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Alternative lengthening of telomeres: remodeling the telomere architecture.
- Published in:
- Frontiers in Oncology, 2013, v. 3, p. 1, doi. 10.3389/fonc.2013.00027
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- Article
TOP3A amplification and ATRX inactivation are mutually exclusive events in pediatric osteosarcomas using ALT.
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- EMBO Molecular Medicine, 2022, v. 14, n. 10, p. 1, doi. 10.15252/emmm.202215859
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- Article
Structural and functional characterization of the RBBP4–ZNF827 interaction and its role in NuRD recruitment to telomeres.
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- Biochemical Journal, 2018, v. 475, n. 16, p. 2667, doi. 10.1042/BCJ20180310
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- Article
Aurora Kinase B, a novel regulator of TERF1 binding and telomeric integrity.
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- Nucleic Acids Research, 2017, v. 45, n. 21, p. 12340, doi. 10.1093/nar/gkx904
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- Article
Histone variant H3.3 provides the heterochromatic H3 lysine 9 tri-methylation mark at telomeres.
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- Nucleic Acids Research, 2015, v. 43, n. 21, p. 10227, doi. 10.1093/nar/gkv847
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- Article
Telomere extension by telomerase and ALT generates variant repeats by mechanistically distinct processes.
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- Nucleic Acids Research, 2014, v. 42, n. 3, p. 1733, doi. 10.1093/nar/gkt1117
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- Article
Human telomeres that contain (CTAGGG)n repeats show replication dependent instability in somatic cells and the male germline.
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- Nucleic Acids Research, 2009, v. 37, n. 18, p. 6225, doi. 10.1093/nar/gkp629
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- Article
Telomerase activity is associated with an increase in DNA methylation at the proximal subtelomere and a reduction in telomeric transcription.
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- Nucleic Acids Research, 2009, v. 37, n. 4, p. 1152, doi. 10.1093/nar/gkn1030
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- Article
DNA C-circles are specific and quantifiable markers of alternative-lengthening-of-telomeres activity.
- Published in:
- Nature Biotechnology, 2009, v. 27, n. 12, p. 1181, doi. 10.1038/nbt.1587
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- Article
Molecular characterization of inter-telomere and intra-telomere mutations in human ALT cells.
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- Nature Genetics, 2002, v. 30, n. 3, p. 301, doi. 10.1038/ng834
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- Article
Molecular mechanisms of activity and derepression of alternative lengthening of telomeres.
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- Nature Structural & Molecular Biology, 2015, v. 22, n. 11, p. 875, doi. 10.1038/nsmb.3106
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- Article
NuRD-ZNF827 recruitment to telomeres creates a molecular scaffold for homologous recombination.
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- Nature Structural & Molecular Biology, 2014, v. 21, n. 9, p. 760, doi. 10.1038/nsmb.2877
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- Article
Spontaneous occurrence of telomeric DNA damage response in the absence of chromosome fusions.
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- Nature Structural & Molecular Biology, 2009, v. 16, n. 12, p. 1244, doi. 10.1038/nsmb.1725
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- Article
The Eyes Absent family members EYA4 and EYA1 promote PLK1 activation and successful mitosis through tyrosine dephosphorylation.
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- Nature Communications, 2024, v. 15, n. 1, p. 1, doi. 10.1038/s41467-024-45683-4
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- Article
The Eyes Absent family members EYA4 and EYA1 promote PLK1 activation and successful mitosis through tyrosine dephosphorylation.
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- Nature Communications, 2024, v. 15, n. 1, p. 1, doi. 10.1038/s41467-024-45683-4
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- Article
Extreme Telomere Length Dimorphism in the Tasmanian Devil and Related Marsupials Suggests Parental Control of Telomere Length.
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- PLoS ONE, 2012, v. 7, n. 9, p. 1, doi. 10.1371/journal.pone.0046195
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- Article
Functional Polymorphisms in the TERT Promoter Are Associated with Risk of Serous Epithelial Ovarian and Breast Cancers.
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- PLoS ONE, 2011, v. 6, n. 9, p. 1, doi. 10.1371/journal.pone.0024987
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- Article
Telomere instability detected in sporadic colon cancers, some showing mutations in a mismatch repair gene.
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- Oncogene, 2004, v. 23, n. 19, p. 3434, doi. 10.1038/sj.onc.1207477
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- Article
Author Correction: The FANCM-BLM-TOP3A-RMI complex suppresses alternative lengthening of telomeres (ALT).
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- 2019
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- Correction Notice
The FANCM-BLM-TOP3A-RMI complex suppresses alternative lengthening of telomeres (ALT).
- Published in:
- Nature Communications, 2019, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41467-019-10180-6
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- Article
BLM and SLX4 play opposing roles in recombination-dependent replication at human telomeres.
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- EMBO Journal, 2017, v. 36, n. 19, p. 2907, doi. 10.15252/embj.201796889
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- Article
Control of telomere length by a trimming mechanism that involves generation of t-circles.
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- EMBO Journal, 2009, v. 28, n. 7, p. 799, doi. 10.1038/emboj.2009.42
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- Article
Deconstructing heterogeneity of replicative senescence in human mesenchymal stem cells at single cell resolution.
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- GeroScience, 2024, v. 46, n. 1, p. 999, doi. 10.1007/s11357-023-00829-y
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- Article
FANCM suppresses DNA replication stress at ALT telomeres by disrupting TERRA R-loops.
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- Scientific Reports, 2019, v. 9, n. 1, p. 1, doi. 10.1038/s41598-019-55537-5
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- Article
Enhanced cardiac repair by telomerase reverse transcriptase over-expression in human cardiac mesenchymal stromal cells.
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- Scientific Reports, 2019, v. 9, n. 1, p. N.PAG, doi. 10.1038/s41598-019-47022-w
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- Article
Non‐telomerase telomere maintenance in human cells (97.2).
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- FASEB Journal, 2014, v. 28, p. N.PAG, doi. 10.1096/fasebj.28.1_supplement.97.2
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- Article
Progerin impairs 3D genome organization and induces fragile telomeres by limiting the dNTP pools.
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- Scientific Reports, 2021, v. 11, n. 1, p. 1, doi. 10.1038/s41598-021-92631-z
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- Article
Normal mammalian cells negatively regulate telomere length by telomere trimming.
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- Human Molecular Genetics, 2011, v. 20, n. 23, p. 4684, doi. 10.1093/hmg/ddr402
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- Article
Mechanism of structure-specific DNA binding by the FANCM branchpoint translocase.
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- Nucleic Acids Research, 2024, v. 52, n. 18, p. 11029, doi. 10.1093/nar/gkae727
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- Article
A Common Cancer Risk-Associated Allele in the hTERT Locus Encodes a Dominant Negative Inhibitor of Telomerase.
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- PLoS Genetics, 2015, v. 11, n. 6, p. 1, doi. 10.1371/journal.pgen.1005286
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- Article
qmotif: determination of telomere content from whole-genome sequence data.
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- Bioinformatics Advances, 2022, v. 2, n. 1, p. 1, doi. 10.1093/bioadv/vbac005
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- Article
A novel cause of DKC1‐related bone marrow failure: Partial deletion of the 3′ untranslated region.
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- EJHaem, 2021, v. 2, n. 2, p. 157, doi. 10.1002/jha2.165
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- Article
The clinical utility and costs of whole-genome sequencing to detect cancer susceptibility variants—a multi-site prospective cohort study.
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- Genome Medicine, 2019, p. 1, doi. 10.1186/s13073-023-01223-1
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- Article