Found: 8
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Cellular Pathophysiology of Mutant Voltage-Dependent Ca<sup>2+</sup> Channel CACNA1H in Primary Aldosteronism.
- Published in:
- Endocrinology, 2020, v. 161, n. 10, p. 1, doi. 10.1210/endocr/bqaa135
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- Publication type:
- Article
Activation of endogenously expressed ion channels by active complement in the retinal pigment epithelium.
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- Pflügers Archiv: European Journal of Physiology, 2015, v. 467, n. 10, p. 2179, doi. 10.1007/s00424-014-1656-2
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- Publication type:
- Article
A POST-MARKET ANALYSIS OF SAFETY DATA IN PEDIATRIC PATIENTS TREATED WITH TTFIELDS (2015-2021).
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- Oncology Nursing Forum, 2022, v. 49, n. 2, p. E58, doi. 10.1188/22.ONF.E2
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- Publication type:
- Article
The safety profile of Tumor Treating Fields (TTFields) therapy in glioblastoma patients with ventriculoperitoneal shunts.
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- Journal of Neuro-Oncology, 2022, v. 158, n. 3, p. 453, doi. 10.1007/s11060-022-04033-4
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- Publication type:
- Article
Oxytocin Stimulates Extracellular Ca<sup>2+</sup> Influx Through TRPV2 Channels in Hypothalamic Neurons to Exert Its Anxiolytic Effects.
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- Neuropsychopharmacology, 2015, v. 40, n. 13, p. 2938, doi. 10.1038/npp.2015.147
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- Publication type:
- Article
Angiotensin-2-Mediated Ca<sup>2+</sup> Signaling in the Retinal Pigment Epithelium: Role of Angiotensin-Receptor-Associated-Protein and TRPV2 Channel.
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- PLoS ONE, 2012, v. 7, n. 11, p. 1, doi. 10.1371/journal.pone.0049624
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- Publication type:
- Article
A case of severe hyperaldosteronism caused by a de novo mutation affecting a critical salt bridge Kir3.4 residue.
- Published in:
- 2015
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- Publication type:
- journal article
A novel KCNJ5-insT149 somatic mutation close to, but outside, the selectivity filter causes resistant hypertension by loss of selectivity for potassium.
- Published in:
- 2014
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- Publication type:
- journal article