Found: 9
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Kelch-like protein 5-mediated ubiquitination of lysine 183 promotes proteasomal degradation of sphingosine kinase 1.
- Published in:
- Biochemical Journal, 2019, v. 476, n. 21, p. 3211, doi. 10.1042/BCJ20190245
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- Article
Mitochondrial translocation of TFEB regulates complex I and inflammation.
- Published in:
- EMBO Reports, 2024, v. 25, n. 2, p. 704, doi. 10.1038/s44319-024-00058-0
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- Article
Slit‐Robo signalling establishes a Sphingosine‐1‐phosphate gradient to polarise fin mesenchyme.
- Published in:
- EMBO Reports, 2022, v. 23, n. 8, p. 1, doi. 10.15252/embr.202154464
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- Article
Disrupted epithelial/macrophage crosstalk via Spinster homologue 2-mediated S1P signaling may drive defective macrophage phagocytic function in COPD.
- Published in:
- PLoS ONE, 2017, v. 12, n. 11, p. 1, doi. 10.1371/journal.pone.0179577
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- Article
Hydrophilic residues surrounding the S1 and S2 pockets contribute to dimerisation and catalysis in human dipeptidyl peptidase 8 (DP8).
- Published in:
- Biological Chemistry, 2010, v. 391, n. 8, p. 959, doi. 10.1515/BC.2010.111
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- Article
Integrated in silico and experimental assessment of disease relevance of PCDH19 missense variants.
- Published in:
- Human Mutation, 2021, v. 42, n. 8, p. 1030, doi. 10.1002/humu.24237
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- Article
The sphingosine 1-phosphate receptor 2/4 antagonist JTE-013 elicits off-target effects on sphingolipid metabolism.
- Published in:
- Scientific Reports, 2022, v. 12, n. 1, p. 1, doi. 10.1038/s41598-021-04009-w
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- Article
Corrigendum to “Stromal cell-derived factors 1α and 1β, inflammatory protein-10 and interferon-inducible T cell chemo-attractant are novel substrates of dipeptidyl peptidase 8” [FEBS Lett. 582 (2008) 819–825]
- Published in:
- 2008
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- Correction notice
Stromal cell-derived factors 1α and 1β, inflammatory protein-10 and interferon-inducible T cell chemo-attractant are novel substrates of dipeptidyl peptidase 8
- Published in:
- FEBS Letters, 2008, v. 582, n. 5, p. 819, doi. 10.1016/j.febslet.2008.02.005
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- Publication type:
- Article