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Deletion of Macrophage-Specific Glycogen Synthase Kinase (GSK)-3α Promotes Atherosclerotic Regression in Ldlr −/− Mice.
- Published in:
- International Journal of Molecular Sciences, 2022, v. 23, n. 16, p. 9293, doi. 10.3390/ijms23169293
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- Article
Macrophage Function and the Role of GSK3.
- Published in:
- International Journal of Molecular Sciences, 2021, v. 22, n. 4, p. 2206, doi. 10.3390/ijms22042206
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- Article
The Role of Endoplasmic Reticulum Stress-Glycogen Synthase Kinase-3 Signaling in Atherogenesis.
- Published in:
- International Journal of Molecular Sciences, 2018, v. 19, n. 6, p. 1607, doi. 10.3390/ijms19061607
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- Article
Correction: Cardiac regenerative capacity is age- and disease-dependent in childhood heart disease.
- Published in:
- 2021
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- Correction Notice
Characterizing the Role of Glycogen Synthase Kinase-3α/β in Macrophage Polarization and the Regulation of Pro-Atherogenic Pathways in Cultured Ldlr<sup>-/-</sup> Macrophages.
- Published in:
- Frontiers in Immunology, 2021, v. 12, p. 1, doi. 10.3389/fimmu.2021.676752
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- Article
Cardiac regenerative capacity is age- and disease-dependent in childhood heart disease.
- Published in:
- PLoS ONE, 2018, v. 13, n. 7, p. 1, doi. 10.1371/journal.pone.0200342
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- Publication type:
- Article
Myeloid GSK3α Deficiency Reduces Lesional Inflammation and Neovascularization during Atherosclerotic Progression.
- Published in:
- International Journal of Molecular Sciences, 2024, v. 25, n. 20, p. 10897, doi. 10.3390/ijms252010897
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- Article