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Dynamic expression of genes associated with schizophrenia and bipolar disorder across development.
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- Translational Psychiatry, 2019, v. 9, n. 1, p. 1, doi. 10.1038/s41398-019-0405-x
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- Article
Analysis of Intellectual Disability Copy Number Variants for Association With Schizophrenia.
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- JAMA Psychiatry, 2016, v. 73, n. 9, p. 963, doi. 10.1001/jamapsychiatry.2016.1831
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- Article
Neurotransmitters Drive Combinatorial Multistate Postsynaptic Density Networks.
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- Science Signaling, 2009, v. 2, n. 68, p. 1, doi. 10.1126/scisignal.2000102
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- Article
Predictive modeling of schizophrenia from genomic data: Comparison of polygenic risk score with kernel support vector machines approach.
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- American Journal of Medical Genetics. Part B: Neuropsychiatric Genetics, 2019, v. 180, n. 1, p. 80, doi. 10.1002/ajmg.b.32705
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- Article
Mutation intolerant genes and targets of FMRP are enriched for nonsynonymous alleles in schizophrenia.
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- American Journal of Medical Genetics. Part B: Neuropsychiatric Genetics, 2017, v. 174, n. 7, p. 724, doi. 10.1002/ajmg.b.32560
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- Article
Clustered Coding Variants in the Glutamate Receptor Complexes of Individuals with Schizophrenia and Bipolar Disorder.
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- PLoS ONE, 2011, v. 6, n. 4, p. 1, doi. 10.1371/journal.pone.0019011
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Genetic Evidence Implicates the Immune System and Cholesterol Metabolism in the Aetiology of Alzheimer's Disease.
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- PLoS ONE, 2010, v. 5, n. 11, p. 1, doi. 10.1371/journal.pone.0013950
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- Article
Reconstructing protein complexes: From proteomics to systems biology.
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- Proteomics, 2006, v. 6, n. 17, p. 4724, doi. 10.1002/pmic.200500895
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- Article
De novo mutations in schizophrenia implicate synaptic networks.
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- Nature, 2014, v. 506, n. 7487, p. 179, doi. 10.1038/nature12929
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- Article
Gender differences in CNV burden do not confound schizophrenia CNV associations.
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- Scientific Reports, 2016, p. 25986, doi. 10.1038/srep25986
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Transcriptional programs regulating neuronal differentiation are disrupted in DLG2 knockout human embryonic stem cells and enriched for schizophrenia and related disorders risk variants.
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- Nature Communications, 2022, v. 13, n. 1, p. 1, doi. 10.1038/s41467-021-27601-0
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- Article
The synapse in schizophrenia.
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- European Journal of Neuroscience, 2014, v. 39, n. 7, p. 1059, doi. 10.1111/ejn.12489
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- Article
Structural and Functional Neuroimaging of Polygenic Risk for Schizophrenia: A Recall-by-Genotype–Based Approach.
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- Schizophrenia Bulletin, 2019, v. 45, n. 2, p. 405, doi. 10.1093/schbul/sby037
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- Article
Evolutionary expansion and anatomical specialization of synapse proteome complexity.
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- Nature Neuroscience, 2008, v. 11, n. 7, p. 799, doi. 10.1038/nn.2135
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- Article
A transcriptome-wide association study implicates specific pre- and post-synaptic abnormalities in schizophrenia.
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- Human Molecular Genetics, 2020, v. 29, n. 1, p. 159, doi. 10.1093/hmg/ddz253
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- Article
Polygenic risk for schizophrenia and subcortical brain anatomy in the UK Biobank cohort.
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- Translational Psychiatry, 2020, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41398-020-00940-0
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- Article
The proteomes of neurotransmitter receptor complexes form modular networks with distributed functionality underlying plasticity and behaviour.
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- Molecular Systems Biology, 2006, v. 2, n. 1, p. E1, doi. 10.1038/msb4100041
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- Article