Found: 21
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Live or Die: Choice Mechanisms in Stressed Cells.
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- Mediators of Inflammation, 2015, v. 2015, p. 1, doi. 10.1155/2015/454863
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- Article
Mast cells are required for angiogenesis and macroscopic expansion of Myc-induced pancreatic islet tumors.
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- Nature Medicine, 2007, v. 13, n. 10, p. 1211, doi. 10.1038/nm1649
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- Article
Integrated requirement of non‐specific and sequence‐specific DNA binding in Myc‐driven transcription.
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- EMBO Journal, 2021, v. 40, n. 10, p. 1, doi. 10.15252/embj.2020105464
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- Article
Temporal dissection of p53 function in vitro and in vivo.
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- Nature Genetics, 2005, v. 37, n. 7, p. 718, doi. 10.1038/ng1572
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- Article
The Action Mechanism of the Myc Inhibitor Termed Omomyc May Give Clues on How to Target Myc for Cancer Therapy.
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- PLoS ONE, 2011, v. 6, n. 7, p. 1, doi. 10.1371/journal.pone.0022284
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- Article
Stretching the Limits of Bicyclic Peptides: A Novel Approach to MYC Inhibition and Beyond.
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- GEN Biotechnology, 2024, v. 3, n. 1, p. 23, doi. 10.1089/genbio.2024.0003
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- Article
c-Myc recruits P-TEFb for transcription, cellular proliferation and apoptosis.
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- Oncogene, 2003, v. 22, n. 36, p. 5707, doi. 10.1038/sj.onc.1206800
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- Article
Pharmacokinetic Analysis of Omomyc Shows Lasting Structural Integrity and Long Terminal Half-Life in Tumor Tissue.
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- Cancers, 2023, v. 15, n. 3, p. 826, doi. 10.3390/cancers15030826
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- Article
Modelling Myc inhibition as a cancer therapy.
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- Nature, 2008, v. 455, n. 7213, p. 679, doi. 10.1038/nature07260
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- Article
The Wnt signaling receptor Fzd9 is essential for Myc-driven tumorigenesis in pancreatic islets.
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- Life Science Alliance, 2021, v. 4, n. 5, p. 1, doi. 10.26508/lsa.201900490
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- Article
Targeting Antitumoral Proteins to Breast Cancer by Local Administration of Functional Inclusion Bodies.
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- Advanced Science, 2019, v. 6, n. 18, p. N.PAG, doi. 10.1002/advs.201900849
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- Article
Design and properties of a Myc derivative that efficiently homodimerizes.
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- Oncogene, 1998, v. 17, n. 19, p. 2463, doi. 10.1038/sj.onc.1202199
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- Article
MYC: there is more to it than cancer.
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- Frontiers in Cell & Developmental Biology, 2024, p. 1, doi. 10.3389/fcell.2024.1342872
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- Article
Myc inhibition is effective against glioma and reveals a role for Myc in proficient mitosis.
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- Nature Communications, 2014, v. 5, n. 8, p. 4632, doi. 10.1038/ncomms5632
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- Article
Inhibition of MYC attenuates tumor cell self‐renewal and promotes senescence in SMARCB1‐deficient Group 2 atypical teratoid rhabdoid tumors to suppress tumor growth in vivo.
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- International Journal of Cancer, 2019, v. 144, n. 8, p. 1983, doi. 10.1002/ijc.31873
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- Article
Tumor microenvironment: becoming sick of Myc.
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- Cellular & Molecular Life Sciences, 2012, v. 69, n. 6, p. 931, doi. 10.1007/s00018-011-0860-x
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- Article
MYC and KRAS cooperation: from historical challenges to therapeutic opportunities in cancer.
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- Signal Transduction & Targeted Therapy, 2024, v. 9, n. 1, p. 1, doi. 10.1038/s41392-024-01907-z
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- Article
Structural and Biophysical Insights into the Function of the Intrinsically Disordered Myc Oncoprotein.
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- Cells (2073-4409), 2020, v. 9, n. 4, p. 1038, doi. 10.3390/cells9041038
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- Article
Blocking Myc to Treat Cancer: Reflecting on Two Decades of Omomyc.
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- Cells (2073-4409), 2020, v. 9, n. 4, p. 883, doi. 10.3390/cells9040883
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- Article
NGF-dependent and tissue-specific transcription of vgf is regulated by a CREB-p300 and bHLH factor interaction.
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- FEBS Letters, 2002, v. 510, n. 1-2, p. 50, doi. 10.1016/S0014-5793(01)03227-6
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- Article
Biophysical characterization of the b-HLH-LZ of ΔMax, an alternatively spliced isoform of Max found in tumor cells: Towards the validation of a tumor suppressor role for the Max homodimers.
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- PLoS ONE, 2017, v. 12, n. 3, p. 1, doi. 10.1371/journal.pone.0174413
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- Article