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Identification of a Thyroid Hormone Derivative as a Pleiotropic Agent for the Treatment of Alzheimer's Disease.
- Published in:
- Pharmaceuticals (14248247), 2021, v. 14, n. 12, p. 1330, doi. 10.3390/ph14121330
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- Article
O2‐02‐02: TARGETING AMYLOID FORMATION USING RATIONALLY DESIGNED ANTIBODIES.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2018, v. 14, p. P611, doi. 10.1016/j.jalz.2018.06.2646
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- Article
Publisher Correction: Two human metabolites rescue a C. elegans model of Alzheimer's disease via a cytosolic unfolded protein response.
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- 2021
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- Correction Notice
Two human metabolites rescue a C. elegans model of Alzheimer's disease via a cytosolic unfolded protein response.
- Published in:
- Communications Biology, 2021, v. 4, n. 1, p. 1, doi. 10.1038/s42003-021-02218-7
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- Article
TDP-43 Inclusion Bodies Formed in Bacteria Are Structurally Amorphous, Non-Amyloid and Inherently Toxic to Neuroblastoma Cells.
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- PLoS ONE, 2014, v. 9, n. 1, p. 1, doi. 10.1371/journal.pone.0086720
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- Article
Cytosolic aggregation of mitochondrial proteins disrupts cellular homeostasis by stimulating the aggregation of other proteins.
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- eLife, 2021, p. 1, doi. 10.7554/eLife.65484
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- Article
Trodusquemine enhances Aβ<sub>42</sub> aggregation but suppresses its toxicity by displacing oligomers from cell membranes.
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- Nature Communications, 2019, v. 10, n. 1, p. 1, doi. 10.1038/s41467-018-07699-5
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- Article
Squalamine and Its Derivatives Modulate the Aggregation of Amyloid-β and α-Synuclein and Suppress the Toxicity of Their Oligomers.
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- Frontiers in Neuroscience, 2021, p. 1, doi. 10.3389/fnins.2021.680026
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- Article
Publisher Correction: A dopamine metabolite stabilizes neurotoxic amyloid-β oligomers.
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- 2021
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- Correction Notice
Publisher Correction: A dopamine metabolite stabilizes neurotoxic amyloid-β oligomers.
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- 2021
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- Publication type:
- Correction Notice
A dopamine metabolite stabilizes neurotoxic amyloid-β oligomers.
- Published in:
- Communications Biology, 2021, v. 4, n. 1, p. 1, doi. 10.1038/s42003-020-01490-3
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- Article
Trodusquemine displaces protein misfolded oligomers from cell membranes and abrogates their cytotoxicity through a generic mechanism.
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- Communications Biology, 2020, v. 3, n. 1, p. 1, doi. 10.1038/s42003-020-01140-8
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- Article
C. elegans expressing D76N β2-microglobulin: a model for in vivo screening of drug candidates targeting amyloidosis.
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- Scientific Reports, 2019, v. 9, n. 1, p. 1, doi. 10.1038/s41598-019-56498-5
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- Article
Exogenous misfolded protein oligomers can cross the intestinal barrier and cause a disease phenotype in C. elegans.
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- Scientific Reports, 2021, v. 11, n. 1, p. 1, doi. 10.1038/s41598-021-93527-8
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- Article
A rationally designed bicyclic peptide remodels Aβ42 aggregation in vitro and reduces its toxicity in a worm model of Alzheimer's disease.
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- Scientific Reports, 2020, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41598-020-69626-3
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- Article
Assessing motor-related phenotypes of Caenorhabditis elegans with the wide field-of-view nematode tracking platform.
- Published in:
- Nature Protocols, 2020, v. 15, n. 6, p. 2071, doi. 10.1038/s41596-020-0321-9
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- Article