the Wnt/β-catenin signaling cascade as the common final pathway for neuroprotection and self-repair.
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- European Journal of Neuroscience, 2013, v. 37, n. 10, p. 1550, doi. 10.1111/ejn.12166
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- Article
Glucocorticoid receptor deficiency increases vulnerability of the nigrostriatal dopaminergic system: critical role of glial nitric oxide.
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- FASEB Journal, 2004, v. 18, n. 1, p. 164, doi. 10.1096/fj.03-0501fje
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- Article
Targeting Wnt signaling at the neuroimmune interface for dopaminergic neuroprotection/repair in Parkinson's disease.
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- Journal of Molecular Cell Biology, 2014, v. 6, n. 1, p. 13, doi. 10.1093/jmcb/mjt053
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- Article